ResearcherTongji UniversityActive in the last 2 years
Dacheng Xie
Matched on ORCID iD 0000-0001-6420-6620; OpenAlex author ID A5109602090. Sources: OpenAlex.
Their work
- Papers
- 15 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 35
2012: 1 paper · 2013: none · 2014: 3 papers · 2015: 3 papers · 2016: none · 2017: 3 papers · 2018: none · 2019: 1 paper · 2020: 3 papers · 2021: none · 2022: none · 2023: none · 2024: none · 2025: 1 paper
Papers 15
- Innate immune cell barrier-related genes inform precision prognosis in pancreatic cancer. Co-author DOI 10.3389/fimmu.2025.1559373↗
- A novel KDM5A/MPC-1 signaling pathway promotes pancreatic cancer progression via redirecting mitochondrial pyruvate metabolism. Co-author DOI 10.1038/s41388-019-1051-8↗
- Methylation of HSP70 Orchestrates Its Binding to and Stabilization of BCL2 mRNA and Renders Pancreatic Cancer Cells Resistant to Therapeutics. Co-author DOI 10.1158/0008-5472.can-19-1738↗
- The FOXM1/ATX signaling contributes to pancreatic cancer development. Co-author PMID 32913521↗
- Pancreatic Acinar-to-Ductal Metaplasia and Pancreatic Cancer. First author DOI 10.1007/978-1-4939-8879-2_26↗
- STK33 Promotes Growth and Progression of Pancreatic Cancer as a Critical Downstream Mediator of HIF1α. Co-author DOI 10.1158/0008-5472.can-17-0067↗
- The Novel KLF4/MSI2 Signaling Pathway Regulates Growth and Metastasis of Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-16-1064↗
- DNA-Methyltransferase 1 Induces Dedifferentiation of Pancreatic Cancer Cells through Silencing of Krüppel-Like Factor 4 Expression. Co-author DOI 10.1158/1078-0432.ccr-17-0387↗
- Pancreatic cancer stromal biology and therapy. Co-author DOI 10.1016/j.gendis.2015.01.002↗
- Activation of vitamin D receptor signaling downregulates the expression of nuclear FOXM1 protein and suppresses pancreatic cancer cell stemness. Co-author DOI 10.1158/1078-0432.ccr-14-2437↗
- Hippo transducer TAZ promotes epithelial mesenchymal transition and supports pancreatic cancer progression. Co-author DOI 10.18632/oncotarget.5772↗
- Down-regulation of microRNA-494 via loss of SMAD4 increases FOXM1 and β-catenin signaling in pancreatic ductal adenocarcinoma cells. Co-author DOI 10.1053/j.gastro.2014.04.048↗
- FOXM1c promotes pancreatic cancer epithelial-to-mesenchymal transition and metastasis via upregulation of expression of the urokinase plasminogen activator sys… First author DOI 10.1158/1078-0432.ccr-13-2311↗
- FOXM1 promotes the warburg effect and pancreatic cancer progression via transactivation of LDHA expression. Co-author DOI 10.1158/1078-0432.ccr-13-2407↗
- Sp/KLF family and tumor angiogenesis in pancreatic cancer. Co-author DOI 10.2174/13816128112092420↗
Closest collaborators
The 25 strongest of 35 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Keping Xie · 12 shared papers, 2014–2025 · strength 1.09
- Paper Innate immune cell barrier-related genes inform precision prognosis in pancreatic cancer. DOI 10.3389/fimmu.2025.1559373↗
- Paper A novel KDM5A/MPC-1 signaling pathway promotes pancreatic cancer progression via redirecting mitochondrial pyruvate met… DOI 10.1038/s41388-019-1051-8↗
- Paper Methylation of HSP70 Orchestrates Its Binding to and Stabilization of BCL2 mRNA and Renders Pancreatic Cancer Cells Res… DOI 10.1158/0008-5472.can-19-1738↗
- Daoyan Wei · 7 shared papers, 2014–2020 · strength 0.54
- Paper Pancreatic Acinar-to-Ductal Metaplasia and Pancreatic Cancer. DOI 10.1007/978-1-4939-8879-2_26↗
- Paper Methylation of HSP70 Orchestrates Its Binding to and Stabilization of BCL2 mRNA and Renders Pancreatic Cancer Cells Res… DOI 10.1158/0008-5472.can-19-1738↗
- Paper The Novel KLF4/MSI2 Signaling Pathway Regulates Growth and Metastasis of Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-16-1064↗
- Jiujie Cui · 8 shared papers, 2012–2020 · strength 0.46
- Paper A novel KDM5A/MPC-1 signaling pathway promotes pancreatic cancer progression via redirecting mitochondrial pyruvate met… DOI 10.1038/s41388-019-1051-8↗
