ResearcherThe First Hospital of ChangshaActive in the last 2 years
Shuangxi Gong
Matched on OpenAlex author ID A5044725722. Sources: OpenAlex.
Their work
- Papers
- 2
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 3
2022: 1 paper · 2023: none · 2024: none · 2025: 1 paper
Papers 2
- [Retracted] SIRT6 promotes ferroptosis and attenuates glycolysis in pancreatic cancer through regulation of the NF‑κB pathway. Co-author DOI 10.3892/etm.2024.12751↗
- SIRT6 promotes ferroptosis and attenuates glycolysis in pancreatic cancer through regulation of the NF-κB pathway. Co-author DOI 10.3892/etm.2022.11430↗
Closest collaborators
The 3 strongest of 3 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Zhen Luo · 2 shared papers, 2022–2025 · strength 0.46
- Paper [Retracted] SIRT6 promotes ferroptosis and attenuates glycolysis in pancreatic cancer through regulation of the NF‑κB p… DOI 10.3892/etm.2024.12751↗
- Paper SIRT6 promotes ferroptosis and attenuates glycolysis in pancreatic cancer through regulation of the NF-κB pathway. DOI 10.3892/etm.2022.11430↗
- Jian Li · 2 shared papers, 2022–2025 · strength 0.46
- Paper [Retracted] SIRT6 promotes ferroptosis and attenuates glycolysis in pancreatic cancer through regulation of the NF‑κB p… DOI 10.3892/etm.2024.12751↗
- Paper SIRT6 promotes ferroptosis and attenuates glycolysis in pancreatic cancer through regulation of the NF-κB pathway. DOI 10.3892/etm.2022.11430↗
- Qinghua Yin · 2 shared papers, 2022–2025 · strength 0.46
- Paper [Retracted] SIRT6 promotes ferroptosis and attenuates glycolysis in pancreatic cancer through regulation of the NF‑κB p… DOI 10.3892/etm.2024.12751↗
- Paper SIRT6 promotes ferroptosis and attenuates glycolysis in pancreatic cancer through regulation of the NF-κB pathway. DOI 10.3892/etm.2022.11430↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.