ResearcherMichael E. DeBakey VA Medical CenterActive in the last 2 years
Qizhi Cathy Yao
Matched on ORCID iD 0000-0002-2267-0588; OpenAlex author ID A5038846608; NIH RePORTER profile ID 6605418. Sources: OpenAlex.
Their work
- Papers
- 19 (first author 0, last author 8)
- Trials
- 0
- Grants
- 4
- Labs led
- 2
- Collaborators
- 48
2011: 5 papers · 2012: none · 2013: 4 papers · 2014: none · 2015: none · 2016: 2 papers · 2017: none · 2018: none · 2019: 1 paper, 1 grant · 2020: 2 papers · 2021: 1 paper · 2022: 1 paper · 2023: none · 2024: 1 paper, 1 grant · 2025: 2 papers, 1 grant · 2026: none · 2027: 1 grant
Papers 19
- Oncolytic adeno-immunotherapy improves allogeneic adoptive HER2.CAR-NK function against pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.omton.2025.201006↗
- Mesothelin-Associated Anti-Senescence Through P53 in Pancreatic Ductal Adenocarcinoma. Last author DOI 10.3390/cancers17122058↗
- Proteome-wide association study and functional validation identify novel protein markers for pancreatic ductal adenocarcinoma. Co-author DOI 10.1093/gigascience/giae012↗
- XDec-CHI reveals immunosuppressive interactions in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.isci.2022.105249↗
- A subset of cytotoxic effector memory T cells enhances CAR T cell efficacy in a model of pancreatic ductal adenocarcinoma. Co-author DOI 10.1126/scitranslmed.abc3196↗
- Histoepigenetic analysis of the mesothelin network within pancreatic ductal adenocarcinoma cells reveals regulation of retinoic acid receptor gamma and AKT by … Co-author DOI 10.1038/s41389-020-00245-3↗
- A Transcriptome-Wide Association Study Identifies Candidate Susceptibility Genes for Pancreatic Cancer Risk. Co-author DOI 10.1158/0008-5472.can-20-1353↗
- Mesothelin and TGF-α predict pancreatic cancer cell sensitivity to EGFR inhibitors and effective combination treatment with trametinib. Last author DOI 10.1371/journal.pone.0213294↗
- Chemo-immunotherapy mediates durable cure of orthotopic KrasG12D/p53-/- pancreatic ductal adenocarcinoma. Co-author DOI 10.1080/2162402x.2016.1213933↗
- Overexpression of Semaphorin-3E enhances pancreatic cancer cell growth and associates with poor patient survival. Last author DOI 10.18632/oncotarget.13704↗
- A novel epigenetic CREB-miR-373 axis mediates ZIP4-induced pancreatic cancer growth. Co-author DOI 10.1002/emmm.201302507↗
- A tumorigenic factor interactome connected through tumor suppressor microRNA-198 in human pancreatic cancer. Last author DOI 10.1158/1078-0432.ccr-12-3776↗
- Mesothelin virus-like particle immunization controls pancreatic cancer growth through CD8+ T cell induction and reduction in the frequency of CD4+ foxp3+ ICOS-… Last author DOI 10.1371/journal.pone.0068303↗
- Gene profile identifies zinc transporters differentially expressed in normal human organs and human pancreatic cancer. Co-author DOI 10.2174/1566524011313030009↗
- Roles and mechanisms of microRNAs in pancreatic cancer. Co-author DOI 10.1007/s00268-010-0952-z↗
- Mesothelin overexpression promotes autocrine IL-6/sIL-6R trans-signaling to stimulate pancreatic cancer cell proliferation. Last author DOI 10.1093/carcin/bgr075↗
- Chimeric Trop2 virus-like particles: a potential immunotherapeutic approach against pancreatic cancer. Last author DOI 10.1097/cji.0b013e318209ee72↗
