ResearcherSouthern University of Science and TechnologyActive in the last 2 years
Stefania Militi
Matched on OpenAlex author ID A5041155406. Sources: OpenAlex.
Their work
- Papers
- 5
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 5
2023: 2 papers · 2024: 2 papers · 2025: 1 paper
Papers 5
- GluProRS Inhibition Abolishes Cancer Stem Cells in Pancreatic Cancer by Inducing the Integrated Stress Response and Countering TGFβ/Activin-SMAD2/3-HIF1A Co-author DOI 10.21203/rs.3.rs-6672561/v1↗
- BRD9-SMAD2/3 Orchestrates Stemness and Tumorigenesis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1053/j.gastro.2023.09.021↗
- Quantitative secretomics identifies ligand-receptor pairs in pancreatic cancer that mediate bidirectional crosstalk between cancer stem cells and nociceptors Co-author DOI 10.21203/rs.3.rs-3994602/v1↗
- KMT2A associates with PHF5A-PHF14-HMG20A-RAI1 subcomplex in pancreatic cancer stem cells and epigenetically regulates their characteristics. Co-author DOI 10.1038/s41467-023-41297-4↗
- BAZ2A-SUV39H1-SMAD2/3-OCT4 regulates stemness, cell cycle exit and chemoresistance in pancreatic ductal adenocarcinoma Co-author DOI 10.21203/rs.3.rs-3570401/v1↗
Closest collaborators
The 5 strongest of 5 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Siim Pauklin · 5 shared papers, 2023–2025 · strength 0.94
- Paper GluProRS Inhibition Abolishes Cancer Stem Cells in Pancreatic Cancer by Inducing the Integrated Stress Response and Cou… DOI 10.21203/rs.3.rs-6672561/v1↗
- Paper Quantitative secretomics identifies ligand-receptor pairs in pancreatic cancer that mediate bidirectional crosstalk bet… DOI 10.21203/rs.3.rs-3994602/v1↗
- Paper BAZ2A-SUV39H1-SMAD2/3-OCT4 regulates stemness, cell cycle exit and chemoresistance in pancreatic ductal adenocarcinoma DOI 10.21203/rs.3.rs-3570401/v1↗
- Chao‐Hui Chang · 3 shared papers, 2024–2025 · strength 0.58
- Paper GluProRS Inhibition Abolishes Cancer Stem Cells in Pancreatic Cancer by Inducing the Integrated Stress Response and Cou… DOI 10.21203/rs.3.rs-6672561/v1↗
- Paper Quantitative secretomics identifies ligand-receptor pairs in pancreatic cancer that mediate bidirectional crosstalk bet… DOI 10.21203/rs.3.rs-3994602/v1↗
- Paper BRD9-SMAD2/3 Orchestrates Stemness and Tumorigenesis in Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2023.09.021↗
- Mai Abdel Mouti · 3 shared papers, 2023–2024 · strength 0.52
- Paper BAZ2A-SUV39H1-SMAD2/3-OCT4 regulates stemness, cell cycle exit and chemoresistance in pancreatic ductal adenocarcinoma DOI 10.21203/rs.3.rs-3570401/v1↗
- Paper KMT2A associates with PHF5A-PHF14-HMG20A-RAI1 subcomplex in pancreatic cancer stem cells and epigenetically regulates t… DOI 10.1038/s41467-023-41297-4↗
- Paper BRD9-SMAD2/3 Orchestrates Stemness and Tumorigenesis in Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2023.09.021↗
- Chang‐Il Hwang · 1 shared paper, 2023 · strength 0.17
- Yuliang Feng · 1 shared paper, 2024 · strength 0.15
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.