Pancreatic Cancer Research

ResearcherUniversity Hospital Schleswig-HolsteinActive in the last 2 years

Hendrik Lehnert

Matched on OpenAlex author ID A5079250860. Sources: OpenAlex.

Their work

Papers
12 (first author 1, last author 1)
Trials
0
Grants
0
Labs led
0
Collaborators
17
Papers, trials and grants by year

2011: 1 paper · 2012: none · 2013: none · 2014: none · 2015: 1 paper · 2016: 2 papers · 2017: none · 2018: none · 2019: 1 paper · 2020: none · 2021: 2 papers · 2022: 1 paper · 2023: 1 paper · 2024: 1 paper · 2025: 1 paper · 2026: 1 paper

Papers 12

  1. Intrapancreatic Fat and Risk of Pancreatic Ductal Adenocarcinoma-Pathophysiology, Clinical Implications, and Future Directions. Last author DOI 10.3390/ijms27187991↗
  2. Regulation of epithelial-mesenchymal plasticity in pancreatic ductal adenocarcinoma: Role of key molecules in tumor differentiation and therapy. Co-author DOI 10.1016/j.ejca.2025.115561↗
  3. RAC1b Collaborates with TAp73α-SMAD4 Signaling to Induce Biglycan Expression and Inhibit Basal and TGF-β-Driven Cell Motility in Human Pancreatic Cancer. Co-author DOI 10.3390/biomedicines12010199↗
  4. TAp73 Inhibits EMT and Cell Migration in Pancreatic Cancer Cells through Promoting SMAD4 Expression and SMAD4-Dependent Inhibition of ERK Activation. Co-author DOI 10.3390/cancers15153791↗
  5. Suppressive Role of ACVR1/ALK2 in Basal and TGFβ1-Induced Cell Migration in Pancreatic Ductal Adenocarcinoma Cells and Identification of a Self-Perpetuating Au… Co-author DOI 10.3390/biomedicines10102640↗
  6. A Comparative Endocrine Trans-Differentiation Approach to Pancreatic Ductal Adenocarcinoma Cells with Different EMT Phenotypes Identifies Quasi-Mesenchymal Tum… Co-author DOI 10.3390/cancers13184663↗
  7. Activation of a Ductal-to-Endocrine Transdifferentiation Transcriptional Program in the Pancreatic Cancer Cell Line PANC-1 Is Controlled by RAC1 and RAC1b thro… Co-author DOI 10.3390/cancers13215541↗
  8. Negative Control of Cell Migration by Rac1b in Highly Metastatic Pancreatic Cancer Cells Is Mediated by Sequential Induction of Nonactivated Smad3 and Biglycan. First author DOI 10.3390/cancers11121959↗
  9. Proteinase-activated receptor 2 promotes TGF-β-dependent cell motility in pancreatic cancer cells by sustaining expression of the TGF-β type I receptor ALK5. Co-author DOI 10.18632/oncotarget.9600↗
  10. Proteinase-Activated Receptor 2 Is a Novel Regulator of TGF-β Signaling in Pancreatic Cancer. Co-author DOI 10.3390/jcm5120111↗
  11. Dasatinib blocks transcriptional and promigratory responses to transforming growth factor-beta in pancreatic adenocarcinoma cells through inhibition of Smad si… Co-author DOI 10.1186/s12943-015-0468-0↗
  12. Differential roles of Smad2 and Smad3 in the regulation of TGF-β1-mediated growth inhibition and cell migration in pancreatic ductal adenocarcinoma cells: cont… Co-author DOI 10.1186/1476-4598-10-67↗

Closest collaborators

The 17 strongest of 17 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Hendrik Ungefroren · 11 shared papers, 2011–2025 · strength 1.50
  2. Jens Uwe Marquardt · 5 shared papers, 2022–2026 · strength 1.26
  3. Rüdiger Braun · 6 shared papers, 2021–2025 · strength 1.15
  4. Ulrich Friedrich Wellner · 5 shared papers, 2021–2024 · strength 0.88
  5. Svenja Meyhöfer · 1 shared paper, 2026 · strength 0.36
  6. Holger Kalthoff · 1 shared paper, 2025 · strength 0.27
  7. Qi Ling · 1 shared paper, 2025 · strength 0.27
  8. Paula Marie Schmidtlein · 2 shared papers, 2021 · strength 0.25
  9. Hannah Otterbein · 1 shared paper, 2019 · strength 0.16
  10. Franziska Zeeh · 2 shared papers, 2016 · strength 0.11
  11. Utz Settmacher · 2 shared papers, 2016 · strength 0.11
  12. David P. Witte · 2 shared papers, 2016 · strength 0.11
  13. Tobias Bartscht · 1 shared paper, 2015 · strength 0.05
  14. Dirk Rades · 1 shared paper, 2015 · strength 0.05
  15. Evelin Grage‐Griebenow · 1 shared paper, 2016 · strength 0.05
  16. Fred Fändrich · 1 shared paper, 2011 · strength 0.03
  17. Susanne Sebens · 1 shared paper, 2011 · strength 0.03

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.