- Mesothelin confers pancreatic cancer cell resistance to TNF-α-induced apoptosis through Akt/PI3K/NF-κB activation and IL-6/Mcl-1 overexpression. Last author DOI 10.1186/1476-4598-10-106↗
- Overcoming drug resistance in pancreatic cancer. Co-author DOI 10.1517/14728222.2011.566216↗
Grants 4
- Novel Pathogenic Roles and Mechanisms of Fibrinogen in Pancreatic Cancer I01BX005806↗
- BLR&D Research Career Scientist Award Application IK6BX007143↗
- Stratification of Pancreatic Cancer Subpopulations for Effective Immunotherapy I01CX001822↗
- A novel miR-198 replacement therapy for pancreatic cancer R01CA183984↗
Closest collaborators
The 25 strongest of 48 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Changyi Chen · 11 shared papers, 2011–2025 · strength 0.87
- Paper Mesothelin-Associated Anti-Senescence Through P53 in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/cancers17122058↗
- Paper Mesothelin and TGF-α predict pancreatic cancer cell sensitivity to EGFR inhibitors and effective combination treatment … DOI 10.1371/journal.pone.0213294↗
- Paper A subset of cytotoxic effector memory T cells enhances CAR T cell efficacy in a model of pancreatic ductal adenocarcino… DOI 10.1126/scitranslmed.abc3196↗
- Dongliang Liu · 4 shared papers, 2019–2025 · strength 0.72
- Paper Mesothelin-Associated Anti-Senescence Through P53 in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/cancers17122058↗
- Paper Histoepigenetic analysis of the mesothelin network within pancreatic ductal adenocarcinoma cells reveals regulation of … DOI 10.1038/s41389-020-00245-3↗
- Paper Mesothelin and TGF-α predict pancreatic cancer cell sensitivity to EGFR inhibitors and effective combination treatment … DOI 10.1371/journal.pone.0213294↗
- Aleksandar Milosavljevic · 2 shared papers, 2020–2022 · strength 0.34
- Emily L. LaPlante · 2 shared papers, 2020–2022 · strength 0.34
- Dalia H. Ghoneim · 2 shared papers, 2020–2024 · strength 0.27
- Paper Proteome-wide association study and functional validation identify novel protein markers for pancreatic ductal adenocar… DOI 10.1093/gigascience/giae012↗
- Paper A Transcriptome-Wide Association Study Identifies Candidate Susceptibility Genes for Pancreatic Cancer Risk. DOI 10.1158/0008-5472.can-20-1353↗
- Lang Wu · 2 shared papers, 2020–2024 · strength 0.27
- Paper Proteome-wide association study and functional validation identify novel protein markers for pancreatic ductal adenocar… DOI 10.1093/gigascience/giae012↗
- Paper A Transcriptome-Wide Association Study Identifies Candidate Susceptibility Genes for Pancreatic Cancer Risk. DOI 10.1158/0008-5472.can-20-1353↗
- Jingjing Zhu · 2 shared papers, 2020–2024 · strength 0.27
- Paper Proteome-wide association study and functional validation identify novel protein markers for pancreatic ductal adenocar… DOI 10.1093/gigascience/giae012↗
- Paper A Transcriptome-Wide Association Study Identifies Candidate Susceptibility Genes for Pancreatic Cancer Risk. DOI 10.1158/0008-5472.can-20-1353↗
- Ryu Matsumoto · 1 shared paper, 2025 · strength 0.24
- Greyson W. G. Biegert · 1 shared paper, 2025 · strength 0.24
- Masataka Suzuki · 1 shared paper, 2025 · strength 0.24
- Ethan C. Poteet · 2 shared papers, 2016–2019 · strength 0.17
- Paper Mesothelin and TGF-α predict pancreatic cancer cell sensitivity to EGFR inhibitors and effective combination treatment … DOI 10.1371/journal.pone.0213294↗
