ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Ignacio Ivan Wistuba Oyarzun
Matched on ORCID iD 0000-0003-3365-6340; OpenAlex author ID A5028085268; NIH RePORTER profile ID 8625310. Sources: OpenAlex.
Their work
- Papers
- 11
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 57
2015: 1 paper · 2016: none · 2017: 2 papers · 2018: 2 papers · 2019: 1 paper · 2020: none · 2021: 1 paper, 1 grant · 2022: none · 2023: none · 2024: 2 papers · 2025: 1 paper · 2026: 1 paper
Papers 11
- Syndecan-1-targeted therapeutic antibody impairs macropinocytosis and elicits antitumor immunity in pancreatic cancer. Co-author DOI 10.1016/j.xcrm.2026.102613↗
- TREM2 Depletion in Pancreatic Cancer Elicits Pathogenic Inflammation and Accelerates Tumor Progression via Enriching IL-1β+ Macrophages. Co-author DOI 10.1053/j.gastro.2025.01.244↗
- Loss of p53 and SMAD4 induces adenosquamous subtype pancreatic cancer in the absence of an oncogenic KRAS mutation. Co-author DOI 10.1016/j.xcrm.2024.101711↗
- Genetic Deletion of Galectin-3 Inhibits Pancreatic Cancer Progression and Enhances the Efficacy of Immunotherapy. Co-author DOI 10.1053/j.gastro.2024.03.007↗
- Overexpression of CD73 in pancreatic ductal adenocarcinoma is associated with immunosuppressive tumor microenvironment and poor survival. Co-author DOI 10.1016/j.pan.2021.03.018↗
- Expression of Epithelial-Mesenchymal Transition Markers in Treated Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1097/mpa.0000000000001432↗
- Surfaceome profiling enables isolation of cancer-specific exosomal cargo in liquid biopsies from pancreatic cancer patients. Co-author DOI 10.1093/annonc/mdx542↗
- Dynamic changes during the treatment of pancreatic cancer. Co-author DOI 10.18632/oncotarget.24483↗
- High prevalence of mutant KRAS in circulating exosome-derived DNA from early-stage pancreatic cancer patients. Co-author DOI 10.1093/annonc/mdx004↗
- Prognostic Significance of Tumor-Infiltrating Lymphocytes in Patients With Pancreatic Ductal Adenocarcinoma Treated With Neoadjuvant Chemotherapy. Co-author DOI 10.1097/mpa.0000000000000914↗
- RSPO2 Enhances Canonical Wnt Signaling to Confer Stemness-Associated Traits to Susceptible Pancreatic Cancer Cells. Co-author DOI 10.1158/0008-5472.can-14-1327↗
Grants 1
Closest collaborators
The 25 strongest of 57 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Anirban Maitra · 9 shared papers, 1 shared grant, 2017–2025 · strength 1.57
- Grant Pancreatic Ductal Adenocarcinoma Translational Resource Center (PATReC) U24CA224020↗
- Paper TREM2 Depletion in Pancreatic Cancer Elicits Pathogenic Inflammation and Accelerates Tumor Progression via Enriching IL… DOI 10.1053/j.gastro.2025.01.244↗
- Paper Loss of p53 and SMAD4 induces adenosquamous subtype pancreatic cancer in the absence of an oncogenic KRAS mutation. DOI 10.1016/j.xcrm.2024.101711↗
- Daowei Yang · 3 shared papers, 2024–2025 · strength 0.70
- Paper TREM2 Depletion in Pancreatic Cancer Elicits Pathogenic Inflammation and Accelerates Tumor Progression via Enriching IL… DOI 10.1053/j.gastro.2025.01.244↗
- Paper Loss of p53 and SMAD4 induces adenosquamous subtype pancreatic cancer in the absence of an oncogenic KRAS mutation. DOI 10.1016/j.xcrm.2024.101711↗
