ResearcherHuntsman Cancer InstituteNo recent work listed

Soledad A. Camolotto

Verified by OpenAlex author ID (good confidence, 0.85), identity_conf@1As of 8 November 2026

Matched on OpenAlex author ID A5021530580. Sources: OpenAlex.

Their work

Papers: 2
Trials: 0
Grants: 0
Labs led: 0
Collaborators: 4

Papers, trials and grants by year

2018: 1 paper · 2019: none · 2020: none · 2021: 1 paper

Papers 2

Reciprocal regulation of pancreatic ductal adenocarcinoma growth and molecular subtype by HNF4α and SIX1/4. Co-author DOI 10.1136/gutjnl-2020-321316 ↗
The role of lineage specifiers in pancreatic ductal adenocarcinoma. Co-author DOI 10.21037/jgo.2018.05.04 ↗

Closest collaborators

The 4 strongest of 4 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Eric Lee Snyder · 2 shared papers, 2018–2021 · strength 0.26
- Paper The role of lineage specifiers in pancreatic ductal adenocarcinoma. DOI 10.21037/jgo.2018.05.04 ↗
- Paper Reciprocal regulation of pancreatic ductal adenocarcinoma growth and molecular subtype by HNF4α and SIX1/4. DOI 10.1136/gutjnl-2020-321316 ↗
Jill Shea · 1 shared paper, 2021 · strength 0.12
- Paper Reciprocal regulation of pancreatic ductal adenocarcinoma growth and molecular subtype by HNF4α and SIX1/4. DOI 10.1136/gutjnl-2020-321316 ↗
Luke A. Torre-Healy · 1 shared paper, 2021 · strength 0.12
- Paper Reciprocal regulation of pancreatic ductal adenocarcinoma growth and molecular subtype by HNF4α and SIX1/4. DOI 10.1136/gutjnl-2020-321316 ↗
Richard A. Moffitt · 1 shared paper, 2021 · strength 0.12
- Paper Reciprocal regulation of pancreatic ductal adenocarcinoma growth and molecular subtype by HNF4α and SIX1/4. DOI 10.1136/gutjnl-2020-321316 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.