Pancreatic Cancer Research

ResearcherUniversity of VeronaActive in the last 2 years

Raffaella Pacchiana

Matched on OpenAlex author ID A5029880735. Sources: OpenAlex.

Their work

Papers
10 (first author 2, last author 0)
Trials
0
Grants
0
Labs led
0
Collaborators
28
Papers, trials and grants by year

2018: 2 papers · 2019: none · 2020: 1 paper · 2021: none · 2022: 2 papers · 2023: 1 paper · 2024: none · 2025: 1 paper · 2026: 3 papers

Papers 10

  1. First-in-Class Covalent Inhibitors of PFKFB3: Discovery and Characterization in PDAC Models. Co-author DOI 10.1021/acs.jmedchem.6c00235↗
  2. Mutant p53 affects the mitochondrial proteome, promoting mitochondrial fragmentation and OXPHOS in pancreatic ductal adenocarcinoma cells. First author DOI 10.1111/febs.70223↗
  3. Metabolic plasticity underlies ferroptosis resistance driven by mutant p53 in pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s12943-026-02746-y↗
  4. Chemotherapy enhances HMGA1 secretion through the mutant p53-CK2 axis in pancreatic ductal adenocarcinoma cells. Co-author DOI 10.1038/s41419-025-08082-1↗
  5. Discovery of a spirocyclic 3-bromo-4,5-dihydroisoxazole covalent inhibitor of hGAPDH with antiproliferative activity against pancreatic cancer cells. Co-author DOI 10.1016/j.ejmech.2023.115286↗
  6. Tumor Suppressor Role of Wild-Type P53-Dependent Secretome and Its Proteomic Identification in PDAC. Co-author DOI 10.3390/biom12020305↗
  7. 3-Bromo-Isoxazoline Derivatives Inhibit GAPDH Enzyme in PDAC Cells Triggering Autophagy and Apoptotic Cell Death. Co-author DOI 10.3390/cancers14133153↗
  8. Extracellular Matrix Composition Modulates the Responsiveness of Differentiated and Stem Pancreatic Cancer Cells to Lipophilic Derivate of Gemcitabine. Co-author DOI 10.3390/ijms22010029↗
  9. Trichostatin A alters cytoskeleton and energy metabolism of pancreatic adenocarcinoma cells: An in depth proteomic study. Co-author DOI 10.1002/jcb.26436↗
  10. Mutant p53 prevents GAPDH nuclear translocation in pancreatic cancer cells favoring glycolysis and 2-deoxyglucose sensitivity. First author DOI 10.1016/j.bbamcr.2018.10.005↗

Closest collaborators

The 25 strongest of 28 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Massimo Donadelli · 10 shared papers, 2018–2026 · strength 1.67
  2. Adriana Celesia · 4 shared papers, 2025–2026 · strength 0.95
  3. Alessandra Fiore · 4 shared papers, 2025–2026 · strength 0.95
  4. Federica Danzi · 3 shared papers, 2025–2026 · strength 0.72
  5. Daniela Cecconi · 4 shared papers, 2018–2026 · strength 0.69
  6. Paola Conti · 4 shared papers, 2018–2026 · strength 0.63
  7. Maria Poles · 2 shared papers, 2026 · strength 0.53
  8. Claudio Luchini · 2 shared papers, 2025–2026 · strength 0.44
  9. Giovanna Butera · 3 shared papers, 2018–2025 · strength 0.41
  10. Chiara Borsari · 2 shared papers, 2023–2026 · strength 0.41
  11. Jessica Brandi · 3 shared papers, 2018–2025 · strength 0.41
  12. Elisa Dalla Pozza · 3 shared papers, 2018–2022 · strength 0.31
  13. Stefania Forciniti · 2 shared papers, 2020–2022 · strength 0.23
  14. Ilaria Dando · 2 shared papers, 2020–2022 · strength 0.23
  15. Emílio Marengo · 2 shared papers, 2018–2022 · strength 0.22
  16. Antonio Pea · 1 shared paper, 2025 · strength 0.19
  17. Costas Andreas Lyssiotis · 1 shared paper, 2025 · strength 0.19
  18. Aldo Scarpa · 1 shared paper, 2025 · strength 0.19
  19. Roberto Salvia · 1 shared paper, 2025 · strength 0.19
  20. Andrea Galbiati · 1 shared paper, 2023 · strength 0.18
  21. Maria Raffaella Greco · 1 shared paper, 2020 · strength 0.09
  22. Tiago M. A. Carvalho · 1 shared paper, 2020 · strength 0.09
  23. Stephan J. Reshkin · 1 shared paper, 2020 · strength 0.09
  24. Marta Palmieri · 1 shared paper, 2020 · strength 0.09
  25. Rosa Angela Cardone · 1 shared paper, 2020 · strength 0.09

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.