ResearcherUniversity of MiamiNo recent work listed
Kousik Kesh
Matched on OpenAlex author ID A5009648827. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 19
2018: 2 papers · 2019: 2 papers · 2020: 3 papers · 2021: 1 paper · 2022: 1 paper
Papers 9
- Obesity enriches for tumor protective microbial metabolites and treatment refractory cells to confer therapy resistance in PDAC. Co-author DOI 10.1080/19490976.2022.2096328↗
- Hypoxia-Driven Oncometabolite L-2HG Maintains Stemness-Differentiation Balance and Facilitates Immune Evasion in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-20-2562↗
- Microbial dysbiosis and polyamine metabolism as predictive markers for early detection of pancreatic cancer. First author DOI 10.1093/carcin/bgz116↗
- Targeting tumor-intrinsic hexosamine biosynthesis sensitizes pancreatic cancer to anti-PD1 therapy. Co-author DOI 10.1172/jci127515↗
- Type 2 diabetes induced microbiome dysbiosis is associated with therapy resistance in pancreatic adenocarcinoma. Co-author DOI 10.1186/s12934-020-01330-3↗
- Targeting tumor intrinsic metabolic node in pancreatic cancer causes tumor regression,remodels extracellular matrix and sensitizes to anti-PD1 therapy Co-author DOI 10.1016/j.pan.2019.05.430↗
- O-GlcNAc modification of Sox2 regulates self-renewal in pancreatic cancer by promoting its stability. Co-author DOI 10.7150/thno.32615↗
- Metastasis and chemoresistance in CD133 expressing pancreatic cancer cells are dependent on their lipid raft integrity. Co-author DOI 10.1016/j.canlet.2018.09.028↗
- Inactivation of Cancer-Associated-Fibroblasts Disrupts Oncogenic Signaling in Pancreatic Cancer Cells and Promotes Its Regression. Co-author DOI 10.1158/0008-5472.can-17-2320↗
Closest collaborators
The 19 strongest of 19 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Sulagna Banerjee · 9 shared papers, 2018–2022 · strength 0.95
- Paper Type 2 diabetes induced microbiome dysbiosis is associated with therapy resistance in pancreatic adenocarcinoma. DOI 10.1186/s12934-020-01330-3↗
- Paper Obesity enriches for tumor protective microbial metabolites and treatment refractory cells to confer therapy resistance… DOI 10.1080/19490976.2022.2096328↗
- Paper Microbial dysbiosis and polyamine metabolism as predictive markers for early detection of pancreatic cancer. DOI 10.1093/carcin/bgz116↗
- Vineet Kumar Gupta · 7 shared papers, 2018–2022 · strength 0.69
- Paper Obesity enriches for tumor protective microbial metabolites and treatment refractory cells to confer therapy resistance… DOI 10.1080/19490976.2022.2096328↗
- Paper Hypoxia-Driven Oncometabolite L-2HG Maintains Stemness-Differentiation Balance and Facilitates Immune Evasion in Pancre… DOI 10.1158/0008-5472.can-20-2562↗
- Paper Targeting tumor-intrinsic hexosamine biosynthesis sensitizes pancreatic cancer to anti-PD1 therapy. DOI 10.1172/jci127515↗
- Santanu Banerjee · 5 shared papers, 2018–2022 · strength 0.57
- Paper Type 2 diabetes induced microbiome dysbiosis is associated with therapy resistance in pancreatic adenocarcinoma. DOI 10.1186/s12934-020-01330-3↗
- Paper Obesity enriches for tumor protective microbial metabolites and treatment refractory cells to confer therapy resistance… DOI 10.1080/19490976.2022.2096328↗
- Paper Microbial dysbiosis and polyamine metabolism as predictive markers for early detection of pancreatic cancer. DOI 10.1093/carcin/bgz116↗
- Nikita Sharma · 6 shared papers, 2018–2021 · strength 0.54
- Paper Hypoxia-Driven Oncometabolite L-2HG Maintains Stemness-Differentiation Balance and Facilitates Immune Evasion in Pancre… DOI 10.1158/0008-5472.can-20-2562↗
- Paper Targeting tumor-intrinsic hexosamine biosynthesis sensitizes pancreatic cancer to anti-PD1 therapy. DOI 10.1172/jci127515↗
- Paper Targeting tumor intrinsic metabolic node in pancreatic cancer causes tumor regression,remodels extracellular matrix and… DOI 10.1016/j.pan.2019.05.430↗
