Hengqiang Zhao

Closest collaborators

The 14 strongest of 14 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

1. Zhengle Zhang · 5 shared papers, 2016–2021 · strength 0.44
   • Paper The reciprocal loop of MALAT1 and HIF-1α facilitates gemcitabine resistance in pancreatic cancer by enhancing glycolysis DOI 10.21203/rs.3.rs-135235/v2↗
   • Paper ROS/KRAS/AMPK Signaling Contributes to Gemcitabine-Induced Stem-like Cell Properties in Pancreatic Cancer. DOI 10.1016/j.omto.2019.07.005↗
   • Paper Up-regulation of glycolysis promotes the stemness and EMT phenotypes in gemcitabine-resistant pancreatic cancer cells. DOI 10.1111/jcmm.13126↗
2. Tao Yin · 5 shared papers, 2016–2019 · strength 0.39
   • Paper ROS/KRAS/AMPK Signaling Contributes to Gemcitabine-Induced Stem-like Cell Properties in Pancreatic Cancer. DOI 10.1016/j.omto.2019.07.005↗
   • Paper High glucose promotes pancreatic cancer cells to escape from immune surveillance via AMPK-Bmi1-GATA2-MICA/B pathway. DOI 10.1186/s13046-019-1209-9↗
   • Paper Up-regulation of glycolysis promotes the stemness and EMT phenotypes in gemcitabine-resistant pancreatic cancer cells. DOI 10.1111/jcmm.13126↗
3. Qingke Duan · 5 shared papers, 2016–2019 · strength 0.39
   • Paper ROS/KRAS/AMPK Signaling Contributes to Gemcitabine-Induced Stem-like Cell Properties in Pancreatic Cancer. DOI 10.1016/j.omto.2019.07.005↗
   • Paper High glucose promotes pancreatic cancer cells to escape from immune surveillance via AMPK-Bmi1-GATA2-MICA/B pathway. DOI 10.1186/s13046-019-1209-9↗
   • Paper Up-regulation of glycolysis promotes the stemness and EMT phenotypes in gemcitabine-resistant pancreatic cancer cells. DOI 10.1111/jcmm.13126↗
4. Chunyou Wang · 5 shared papers, 2016–2019 · strength 0.39
   • Paper ROS/KRAS/AMPK Signaling Contributes to Gemcitabine-Induced Stem-like Cell Properties in Pancreatic Cancer. DOI 10.1016/j.omto.2019.07.005↗
   • Paper High glucose promotes pancreatic cancer cells to escape from immune surveillance via AMPK-Bmi1-GATA2-MICA/B pathway. DOI 10.1186/s13046-019-1209-9↗
   • Paper Up-regulation of glycolysis promotes the stemness and EMT phenotypes in gemcitabine-resistant pancreatic cancer cells. DOI 10.1111/jcmm.13126↗
5. Hehe Li · 3 shared papers, 2017–2019 · strength 0.27
   • Paper ROS/KRAS/AMPK Signaling Contributes to Gemcitabine-Induced Stem-like Cell Properties in Pancreatic Cancer. DOI 10.1016/j.omto.2019.07.005↗
   • Paper High glucose promotes pancreatic cancer cells to escape from immune surveillance via AMPK-Bmi1-GATA2-MICA/B pathway. DOI 10.1186/s13046-019-1209-9↗
   • Paper Up-regulation of glycolysis promotes the stemness and EMT phenotypes in gemcitabine-resistant pancreatic cancer cells. DOI 10.1111/jcmm.13126↗
6. Heshui Wu · 3 shared papers, 2016–2019 · strength 0.23
   • Paper High glucose promotes pancreatic cancer cells to escape from immune surveillance via AMPK-Bmi1-GATA2-MICA/B pathway. DOI 10.1186/s13046-019-1209-9↗
   • Paper Up-regulation of glycolysis promotes the stemness and EMT phenotypes in gemcitabine-resistant pancreatic cancer cells. DOI 10.1111/jcmm.13126↗
   • Paper Gemcitabine treatment promotes pancreatic cancer stemness through the Nox/ROS/NF-κB/STAT3 signaling cascade. DOI 10.1016/j.canlet.2016.08.023↗
7. Shihong Wu · 2 shared papers, 2019 · strength 0.20
   • Paper ROS/KRAS/AMPK Signaling Contributes to Gemcitabine-Induced Stem-like Cell Properties in Pancreatic Cancer. DOI 10.1016/j.omto.2019.07.005↗
   • Paper High glucose promotes pancreatic cancer cells to escape from immune surveillance via AMPK-Bmi1-GATA2-MICA/B pathway. DOI 10.1186/s13046-019-1209-9↗
8. Pei Mei · 1 shared paper, 2021 · strength 0.14
   • Paper The reciprocal loop of MALAT1 and HIF-1α facilitates gemcitabine resistance in pancreatic cancer by enhancing glycolysis DOI 10.21203/rs.3.rs-135235/v2↗
9. Zhi‐Gang Tang · 1 shared paper, 2021 · strength 0.14
   • Paper The reciprocal loop of MALAT1 and HIF-1α facilitates gemcitabine resistance in pancreatic cancer by enhancing glycolysis DOI 10.21203/rs.3.rs-135235/v2↗
10. Jing Tao · 1 shared paper, 2021 · strength 0.14
    • Paper The reciprocal loop of MALAT1 and HIF-1α facilitates gemcitabine resistance in pancreatic cancer by enhancing glycolysis DOI 10.21203/rs.3.rs-135235/v2↗
11. Jie Sun · 1 shared paper, 2016 · strength 0.07
    • Paper Construction and analysis of dysregulated lncRNA-associated ceRNA network identified novel lncRNA biomarkers for early … DOI 10.18632/oncotarget.10891↗
12. Meng Zhou · 1 shared paper, 2016 · strength 0.07
    • Paper Construction and analysis of dysregulated lncRNA-associated ceRNA network identified novel lncRNA biomarkers for early … DOI 10.18632/oncotarget.10891↗
13. Tao Liu · 1 shared paper, 2016 · strength 0.06
    • Paper Gemcitabine treatment promotes pancreatic cancer stemness through the Nox/ROS/NF-κB/STAT3 signaling cascade. DOI 10.1016/j.canlet.2016.08.023↗
14. Pengfei Shi · 1 shared paper, 2016 · strength 0.06
    • Paper Bmi1 inhibition enhances the sensitivity of pancreatic cancer cells to gemcitabine. DOI 10.18632/oncotarget.9293↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.