- Paper Genetic Deletion of Galectin-3 Inhibits Pancreatic Cancer Progression and Enhances the Efficacy of Immunotherapy. DOI 10.1053/j.gastro.2024.03.007↗
- Yang Chen · 3 shared papers, 2024–2025 · strength 0.70
- Paper TREM2 Depletion in Pancreatic Cancer Elicits Pathogenic Inflammation and Accelerates Tumor Progression via Enriching IL… DOI 10.1053/j.gastro.2025.01.244↗
- Paper Loss of p53 and SMAD4 induces adenosquamous subtype pancreatic cancer in the absence of an oncogenic KRAS mutation. DOI 10.1016/j.xcrm.2024.101711↗
- Paper Genetic Deletion of Galectin-3 Inhibits Pancreatic Cancer Progression and Enhances the Efficacy of Immunotherapy. DOI 10.1053/j.gastro.2024.03.007↗
- Xinlei Sun · 3 shared papers, 2024–2025 · strength 0.70
- Paper TREM2 Depletion in Pancreatic Cancer Elicits Pathogenic Inflammation and Accelerates Tumor Progression via Enriching IL… DOI 10.1053/j.gastro.2025.01.244↗
- Paper Loss of p53 and SMAD4 induces adenosquamous subtype pancreatic cancer in the absence of an oncogenic KRAS mutation. DOI 10.1016/j.xcrm.2024.101711↗
- Paper Genetic Deletion of Galectin-3 Inhibits Pancreatic Cancer Progression and Enhances the Efficacy of Immunotherapy. DOI 10.1053/j.gastro.2024.03.007↗
- Huamin Wang · 5 shared papers, 2017–2024 · strength 0.68
- Paper Loss of p53 and SMAD4 induces adenosquamous subtype pancreatic cancer in the absence of an oncogenic KRAS mutation. DOI 10.1016/j.xcrm.2024.101711↗
- Paper Genetic Deletion of Galectin-3 Inhibits Pancreatic Cancer Progression and Enhances the Efficacy of Immunotherapy. DOI 10.1053/j.gastro.2024.03.007↗
- Paper Overexpression of CD73 in pancreatic ductal adenocarcinoma is associated with immunosuppressive tumor microenvironment … DOI 10.1016/j.pan.2021.03.018↗
- Subrata Sen · 1 shared grant, 2021 · strength 0.44
- Matthew H. G. Katz · 6 shared papers, 2017–2021 · strength 0.43
- Paper Overexpression of CD73 in pancreatic ductal adenocarcinoma is associated with immunosuppressive tumor microenvironment … DOI 10.1016/j.pan.2021.03.018↗
- Paper Expression of Epithelial-Mesenchymal Transition Markers in Treated Pancreatic Ductal Adenocarcinoma. DOI 10.1097/mpa.0000000000001432↗
- Paper Surfaceome profiling enables isolation of cancer-specific exosomal cargo in liquid biopsies from pancreatic cancer pati… DOI 10.1093/annonc/mdx542↗
- Hua Wang · 3 shared papers, 2017–2024 · strength 0.38
- Paper Loss of p53 and SMAD4 induces adenosquamous subtype pancreatic cancer in the absence of an oncogenic KRAS mutation. DOI 10.1016/j.xcrm.2024.101711↗
- Paper Overexpression of CD73 in pancreatic ductal adenocarcinoma is associated with immunosuppressive tumor microenvironment … DOI 10.1016/j.pan.2021.03.018↗
- Paper Prognostic Significance of Tumor-Infiltrating Lymphocytes in Patients With Pancreatic Ductal Adenocarcinoma Treated Wit… DOI 10.1097/mpa.0000000000000914↗
- Robert A. Wolff · 5 shared papers, 2017–2021 · strength 0.38
- Paper Overexpression of CD73 in pancreatic ductal adenocarcinoma is associated with immunosuppressive tumor microenvironment … DOI 10.1016/j.pan.2021.03.018↗
- Paper Expression of Epithelial-Mesenchymal Transition Markers in Treated Pancreatic Ductal Adenocarcinoma. DOI 10.1097/mpa.0000000000001432↗
