ResearcherColumbia UniversityActive in the last 2 years
Alina Cornelia Iuga
Matched on ORCID iD 0000-0002-2351-8289; OpenAlex author ID A5032670177; NIH RePORTER profile ID 14408862. Sources: OpenAlex.
Their work
- Papers
- 13
- Trials
- 0
- Grants
- 1
- Labs led
- 0
- Collaborators
- 80
2012: 1 paper · 2013: none · 2014: none · 2015: none · 2016: none · 2017: none · 2018: 2 papers · 2019: 1 paper · 2020: 1 paper · 2021: 1 paper · 2022: none · 2023: 2 papers · 2024: none · 2025: 1 paper · 2026: 4 papers, 1 grant
Papers 13
- Limitations of GATA6 and CK5 as surrogate markers for molecular subtyping in pancreatic cancer. Co-author DOI 10.1016/j.hpb.2026.08.001↗
- Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study with single-cell correlatives. Co-author DOI 10.1038/s41467-026-76559-4↗
- Developmental and MAPK-responsive transcription factors regulate distinct malignant cell states and associated genetic dependencies in pancreatic cancer. Co-author DOI 10.1038/s41588-026-02714-8↗
- Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Clinical Models: MEKiAUTO. Co-author DOI 10.1200/po-26-00138↗
- Tumor-Intrinsic Kinome Landscape of Pancreatic Cancer Reveals New Therapeutic Approaches. Co-author DOI 10.1158/2159-8290.cd-23-1480↗
- Ras-dependent activation of BMAL2 regulates hypoxic metabolism in pancreatic cancer Co-author DOI 10.1101/2023.03.19.533333↗
- Cytotoxic chemotherapy potentiates the immune response and efficacy of combination CXCR4/PD-1 inhibition in models of pancreatic ductal adenocarcinoma Co-author DOI 10.1101/2023.12.24.573257↗
- Interleukin-1β-induced pancreatitis promotes pancreatic ductal adenocarcinoma via B lymphocyte-mediated immune suppression. Co-author DOI 10.1136/gutjnl-2019-319912↗
- Noninvasive Young's modulus visualization of fibrosis progression and delineation of pancreatic ductal adenocarcinoma (PDAC) tumors using Harmonic Motion Elast… Co-author DOI 10.7150/thno.37965↗
- Experimental microdissection enables functional harmonisation of pancreatic cancer subtypes. Co-author DOI 10.1136/gutjnl-2018-317706↗
- β2 Adrenergic-Neurotrophin Feedforward Loop Promotes Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2017.11.007↗
- Transcriptional deconvolution reveals consistent functional subtypes of pancreatic cancer epithelium and stroma Co-author DOI 10.1101/288779↗
- Perineural and intraneural invasion in posttherapy pancreaticoduodenectomy specimens predicts poor prognosis in patients with pancreatic ductal adenocarcinoma. Co-author DOI 10.1097/pas.0b013e31824104c5↗
Grants 1
Closest collaborators
The 25 strongest of 80 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Kenneth P. Olive · 10 shared papers, 2018–2026 · strength 1.30
- Paper Developmental and MAPK-responsive transcription factors regulate distinct malignant cell states and associated genetic … DOI 10.1038/s41588-026-02714-8↗
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Paper Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study w… DOI 10.1038/s41467-026-76559-4↗
- Jen Jen Yeh · 2 shared papers, 1 shared grant, 2025–2026 · strength 1.15
- Grant SToP Cancer SPORE: Administrative Core A P50CA257911↗
- Paper Limitations of GATA6 and CK5 as surrogate markers for molecular subtyping in pancreatic cancer. DOI 10.1016/j.hpb.2026.08.001↗
- Paper Tumor-Intrinsic Kinome Landscape of Pancreatic Cancer Reveals New Therapeutic Approaches. DOI 10.1158/2159-8290.cd-23-1480↗
- Gulam Abbas Manji · 5 shared papers, 2023–2026 · strength 0.90
- Paper Developmental and MAPK-responsive transcription factors regulate distinct malignant cell states and associated genetic … DOI 10.1038/s41588-026-02714-8↗
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Paper Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study w… DOI 10.1038/s41467-026-76559-4↗
- H. Carlo Maurer · 6 shared papers, 2018–2026 · strength 0.70
- Paper Developmental and MAPK-responsive transcription factors regulate distinct malignant cell states and associated genetic … DOI 10.1038/s41588-026-02714-8↗
- Paper Cytotoxic chemotherapy potentiates the immune response and efficacy of combination CXCR4/PD-1 inhibition in models of p… DOI 10.1101/2023.12.24.573257↗
- Paper Ras-dependent activation of BMAL2 regulates hypoxic metabolism in pancreatic cancer DOI 10.1101/2023.03.19.533333↗
