ResearcherUniversity of Notre DameNo recent work listed
Reginald Hill
Matched on ORCID iD 0000-0002-0239-0431; OpenAlex author ID A5103555917; NIH RePORTER profile ID 11180077. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 0, last author 4)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 13
2012: 2 papers · 2013: none · 2014: none · 2015: 1 paper · 2016: 1 paper · 2017: 2 papers, 1 grant · 2018: 1 paper
Papers 7
- PTEN-Dependent Stabilization of MTSS1 Inhibits Metastatic Phenotype in Pancreatic Ductal Adenocarcinoma. Last author DOI 10.1016/j.neo.2017.10.004↗
- Cancer-associated fibroblast exosomes regulate survival and proliferation of pancreatic cancer cells. Last author DOI 10.1038/onc.2016.353↗
- Loss of MTSS1 results in increased metastatic potential in pancreatic cancer. Last author DOI 10.18632/oncotarget.14869↗
- Expression of GRP78, Master Regulator of the Unfolded Protein Response, Increases Chemoresistance in Pancreatic Ductal Adenocarcinoma. Last author DOI 10.1158/1535-7163.mct-15-0774↗
- On-chip surface acoustic wave lysis and ion-exchange nanomembrane detection of exosomal RNA for pancreatic cancer study and diagnosis. Co-author DOI 10.1039/c5lc00036j↗
- Integrative survival-based molecular profiling of human pancreatic cancer. Co-author DOI 10.1158/1078-0432.ccr-11-1539↗
- Cell intrinsic role of COX-2 in pancreatic cancer development. Co-author DOI 10.1158/1535-7163.mct-12-0342↗
Grants 1
Closest collaborators
The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Ann E. Zeleniak · 4 shared papers, 2016–2018 · strength 0.36
- Paper PTEN-Dependent Stabilization of MTSS1 Inhibits Metastatic Phenotype in Pancreatic Ductal Adenocarcinoma. DOI 10.1016/j.neo.2017.10.004↗
- Paper Loss of MTSS1 results in increased metastatic potential in pancreatic cancer. DOI 10.18632/oncotarget.14869↗
- Paper Cancer-associated fibroblast exosomes regulate survival and proliferation of pancreatic cancer cells. DOI 10.1038/onc.2016.353↗
- Melissa L. Fishel · 3 shared papers, 2017–2018 · strength 0.29
- Paper PTEN-Dependent Stabilization of MTSS1 Inhibits Metastatic Phenotype in Pancreatic Ductal Adenocarcinoma. DOI 10.1016/j.neo.2017.10.004↗
- Paper Loss of MTSS1 results in increased metastatic potential in pancreatic cancer. DOI 10.18632/oncotarget.14869↗
- Paper Cancer-associated fibroblast exosomes regulate survival and proliferation of pancreatic cancer cells. DOI 10.1038/onc.2016.353↗
- Satyajyoti Senapati · 1 shared paper, 1 shared grant, 2015–2017 · strength 0.26
- David B. Go · 1 shared paper, 1 shared grant, 2015–2017 · strength 0.26
- Hsueh-Chia Chang · 1 shared paper, 1 shared grant, 2015–2017 · strength 0.26
- Katherine E. Richards · 2 shared papers, 2015–2017 · strength 0.14
- Timothy R. Donahue · 3 shared papers, 2012–2016 · strength 0.12
- Paper Expression of GRP78, Master Regulator of the Unfolded Protein Response, Increases Chemoresistance in Pancreatic Ductal … DOI 10.1158/1535-7163.mct-15-0774↗
- Paper Cell intrinsic role of COX-2 in pancreatic cancer development. DOI 10.1158/1535-7163.mct-12-0342↗
- Paper Integrative survival-based molecular profiling of human pancreatic cancer. DOI 10.1158/1078-0432.ccr-11-1539↗
- Hong Wu · 3 shared papers, 2012–2016 · strength 0.12
- Paper Expression of GRP78, Master Regulator of the Unfolded Protein Response, Increases Chemoresistance in Pancreatic Ductal … DOI 10.1158/1535-7163.mct-15-0774↗
- Paper Cell intrinsic role of COX-2 in pancreatic cancer development. DOI 10.1158/1535-7163.mct-12-0342↗
- Paper Integrative survival-based molecular profiling of human pancreatic cancer. DOI 10.1158/1078-0432.ccr-11-1539↗
- Jenifer B. Gifford · 1 shared paper, 2016 · strength 0.07
- Daniel Taller · 1 shared paper, 2015 · strength 0.06
- Anne Kovochich · 1 shared paper, 2012 · strength 0.03
- James J. Farrell · 1 shared paper, 2012 · strength 0.03
- David W. Dawson · 1 shared paper, 2012 · strength 0.03
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.