- Paper Surfaceome profiling enables isolation of cancer-specific exosomal cargo in liquid biopsies from pancreatic cancer pati… DOI 10.1093/annonc/mdx542↗
- Gauri Rajani Varadhachary · 5 shared papers, 2017–2019 · strength 0.31
- Paper Expression of Epithelial-Mesenchymal Transition Markers in Treated Pancreatic Ductal Adenocarcinoma. DOI 10.1097/mpa.0000000000001432↗
- Paper Surfaceome profiling enables isolation of cancer-specific exosomal cargo in liquid biopsies from pancreatic cancer pati… DOI 10.1093/annonc/mdx542↗
- Paper Prognostic Significance of Tumor-Infiltrating Lymphocytes in Patients With Pancreatic Ductal Adenocarcinoma Treated Wit… DOI 10.1097/mpa.0000000000000914↗
- Jeffrey E. Lee · 3 shared papers, 2017–2021 · strength 0.26
- Paper Overexpression of CD73 in pancreatic ductal adenocarcinoma is associated with immunosuppressive tumor microenvironment … DOI 10.1016/j.pan.2021.03.018↗
- Paper Expression of Epithelial-Mesenchymal Transition Markers in Treated Pancreatic Ductal Adenocarcinoma. DOI 10.1097/mpa.0000000000001432↗
- Paper Prognostic Significance of Tumor-Infiltrating Lymphocytes in Patients With Pancreatic Ductal Adenocarcinoma Treated Wit… DOI 10.1097/mpa.0000000000000914↗
- Zecheng Yang · 1 shared paper, 2026 · strength 0.22
- Yongkun Wei · 1 shared paper, 2026 · strength 0.22
- Timothy Paul Heffernan · 1 shared paper, 2026 · strength 0.22
- Haoqiang Ying · 1 shared paper, 2026 · strength 0.22
- Wantong Yao · 1 shared paper, 2026 · strength 0.22
- Giulio Francesco Draetta · 1 shared paper, 2026 · strength 0.22
- Hua Wang · 1 shared paper, 2024 · strength 0.21
- Michael P. Kim · 2 shared papers, 2019–2021 · strength 0.21
- Kelvin Allenson · 3 shared papers, 2017–2018 · strength 0.17
- Paper Surfaceome profiling enables isolation of cancer-specific exosomal cargo in liquid biopsies from pancreatic cancer pati… DOI 10.1093/annonc/mdx542↗
- Paper Dynamic changes during the treatment of pancreatic cancer. DOI 10.18632/oncotarget.24483↗
- Paper High prevalence of mutant KRAS in circulating exosome-derived DNA from early-stage pancreatic cancer patients. DOI 10.1093/annonc/mdx004↗
- Vincent Bernard · 3 shared papers, 2017–2018 · strength 0.17
- Paper Surfaceome profiling enables isolation of cancer-specific exosomal cargo in liquid biopsies from pancreatic cancer pati… DOI 10.1093/annonc/mdx542↗
- Paper Dynamic changes during the treatment of pancreatic cancer. DOI 10.18632/oncotarget.24483↗
- Paper High prevalence of mutant KRAS in circulating exosome-derived DNA from early-stage pancreatic cancer patients. DOI 10.1093/annonc/mdx004↗
- Hector A. Alvarez · 3 shared papers, 2017–2018 · strength 0.17
- Paper Surfaceome profiling enables isolation of cancer-specific exosomal cargo in liquid biopsies from pancreatic cancer pati… DOI 10.1093/annonc/mdx542↗
- Paper Dynamic changes during the treatment of pancreatic cancer. DOI 10.18632/oncotarget.24483↗
- Paper High prevalence of mutant KRAS in circulating exosome-derived DNA from early-stage pancreatic cancer patients. DOI 10.1093/annonc/mdx004↗
- Jun Zhao · 1 shared paper, 2021 · strength 0.12
- Laura R. Prakash · 1 shared paper, 2021 · strength 0.12
- Ching-Wei David Tzeng · 1 shared paper, 2021 · strength 0.12
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.