ResearcherUniversity of HelsinkiNo recent work listed

Leena Arpalahti

Verified by OpenAlex author ID (good confidence, 0.85), identity_conf@1As of 8 November 2026

Matched on OpenAlex author ID A5031372005. Sources: OpenAlex.

Their work

Papers: 2
Trials: 0
Grants: 0
Labs led: 0
Collaborators: 4

Papers, trials and grants by year

2017: 1 paper · 2018: none · 2019: none · 2020: 1 paper

Papers 2

Proteostasis Dysregulation in Pancreatic Cancer. Co-author DOI 10.1007/978-3-030-38266-7_4 ↗
Nuclear ubiquitin C-terminal hydrolase L5 expression associates with increased patient survival in pancreatic ductal adenocarcinoma. Co-author DOI 10.1177/1010428317710411 ↗

Closest collaborators

The 4 strongest of 4 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Carina I. Holmberg · 2 shared papers, 2017–2020 · strength 0.27
- Paper Proteostasis Dysregulation in Pancreatic Cancer. DOI 10.1007/978-3-030-38266-7_4 ↗
- Paper Nuclear ubiquitin C-terminal hydrolase L5 expression associates with increased patient survival in pancreatic ductal ad… DOI 10.1177/1010428317710411 ↗
Caj H. Haglund · 2 shared papers, 2017–2020 · strength 0.27
- Paper Proteostasis Dysregulation in Pancreatic Cancer. DOI 10.1007/978-3-030-38266-7_4 ↗
- Paper Nuclear ubiquitin C-terminal hydrolase L5 expression associates with increased patient survival in pancreatic ductal ad… DOI 10.1177/1010428317710411 ↗
Kapo Saukkonen · 1 shared paper, 2017 · strength 0.08
- Paper Nuclear ubiquitin C-terminal hydrolase L5 expression associates with increased patient survival in pancreatic ductal ad… DOI 10.1177/1010428317710411 ↗
Hanna E. Seppänen · 1 shared paper, 2017 · strength 0.08
- Paper Nuclear ubiquitin C-terminal hydrolase L5 expression associates with increased patient survival in pancreatic ductal ad… DOI 10.1177/1010428317710411 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.