ResearcherFred Hutch Cancer CenterActive in the last 2 years
Paolo Provenzano
Matched on ORCID iD 0000-0002-9313-1370; OpenAlex author ID A5070953226, A5160284263; NIH RePORTER profile ID 8646666. Sources: OpenAlex.
Their work
- Papers
- 14 (first author 0, last author 7)
- Trials
- 0
- Grants
- 6
- Labs led
- 2
- Collaborators
- 43
2011: 1 grant · 2012: 1 paper · 2013: none · 2014: none · 2015: none · 2016: 1 paper · 2017: none · 2018: 1 paper, 1 grant · 2019: 2 papers · 2020: 1 paper, 1 grant · 2021: none · 2022: 2 papers · 2023: 1 paper · 2024: 1 paper · 2025: 2 papers, 1 grant · 2026: 2 papers, 2 grants
Papers 14
- Decoding spatiotemporal fibrotic and cellular immunosuppression of therapeutic T cells in live pancreatic ductal adenocarcinoma. Last author DOI 10.1038/s44320-026-00243-4↗
- Promoting tumor antigen presentation unleashes therapeutic efficacy of Tgfbr2-deficient TCR-engineered T cells in pancreatic ductal adenocarcinoma. Co-author DOI 10.1136/jitc-2026-015489↗
- Computationally Enabled Polychromatic Polarized Imaging Enables Mapping of Matrix Architectures that Promote Pancreatic Ductal Adenocarcinoma Dissemination. Last author DOI 10.1016/j.ajpath.2025.04.017↗
- Extracellular matrix architecture promotes immunosuppressive microenvironments in pancreatic cancer. Last author DOI 10.1016/j.matbio.2025.09.004↗
- Senescence Defines a Distinct Subset of Myofibroblasts That Orchestrates Immunosuppression in Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-23-0428↗
- Protocol to culture and image pancreatic ductal adenocarcinoma tumor slices to study T cell migration. Last author DOI 10.1016/j.xpro.2023.102135↗
- Stromal architecture directs early dissemination in pancreatic ductal adenocarcinoma. Last author DOI 10.1172/jci.insight.150330↗
- Targeting TNF-α-producing macrophages activates antitumor immunity in pancreatic cancer via IL-33 signaling. Last author DOI 10.1172/jci.insight.153242↗
- Loss of HIF1A From Pancreatic Cancer Cells Increases Expression of PPP1R1B and Degradation of p53 to Promote Invasion and Metastasis. Co-author DOI 10.1053/j.gastro.2020.07.046↗
- Antifibrotic Therapy Disrupts Stromal Barriers and Modulates the Immune Landscape in Pancreatic Ductal Adenocarcinoma. Last author DOI 10.1158/0008-5472.can-18-1334↗
- Non-Invasive Monitoring of Stromal Biophysics with Targeted Depletion of Hyaluronan in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3390/cancers11060772↗
- Physical and Chemical Enhancement of and Adaptive Resistance to Irreversible Electroporation of Pancreatic Cancer. Co-author DOI 10.1007/s10439-017-1932-3↗
- Interstitial Pressure in Pancreatic Ductal Adenocarcinoma Is Dominated by a Gel-Fluid Phase. Co-author DOI 10.1016/j.bpj.2016.03.040↗
- Enzymatic targeting of the stroma ablates physical barriers to treatment of pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.ccr.2012.01.007↗
Grants 6
- Stomal Senescence Regulator of Tumor Immunity and Progression R01CA286615↗
- Research Testbed 1 U54CA268069↗
- Project 1 P01CA254849↗
- Project 1: Physical modeling and dynamics of tumor cell migration mechanics U54CA210190↗
- Stellate cells and their progenitor precursors in pancreas cancer progression R01CA181385↗
- Dissecting the roles of stellate cells in pancreas cancer progression R21CA152249↗
Closest collaborators
The 25 strongest of 43 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- David G DeNardo · 2 shared papers, 1 shared grant, 2022–2026 · strength 1.57
- Grant Stomal Senescence Regulator of Tumor Immunity and Progression R01CA286615↗
- Paper Senescence Defines a Distinct Subset of Myofibroblasts That Orchestrates Immunosuppression in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-0428↗
- Paper Stromal architecture directs early dissemination in pancreatic ductal adenocarcinoma. DOI 10.1172/jci.insight.150330↗
- David J. Odde · 2 shared grants, 2020–2025 · strength 1.21
- Guhan Qian · 4 shared papers, 2024–2026 · strength 1.09
