ResearcherWuxi People's HospitalActive in the last 2 years
Junli Ding
Matched on OpenAlex author ID A5102491631. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 1, last author 5)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 13
2013: 1 paper · 2014: none · 2015: none · 2016: none · 2017: 1 paper · 2018: none · 2019: 2 papers · 2020: 1 paper · 2021: none · 2022: 1 paper · 2023: 1 paper · 2024: none · 2025: 1 paper
Papers 8
- The role of formin-like protein 1 in pancreatic cancer and its specific effects on immunity. First author DOI 10.1177/03000605251365844↗
- PSMC2 is a Novel Prognostic Biomarker and Predicts Immunotherapeutic Responses: From Pancreatic Cancer to Pan-Cancer. Last author DOI 10.2147/pgpm.s418533↗
- NEUROD1 predicts better prognosis in pancreatic cancer revealed by a TILs-based prognostic signature. Last author DOI 10.3389/fphar.2022.1025921↗
- MicroRNA-23b-3p promotes pancreatic cancer cell tumorigenesis and metastasis via the JAK/PI3K and Akt/NF-κB signaling pathways. Co-author DOI 10.3892/ol.2020.12021↗
- The Importance of a Conjoint Analysis of Tumor-Associated Macrophages and Immune Checkpoints in Pancreatic Cancer. Last author DOI 10.1097/mpa.0000000000001364↗
- PSMC2 is Up-regulated in Pancreatic Cancer and Promotes Cancer Cell Proliferation and Inhibits Apoptosis. Last author DOI 10.7150/jca.27616↗
- Upregulated exosomic miR‑23b‑3p plays regulatory roles in the progression of pancreatic cancer. Co-author DOI 10.3892/or.2017.5919↗
- M2-polarized tumor-associated macrophages promoted epithelial-mesenchymal transition in pancreatic cancer cells, partially through TLR4/IL-10 signaling pathway. Last author DOI 10.1038/labinvest.2013.69↗
Closest collaborators
The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Junying Xu · 4 shared papers, 2013–2023 · strength 0.49
- Paper PSMC2 is a Novel Prognostic Biomarker and Predicts Immunotherapeutic Responses: From Pancreatic Cancer to Pan-Cancer. DOI 10.2147/pgpm.s418533↗
- Paper NEUROD1 predicts better prognosis in pancreatic cancer revealed by a TILs-based prognostic signature. DOI 10.3389/fphar.2022.1025921↗
- Paper The Importance of a Conjoint Analysis of Tumor-Associated Macrophages and Immune Checkpoints in Pancreatic Cancer. DOI 10.1097/mpa.0000000000001364↗
- Jie Mei · 2 shared papers, 2022–2023 · strength 0.34
- Wei Huang · 2 shared papers, 2019–2023 · strength 0.32
- Fangmei An · 3 shared papers, 2017–2020 · strength 0.31
- Paper PSMC2 is Up-regulated in Pancreatic Cancer and Promotes Cancer Cell Proliferation and Inhibits Apoptosis. DOI 10.7150/jca.27616↗
- Paper MicroRNA-23b-3p promotes pancreatic cancer cell tumorigenesis and metastasis via the JAK/PI3K and Akt/NF-κB signaling p… DOI 10.3892/ol.2020.12021↗
- Paper Upregulated exosomic miR‑23b‑3p plays regulatory roles in the progression of pancreatic cancer. DOI 10.3892/or.2017.5919↗
- Jian Liu · 1 shared paper, 2025 · strength 0.30
- Xuejing Yang · 1 shared paper, 2025 · strength 0.30
- Chaoying Liu · 3 shared papers, 2013–2022 · strength 0.29
- Paper NEUROD1 predicts better prognosis in pancreatic cancer revealed by a TILs-based prognostic signature. DOI 10.3389/fphar.2022.1025921↗
- Paper The Importance of a Conjoint Analysis of Tumor-Associated Macrophages and Immune Checkpoints in Pancreatic Cancer. DOI 10.1097/mpa.0000000000001364↗
- Paper M2-polarized tumor-associated macrophages promoted epithelial-mesenchymal transition in pancreatic cancer cells, partia… DOI 10.1038/labinvest.2013.69↗
- Dayang Chen · 2 shared papers, 2017–2020 · strength 0.19
- Qiang Zhan · 2 shared papers, 2017–2020 · strength 0.19
- Zhiyang Jiang · 1 shared paper, 2022 · strength 0.15
- Rui Xu · 1 shared paper, 2022 · strength 0.15
- Jing Qin · 1 shared paper, 2019 · strength 0.12
- Yunan Zhang · 1 shared paper, 2020 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.