ResearcherUniversity of Tennessee at KnoxvilleNo recent work listed
Hussein A.N. Al-Wadei
Matched on OpenAlex author ID A5110232793.
Their work
- Papers
- 10 (first author 2, last author 1)
- Trials
- 0
- Grants
- 0
- Collaborators
- 4
2011: 1 paper · 2012: 5 papers · 2013: 2 papers · 2014: 1 paper · 2015: none · 2016: 1 paper
Papers 10
- Nicotine induces self-renewal of pancreatic cancer stem cells via neurotransmitter-driven activation of sonic hedgehog signalling. Co-author DOI 10.1016/j.ejca.2015.10.003↗
- Differential modulation of nicotine-induced gemcitabine resistance by GABA receptor agonists in pancreatic cancer cell xenografts and in vitro. Co-author DOI 10.1186/1471-2407-14-725↗
- Chronic nicotine inhibits the therapeutic effects of gemcitabine on pancreatic cancer in vitro and in mouse xenografts. Co-author DOI 10.1016/j.ejca.2012.10.015↗
- Gamma-amino butyric acid (GABA) prevents the induction of nicotinic receptor-regulated signaling by chronic ethanol in pancreatic cancer cells and normal duct … Co-author DOI 10.1158/1940-6207.capr-12-0388↗
- Regulation of pancreatic cancer by neuropsychological stress responses: a novel target for intervention. Co-author DOI 10.1093/carcin/bgr251↗
- Effects of chronic nicotine on the autocrine regulation of pancreatic cancer cells and pancreatic duct epithelial cells by stimulatory and inhibitory neurotran… Co-author DOI 10.1093/carcin/bgs229↗
- Pancreatic cancer cells and normal pancreatic duct epithelial cells express an autocrine catecholamine loop that is activated by nicotinic acetylcholine recept… Co-author DOI 10.1158/1541-7786.mcr-11-0332↗
- Celecoxib and GABA cooperatively prevent the progression of pancreatic cancer in vitro and in xenograft models of stress-free and stress-exposed mice. First author DOI 10.1371/journal.pone.0043376↗
- Beta-adrenergic signaling in the development and progression of pulmonary and pancreatic adenocarcinoma. Last author DOI 10.2174/157339412800675351↗
- GABA (γ-aminobutyric acid), a non-protein amino acid counters the β-adrenergic cascade-activated oncogenic signaling in pancreatic cancer: a review of experime… First author DOI 10.1002/mnfr.201100229↗
Closest collaborators
The 4 strongest of 4 ranked by shared papers, trials and grants, with recent and small-team work weighing most.
- Hildegard M. Schuller · 9 shared papers, 2012–2016
- Paper Nicotine induces self-renewal of pancreatic cancer stem cells via neurotransmitter-driven activation of sonic hedgehog … DOI 10.1016/j.ejca.2015.10.003↗
- Paper Beta-adrenergic signaling in the development and progression of pulmonary and pancreatic adenocarcinoma. DOI 10.2174/157339412800675351↗
- Paper Chronic nicotine inhibits the therapeutic effects of gemcitabine on pancreatic cancer in vitro and in mouse xenografts. DOI 10.1016/j.ejca.2012.10.015↗
- Mohammed H. Al-Wadei · 7 shared papers, 2011–2016
- Paper Nicotine induces self-renewal of pancreatic cancer stem cells via neurotransmitter-driven activation of sonic hedgehog … DOI 10.1016/j.ejca.2015.10.003↗
- Paper Gamma-amino butyric acid (GABA) prevents the induction of nicotinic receptor-regulated signaling by chronic ethanol in … DOI 10.1158/1940-6207.capr-12-0388↗
- Paper Differential modulation of nicotine-induced gemcitabine resistance by GABA receptor agonists in pancreatic cancer cell … DOI 10.1186/1471-2407-14-725↗
- Jheelam Banerjee · 3 shared papers, 2013–2016
- Paper Nicotine induces self-renewal of pancreatic cancer stem cells via neurotransmitter-driven activation of sonic hedgehog … DOI 10.1016/j.ejca.2015.10.003↗
- Paper Chronic nicotine inhibits the therapeutic effects of gemcitabine on pancreatic cancer in vitro and in mouse xenografts. DOI 10.1016/j.ejca.2012.10.015↗
- Paper Differential modulation of nicotine-induced gemcitabine resistance by GABA receptor agonists in pancreatic cancer cell … DOI 10.1186/1471-2407-14-725↗
- Howard K. Plummer · 1 shared paper, 2012
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (the fewest links first, then the links with the most shared work). Built from public records only; no contact details are kept.