- Ashok Kumar Saluja · 6 shared papers, 2018–2021 · strength 0.54
- Paper Hypoxia-Driven Oncometabolite L-2HG Maintains Stemness-Differentiation Balance and Facilitates Immune Evasion in Pancre… DOI 10.1158/0008-5472.can-20-2562↗
- Paper Targeting tumor-intrinsic hexosamine biosynthesis sensitizes pancreatic cancer to anti-PD1 therapy. DOI 10.1172/jci127515↗
- Paper Targeting tumor intrinsic metabolic node in pancreatic cancer causes tumor regression,remodels extracellular matrix and… DOI 10.1016/j.pan.2019.05.430↗
- Vikas Dudeja · 5 shared papers, 2018–2020 · strength 0.43
- Paper Targeting tumor-intrinsic hexosamine biosynthesis sensitizes pancreatic cancer to anti-PD1 therapy. DOI 10.1172/jci127515↗
- Paper Targeting tumor intrinsic metabolic node in pancreatic cancer causes tumor regression,remodels extracellular matrix and… DOI 10.1016/j.pan.2019.05.430↗
- Paper O-GlcNAc modification of Sox2 regulates self-renewal in pancreatic cancer by promoting its stability. DOI 10.7150/thno.32615↗
- Roberto Méndez · 3 shared papers, 2020–2022 · strength 0.41
- Paper Type 2 diabetes induced microbiome dysbiosis is associated with therapy resistance in pancreatic adenocarcinoma. DOI 10.1186/s12934-020-01330-3↗
- Paper Obesity enriches for tumor protective microbial metabolites and treatment refractory cells to confer therapy resistance… DOI 10.1080/19490976.2022.2096328↗
- Paper Microbial dysbiosis and polyamine metabolism as predictive markers for early detection of pancreatic cancer. DOI 10.1093/carcin/bgz116↗
- Bhuwan Giri · 4 shared papers, 2018–2020 · strength 0.35
- Paper Targeting tumor-intrinsic hexosamine biosynthesis sensitizes pancreatic cancer to anti-PD1 therapy. DOI 10.1172/jci127515↗
- Paper Targeting tumor intrinsic metabolic node in pancreatic cancer causes tumor regression,remodels extracellular matrix and… DOI 10.1016/j.pan.2019.05.430↗
- Paper O-GlcNAc modification of Sox2 regulates self-renewal in pancreatic cancer by promoting its stability. DOI 10.7150/thno.32615↗
- Nipun B. Merchant · 2 shared papers, 2020–2022 · strength 0.26
- Paper Obesity enriches for tumor protective microbial metabolites and treatment refractory cells to confer therapy resistance… DOI 10.1080/19490976.2022.2096328↗
- Paper Microbial dysbiosis and polyamine metabolism as predictive markers for early detection of pancreatic cancer. DOI 10.1093/carcin/bgz116↗
- Beatriz Mateo-Victoriano · 2 shared papers, 2021–2022 · strength 0.25
- Paper Obesity enriches for tumor protective microbial metabolites and treatment refractory cells to confer therapy resistance… DOI 10.1080/19490976.2022.2096328↗
- Paper Hypoxia-Driven Oncometabolite L-2HG Maintains Stemness-Differentiation Balance and Facilitates Immune Evasion in Pancre… DOI 10.1158/0008-5472.can-20-2562↗
- Patricia Dauer · 3 shared papers, 2018–2019 · strength 0.24
- Paper O-GlcNAc modification of Sox2 regulates self-renewal in pancreatic cancer by promoting its stability. DOI 10.7150/thno.32615↗
- Paper Inactivation of Cancer-Associated-Fibroblasts Disrupts Oncogenic Signaling in Pancreatic Cancer Cells and Promotes Its … DOI 10.1158/0008-5472.can-17-2320↗
- Paper Metastasis and chemoresistance in CD133 expressing pancreatic cancer cells are dependent on their lipid raft integrity. DOI 10.1016/j.canlet.2018.09.028↗
- Anthony R. Ferrantella · 2 shared papers, 2019–2020 · strength 0.19
- Selwyn Maurice Vickers · 2 shared papers, 2018–2019 · strength 0.16
- Jashodeep Datta · 1 shared paper, 2022 · strength 0.15
- Florencia M. McAllister · 1 shared paper, 2020 · strength 0.12
- Sai Sundeep Kollala · 1 shared paper, 2021 · strength 0.11
- Pankaj Kumar Singh · 1 shared paper, 2021 · strength 0.11
- Xianda Zhao · 1 shared paper, 2018 · strength 0.08
- Alice Nomura · 1 shared paper, 2018 · strength 0.08
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.