ResearcherUniversity of ArizonaActive in the last 2 years

Nathan A. Ellis

ORCID-verified (high confidence, 0.95), identity_conf@1As of 8 November 2026

Matched on ORCID iD 0000-0003-4231-502X; OpenAlex author ID A5054189237; NIH RePORTER profile ID 1925352. Sources: OpenAlex.

Their work

Papers: 1
Trials: 0
Grants: 1
Labs led: 0
Collaborators: 4

Papers, trials and grants by year

2026: 1 paper, 1 grant

Papers 1

An intron retention-derived secreted Endoglin Isoform as a circulating biomarker in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.neo.2026.101379 ↗

Grants 1

Cancer Biology Program (CBP) P30CA023074 ↗

Closest collaborators

The 4 strongest of 4 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Koenraad Van Doorslaer · 1 shared grant, 2026 · strength 1.26
- Grant Cancer Biology Program (CBP) P30CA023074 ↗
Christopher Cartmell · 1 shared paper, 2026 · strength 0.28
- Paper An intron retention-derived secreted Endoglin Isoform as a circulating biomarker in pancreatic ductal adenocarcinoma. DOI 10.1016/j.neo.2026.101379 ↗
Hannah R. Ortiz · 1 shared paper, 2026 · strength 0.28
- Paper An intron retention-derived secreted Endoglin Isoform as a circulating biomarker in pancreatic ductal adenocarcinoma. DOI 10.1016/j.neo.2026.101379 ↗
Nam Y Lee · 1 shared paper, 2026 · strength 0.28
- Paper An intron retention-derived secreted Endoglin Isoform as a circulating biomarker in pancreatic ductal adenocarcinoma. DOI 10.1016/j.neo.2026.101379 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.