ResearcherWenzhou Medical UniversityActive in the last 2 years
Jiayue Zeng
Matched on OpenAlex author ID A5100591042.
Their work
- Papers
- 2 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Collaborators
- 3
2025: 2 papers
Papers 2
- Sialyltransferase ST3GAL4 directs a dual mechanism to promote pancreatic ductal adenocarcinoma progression by regulating endoplasmic reticulum stress and mitoc… First author DOI 10.1016/j.bbadis.2025.167900↗
- GALNT6 dual regulates innate immunity STING signaling and PD-L1 expression to promote immune evasion in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.cellsig.2025.111942↗
Closest collaborators
The 3 strongest of 3 ranked by shared papers, trials and grants, with recent and small-team work weighing most.
- Suli Chong · 2 shared papers, 2025
- Paper Sialyltransferase ST3GAL4 directs a dual mechanism to promote pancreatic ductal adenocarcinoma progression by regulatin… DOI 10.1016/j.bbadis.2025.167900↗
- Paper GALNT6 dual regulates innate immunity STING signaling and PD-L1 expression to promote immune evasion in pancreatic duct… DOI 10.1016/j.cellsig.2025.111942↗
- Xiaoming Chen · 2 shared papers, 2025
- Paper Sialyltransferase ST3GAL4 directs a dual mechanism to promote pancreatic ductal adenocarcinoma progression by regulatin… DOI 10.1016/j.bbadis.2025.167900↗
- Paper GALNT6 dual regulates innate immunity STING signaling and PD-L1 expression to promote immune evasion in pancreatic duct… DOI 10.1016/j.cellsig.2025.111942↗
- Sairong Fan · 2 shared papers, 2025
- Paper Sialyltransferase ST3GAL4 directs a dual mechanism to promote pancreatic ductal adenocarcinoma progression by regulatin… DOI 10.1016/j.bbadis.2025.167900↗
- Paper GALNT6 dual regulates innate immunity STING signaling and PD-L1 expression to promote immune evasion in pancreatic duct… DOI 10.1016/j.cellsig.2025.111942↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (the fewest links first, then the links with the most shared work). Built from public records only; no contact details are kept.