- Andrea Califano · 5 shared papers, 2018–2026 · strength 0.68
- Paper Developmental and MAPK-responsive transcription factors regulate distinct malignant cell states and associated genetic … DOI 10.1038/s41588-026-02714-8↗
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Paper Ras-dependent activation of BMAL2 regulates hypoxic metabolism in pancreatic cancer DOI 10.1101/2023.03.19.533333↗
- Michael R Kosorok · 1 shared grant, 2026 · strength 0.67
- Gianpietro Dotti · 1 shared grant, 2026 · strength 0.67
- Channing J. Der · 1 shared grant, 2026 · strength 0.67
- Jonathan S. Serody · 1 shared grant, 2026 · strength 0.67
- ALBERT Sidney BALDWIN · 1 shared grant, 2026 · strength 0.67
- Michael S. May · 3 shared papers, 2023–2026 · strength 0.56
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Paper Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study w… DOI 10.1038/s41467-026-76559-4↗
- Paper Cytotoxic chemotherapy potentiates the immune response and efficacy of combination CXCR4/PD-1 inhibition in models of p… DOI 10.1101/2023.12.24.573257↗
- Alexander Grenander Raufi · 3 shared papers, 2023–2026 · strength 0.56
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Paper Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study w… DOI 10.1038/s41467-026-76559-4↗
- Paper Cytotoxic chemotherapy potentiates the immune response and efficacy of combination CXCR4/PD-1 inhibition in models of p… DOI 10.1101/2023.12.24.573257↗
- Naim U. Rashid · 2 shared papers, 2025–2026 · strength 0.48
- John A. Chabot · 4 shared papers, 2018–2026 · strength 0.43
- Paper Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study w… DOI 10.1038/s41467-026-76559-4↗
- Paper Noninvasive Young's modulus visualization of fibrosis progression and delineation of pancreatic ductal adenocarcinoma (… DOI 10.7150/thno.37965↗
- Paper Experimental microdissection enables functional harmonisation of pancreatic cancer subtypes. DOI 10.1136/gutjnl-2018-317706↗
- Pasquale Laise · 3 shared papers, 2019–2026 · strength 0.42
- Paper Developmental and MAPK-responsive transcription factors regulate distinct malignant cell states and associated genetic … DOI 10.1038/s41588-026-02714-8↗
- Paper Ras-dependent activation of BMAL2 regulates hypoxic metabolism in pancreatic cancer DOI 10.1101/2023.03.19.533333↗
- Paper Experimental microdissection enables functional harmonisation of pancreatic cancer subtypes. DOI 10.1136/gutjnl-2018-317706↗
- Urszula N. Wasko · 2 shared papers, 2026 · strength 0.41
- Paper Developmental and MAPK-responsive transcription factors regulate distinct malignant cell states and associated genetic … DOI 10.1038/s41588-026-02714-8↗
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Susan Elaine Bates · 2 shared papers, 2026 · strength 0.39
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Paper Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study w… DOI 10.1038/s41467-026-76559-4↗
- Linda Y. Wu · 2 shared papers, 2026 · strength 0.39
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Paper Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study w… DOI 10.1038/s41467-026-76559-4↗
- Benjamin Izar · 2 shared papers, 2026 · strength 0.39
- Paper Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Cl… DOI 10.1200/po-26-00138↗
- Paper Motixafortide, cemiplimab, gemcitabine and nab-paclitaxel in metastatic pancreatic cancer: a single-arm phase 2 study w… DOI 10.1038/s41467-026-76559-4↗
- Jeanine M. Genkinger · 4 shared papers, 2018–2023 · strength 0.38
- Paper Ras-dependent activation of BMAL2 regulates hypoxic metabolism in pancreatic cancer DOI 10.1101/2023.03.19.533333↗
- Paper Interleukin-1β-induced pancreatitis promotes pancreatic ductal adenocarcinoma via B lymphocyte-mediated immune suppress… DOI 10.1136/gutjnl-2019-319912↗
- Paper Experimental microdissection enables functional harmonisation of pancreatic cancer subtypes. DOI 10.1136/gutjnl-2018-317706↗
- Álvaro Curiel‐García · 2 shared papers, 2023–2026 · strength 0.34
- Jaewon James Lee · 1 shared paper, 2026 · strength 0.29
- Saryn Doucette · 1 shared paper, 2026 · strength 0.29
- Mohammed Aldakkak · 1 shared paper, 2026 · strength 0.29
- Douglas B. Evans · 1 shared paper, 2026 · strength 0.29
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.