- Paper Overexpression of Semaphorin-3E enhances pancreatic cancer cell growth and associates with poor patient survival. DOI 10.18632/oncotarget.13704↗
- George Van Buren · 2 shared papers, 2016–2019 · strength 0.17
- Paper Mesothelin and TGF-α predict pancreatic cancer cell sensitivity to EGFR inhibitors and effective combination treatment … DOI 10.1371/journal.pone.0213294↗
- Paper Overexpression of Semaphorin-3E enhances pancreatic cancer cell growth and associates with poor patient survival. DOI 10.18632/oncotarget.13704↗
- Tanxin Liu · 1 shared paper, 2024 · strength 0.16
- Tao Liu · 1 shared paper, 2024 · strength 0.16
- Youping Deng · 1 shared paper, 2024 · strength 0.16
- Hua Zhong · 1 shared paper, 2024 · strength 0.16
- Shuai Liu · 1 shared paper, 2024 · strength 0.16
- Vanaja Konduri · 2 shared papers, 2016–2021 · strength 0.16
- Paper A subset of cytotoxic effector memory T cells enhances CAR T cell efficacy in a model of pancreatic ductal adenocarcino… DOI 10.1126/scitranslmed.abc3196↗
- Paper Chemo-immunotherapy mediates durable cure of orthotopic KrasG12D/p53-/- pancreatic ductal adenocarcinoma. DOI 10.1080/2162402x.2016.1213933↗
- William Karl Decker · 2 shared papers, 2016–2021 · strength 0.16
- Paper A subset of cytotoxic effector memory T cells enhances CAR T cell efficacy in a model of pancreatic ductal adenocarcino… DOI 10.1126/scitranslmed.abc3196↗
- Paper Chemo-immunotherapy mediates durable cure of orthotopic KrasG12D/p53-/- pancreatic ductal adenocarcinoma. DOI 10.1080/2162402x.2016.1213933↗
- William E. Fisher · 4 shared papers, 2013–2016 · strength 0.15
- Paper Chemo-immunotherapy mediates durable cure of orthotopic KrasG12D/p53-/- pancreatic ductal adenocarcinoma. DOI 10.1080/2162402x.2016.1213933↗
- Paper A tumorigenic factor interactome connected through tumor suppressor microRNA-198 in human pancreatic cancer. DOI 10.1158/1078-0432.ccr-12-3776↗
- Paper Gene profile identifies zinc transporters differentially expressed in normal human organs and human pancreatic cancer. DOI 10.2174/1566524011313030009↗
- Eugene Lurie · 1 shared paper, 2020 · strength 0.13
- Duo Liu · 1 shared paper, 2020 · strength 0.11
- Eric R. Gamazon · 1 shared paper, 2020 · strength 0.11
- Christian Marín‐Müller · 3 shared papers, 2011–2013 · strength 0.10
- Paper A tumorigenic factor interactome connected through tumor suppressor microRNA-198 in human pancreatic cancer. DOI 10.1158/1078-0432.ccr-12-3776↗
- Paper Mesothelin overexpression promotes autocrine IL-6/sIL-6R trans-signaling to stimulate pancreatic cancer cell proliferat… DOI 10.1093/carcin/bgr075↗
- Paper Mesothelin confers pancreatic cancer cell resistance to TNF-α-induced apoptosis through Akt/PI3K/NF-κB activation and I… DOI 10.1186/1476-4598-10-106↗
- Uddalak Bharadwaj · 3 shared papers, 2011–2013 · strength 0.10
- Paper A tumorigenic factor interactome connected through tumor suppressor microRNA-198 in human pancreatic cancer. DOI 10.1158/1078-0432.ccr-12-3776↗
- Paper Mesothelin overexpression promotes autocrine IL-6/sIL-6R trans-signaling to stimulate pancreatic cancer cell proliferat… DOI 10.1093/carcin/bgr075↗
- Paper Mesothelin confers pancreatic cancer cell resistance to TNF-α-induced apoptosis through Akt/PI3K/NF-κB activation and I… DOI 10.1186/1476-4598-10-106↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.