ResearcherKindai UniversityActive in the last 2 years
Hitoshi Okada
Matched on ORCID iD 0000-0001-5597-7212; OpenAlex author ID A5069440644. Sources: OpenAlex.
Their work
- Papers
- 4 (first author 0, last author 3)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 5
2023: 1 paper · 2024: 3 papers
Papers 4
- Deoxycytidine kinase inactivation enhances gemcitabine resistance and sensitizes mitochondrial metabolism interference in pancreatic cancer. Last author DOI 10.1038/s41419-024-06531-x↗
- Correction: Deoxycytidine kinase inactivation enhances gemcitabine resistance and sensitizes mitochondrial metabolism interference in pancreatic cancer. Last author DOI 10.1038/s41419-024-06628-3↗
- ACA-28, an ERK MAPK Signaling Modulator, Exerts Anticancer Activity through ROS Induction in Melanoma and Pancreatic Cancer Cells. Co-author DOI 10.1155/2024/7683793↗
- MYC/Glutamine Dependency Is a Therapeutic Vulnerability in Pancreatic Cancer with Deoxycytidine Kinase Inactivation-Induced Gemcitabine Resistance. Last author DOI 10.1158/1541-7786.mcr-22-0554↗
Closest collaborators
The 5 strongest of 5 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Suman Dash · 3 shared papers, 2023–2024 · strength 0.72
- Paper Deoxycytidine kinase inactivation enhances gemcitabine resistance and sensitizes mitochondrial metabolism interference … DOI 10.1038/s41419-024-06531-x↗
- Paper Correction: Deoxycytidine kinase inactivation enhances gemcitabine resistance and sensitizes mitochondrial metabolism i… DOI 10.1038/s41419-024-06628-3↗
- Paper MYC/Glutamine Dependency Is a Therapeutic Vulnerability in Pancreatic Cancer with Deoxycytidine Kinase Inactivation-Ind… DOI 10.1158/1541-7786.mcr-22-0554↗
- Golam Iftakhar Khandakar · 1 shared paper, 2024 · strength 0.21
- Teruaki Takasaki · 1 shared paper, 2024 · strength 0.21
- Kazuto Nishio · 1 shared paper, 2024 · strength 0.21
- Reiko Sugiura · 1 shared paper, 2024 · strength 0.21
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.