- Paper The Novel KLF4/MSI2 Signaling Pathway Regulates Growth and Metastasis of Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-16-1064↗
- Paper FOXM1c promotes pancreatic cancer epithelial-to-mesenchymal transition and metastasis via upregulation of expression of… DOI 10.1158/1078-0432.ccr-13-2311↗
- Ming Quan · 4 shared papers, 2015–2020 · strength 0.38
- Paper The FOXM1/ATX signaling contributes to pancreatic cancer development. PMID 32913521↗
- Paper A novel KDM5A/MPC-1 signaling pathway promotes pancreatic cancer progression via redirecting mitochondrial pyruvate met… DOI 10.1038/s41388-019-1051-8↗
- Paper The Novel KLF4/MSI2 Signaling Pathway Regulates Growth and Metastasis of Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-16-1064↗
- Yong Gao · 6 shared papers, 2014–2020 · strength 0.38
- Paper A novel KDM5A/MPC-1 signaling pathway promotes pancreatic cancer progression via redirecting mitochondrial pyruvate met… DOI 10.1038/s41388-019-1051-8↗
- Paper The Novel KLF4/MSI2 Signaling Pathway Regulates Growth and Metastasis of Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-16-1064↗
- Paper FOXM1c promotes pancreatic cancer epithelial-to-mesenchymal transition and metastasis via upregulation of expression of… DOI 10.1158/1078-0432.ccr-13-2311↗
- Qiang Yi Luo · 1 shared paper, 2025 · strength 0.33
- Xiaojia Li · 1 shared paper, 2025 · strength 0.33
- Liang Wang · 3 shared papers, 2015–2019 · strength 0.28
- Paper Pancreatic Acinar-to-Ductal Metaplasia and Pancreatic Cancer. DOI 10.1007/978-1-4939-8879-2_26↗
- Paper The Novel KLF4/MSI2 Signaling Pathway Regulates Growth and Metastasis of Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-16-1064↗
- Paper Hippo transducer TAZ promotes epithelial mesenchymal transition and supports pancreatic cancer progression. DOI 10.18632/oncotarget.5772↗
- Fanyang Kong · 2 shared papers, 2017–2020 · strength 0.18
- Paper Methylation of HSP70 Orchestrates Its Binding to and Stabilization of BCL2 mRNA and Renders Pancreatic Cancer Cells Res… DOI 10.1158/0008-5472.can-19-1738↗
- Paper STK33 Promotes Growth and Progression of Pancreatic Cancer as a Critical Downstream Mediator of HIF1α. DOI 10.1158/0008-5472.can-17-0067↗
- Li Li · 1 shared paper, 2020 · strength 0.15
- Yong Gao · 1 shared paper, 2020 · strength 0.15
- Jiawei Du · 2 shared papers, 2017 · strength 0.13
- Paper DNA-Methyltransferase 1 Induces Dedifferentiation of Pancreatic Cancer Cells through Silencing of Krüppel-Like Factor 4… DOI 10.1158/1078-0432.ccr-17-0387↗
- Paper STK33 Promotes Growth and Progression of Pancreatic Cancer as a Critical Downstream Mediator of HIF1α. DOI 10.1158/0008-5472.can-17-0067↗
- Sushovan Guha · 1 shared paper, 2020 · strength 0.12
- Tiansuo Zhao · 1 shared paper, 2020 · strength 0.11
- Liang Wang · 1 shared paper, 2020 · strength 0.11
- Xiangyu Kong · 2 shared papers, 2014–2017 · strength 0.10
- Paper STK33 Promotes Growth and Progression of Pancreatic Cancer as a Critical Downstream Mediator of HIF1α. DOI 10.1158/0008-5472.can-17-0067↗
- Paper Down-regulation of microRNA-494 via loss of SMAD4 increases FOXM1 and β-catenin signaling in pancreatic ductal adenocar… DOI 10.1053/j.gastro.2014.04.048↗
- Yiqi Du · 2 shared papers, 2014–2017 · strength 0.10
- Paper STK33 Promotes Growth and Progression of Pancreatic Cancer as a Critical Downstream Mediator of HIF1α. DOI 10.1158/0008-5472.can-17-0067↗
- Paper Down-regulation of microRNA-494 via loss of SMAD4 increases FOXM1 and β-catenin signaling in pancreatic ductal adenocar… DOI 10.1053/j.gastro.2014.04.048↗
- Weihua Jiang · 2 shared papers, 2012–2014 · strength 0.08
- Qingyong Ma · 1 shared paper, 2017 · strength 0.07
- Kun Guo · 1 shared paper, 2017 · strength 0.07
- Zhiwei Li · 1 shared paper, 2017 · strength 0.06
- Yongmin Yan · 1 shared paper, 2017 · strength 0.06
- Victoria K. Xie · 1 shared paper, 2017 · strength 0.06
- Xiangsheng Zuo · 1 shared paper, 2017 · strength 0.06
- Zhao‐Shen Li · 1 shared paper, 2017 · strength 0.06
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.