- Paper Decoding spatiotemporal fibrotic and cellular immunosuppression of therapeutic T cells in live pancreatic ductal adenoc… DOI 10.1038/s44320-026-00243-4↗
- Paper Computationally Enabled Polychromatic Polarized Imaging Enables Mapping of Matrix Architectures that Promote Pancreatic… DOI 10.1016/j.ajpath.2025.04.017↗
- Paper Promoting tumor antigen presentation unleashes therapeutic efficacy of Tgfbr2-deficient TCR-engineered T cells in pancr… DOI 10.1136/jitc-2026-015489↗
- David Masopust · 1 shared paper, 1 shared grant, 2025–2026 · strength 0.96
- Mackenzie Kate Callaway · 3 shared papers, 2022–2025 · strength 0.81
- Paper Extracellular matrix architecture promotes immunosuppressive microenvironments in pancreatic cancer. DOI 10.1016/j.matbio.2025.09.004↗
- Paper Protocol to culture and image pancreatic ductal adenocarcinoma tumor slices to study T cell migration. DOI 10.1016/j.xpro.2023.102135↗
- Paper Stromal architecture directs early dissemination in pancreatic ductal adenocarcinoma. DOI 10.1172/jci.insight.150330↗
- David Andrew Largaespada · 1 shared grant, 2025 · strength 0.73
- Grant Project 1 P01CA254849↗
- Kevin W. Eliceiri · 2 shared papers, 2025–2026 · strength 0.69
- Paper Decoding spatiotemporal fibrotic and cellular immunosuppression of therapeutic T cells in live pancreatic ductal adenoc… DOI 10.1038/s44320-026-00243-4↗
- Paper Computationally Enabled Polychromatic Polarized Imaging Enables Mapping of Matrix Architectures that Promote Pancreatic… DOI 10.1016/j.ajpath.2025.04.017↗
- Ingunn M. Stromnes · 2 shared papers, 2026 · strength 0.61
- Paper Decoding spatiotemporal fibrotic and cellular immunosuppression of therapeutic T cells in live pancreatic ductal adenoc… DOI 10.1038/s44320-026-00243-4↗
- Paper Promoting tumor antigen presentation unleashes therapeutic efficacy of Tgfbr2-deficient TCR-engineered T cells in pancr… DOI 10.1136/jitc-2026-015489↗
- Sunil R. Hingorani · 4 shared papers, 1 shared grant, 2011–2026 · strength 0.53
- Paper Promoting tumor antigen presentation unleashes therapeutic efficacy of Tgfbr2-deficient TCR-engineered T cells in pancr… DOI 10.1136/jitc-2026-015489↗
- Grant Dissecting the roles of stellate cells in pancreas cancer progression R21CA152249↗
- Paper Non-Invasive Monitoring of Stromal Biophysics with Targeted Depletion of Hyaluronan in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/cancers11060772↗
- Nelson J. Rodríguez-Merced · 2 shared papers, 2022–2023 · strength 0.44
- Ajay Kumar Dixit · 2 shared papers, 2020–2022 · strength 0.26
- Paper Targeting TNF-α-producing macrophages activates antitumor immunity in pancreatic cancer via IL-33 signaling. DOI 10.1172/jci.insight.153242↗
- Paper Loss of HIF1A From Pancreatic Cancer Cells Increases Expression of PPP1R1B and Degradation of p53 to Promote Invasion a… DOI 10.1053/j.gastro.2020.07.046↗
- Kianna Y. Elahi-Gedwillo · 2 shared papers, 2018–2019 · strength 0.23
- Paper Antifibrotic Therapy Disrupts Stromal Barriers and Modulates the Immune Landscape in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/0008-5472.can-18-1334↗
- Paper Physical and Chemical Enhancement of and Adaptive Resistance to Irreversible Electroporation of Pancreatic Cancer. DOI 10.1007/s10439-017-1932-3↗
- Adam L. Burrack · 1 shared paper, 2026 · strength 0.23
- Zoe C. Schmiechen · 1 shared paper, 2026 · strength 0.23
- Grant H. Hickok · 1 shared paper, 2026 · strength 0.23
- Alexander K. Tsai · 1 shared paper, 2026 · strength 0.23
- Philip D. Greenberg · 1 shared paper, 2026 · strength 0.23
- Huocong Huang · 1 shared paper, 2022 · strength 0.17
- Rolf A. Brekken · 1 shared paper, 2022 · strength 0.17
- Varintra E. Lander · 1 shared paper, 2024 · strength 0.17
- John M. Baer · 1 shared paper, 2024 · strength 0.17
- Blake E. Sells · 1 shared paper, 2024 · strength 0.17
- Liang‐I Kang · 1 shared paper, 2024 · strength 0.17
- Jad I. Belle · 1 shared paper, 2024 · strength 0.17
- Hyun Jee Kim · 1 shared paper, 2024 · strength 0.17
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.