ResearcherBC Cancer AgencyActive in the last 2 years
Joanna M. Karasinska
Matched on ORCID iD 0000-0001-6861-4189; OpenAlex author ID A5036473360. Sources: OpenAlex.
Their work
- Papers
- 26 (first author 6, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 73
2017: 1 paper · 2018: 1 paper · 2019: 3 papers · 2020: 4 papers · 2021: 6 papers · 2022: 1 paper · 2023: 1 paper · 2024: 3 papers · 2025: 4 papers · 2026: 2 papers
Papers 26
- DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab in metastatic pancreatic ductal adenocarc… Co-author DOI 10.1038/s41467-026-70120-z↗
- Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1078-0432.ccr-26-0793↗
- Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. First author DOI 10.1016/j.isci.2025.113176↗
- The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. Co-author DOI 10.1038/s41698-025-00888-8↗
- KRAS-induced STN1 (OBFC1) promotes proper CTC1-STN1-TEN1 complex-independent DNA double-strand break repair and cell cycle checkpoint maintenance in pancreatic… Co-author DOI 10.1093/nar/gkaf983↗
- New-onset diabetes as an emerging risk group for early detection of pancreatic cancer: current evidence, clinical challenge, and future directions. First author DOI 10.3389/fgstr.2025.1645459↗
- A Deep Learning Approach for the Identification of the Molecular Subtypes of Pancreatic Ductal Adenocarcinoma Based on Whole Slide Pathology Images. Co-author DOI 10.1016/j.ajpath.2024.08.006↗
- Nutrient scavenging-fueled growth in pancreatic cancer depends on caveolae-mediated endocytosis under nutrient-deprived conditions. Co-author DOI 10.1126/sciadv.adj3551↗
- Pancreatic cancer tumor organoids exhibit subtype-specific differences in metabolic profiles. First author DOI 10.1186/s40170-024-00357-z↗
- Clinically impactful metabolic subtypes of pancreatic ductal adenocarcinoma (PDAC). Co-author DOI 10.3389/fgene.2023.1282824↗
- Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based similarities to cholangiocarcinoma. Co-author DOI 10.1038/s41467-022-33718-7↗
- Tumor infiltrating neutrophils and gland formation predict overall survival and molecular subgroups in pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/cam4.3695↗
- Stroma vs epithelium-enhanced prognostics through histologic stratification in pancreatic ductal adenocarcinoma. First author DOI 10.1002/ijc.33304↗
- Clinical and genomic characterisation of mismatch repair deficient pancreatic adenocarcinoma. Co-author DOI 10.1136/gutjnl-2020-320730↗
- Delving into Early-onset Pancreatic Ductal Adenocarcinoma: How Does Age Fit In? Co-author DOI 10.1158/1078-0432.ccr-20-1042↗
- Subtype-Discordant Pancreatic Ductal Adenocarcinoma Tumors Show Intermediate Clinical and Molecular Characteristics. First author DOI 10.1158/1078-0432.ccr-20-2831↗
- Advancing the Care of Pancreatic Cancer Patients: Moving Beyond Just Tumour Tissue. First author DOI 10.1177/11772719211049852↗
- Burden of hereditary cancer susceptibility in unselected patients with pancreatic ductal adenocarcinoma referred for germline screening. Co-author DOI 10.1002/cam4.2973↗
- Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. Co-author DOI 10.1038/s41588-019-0566-9↗
- Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. Co-author DOI 10.1038/s41588-020-0588-3↗
- Altered Gene Expression along the Glycolysis-Cholesterol Synthesis Axis Is Associated with Outcome in Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-19-1543↗
- Regulation of pH by Carbonic Anhydrase 9 Mediates Survival of Pancreatic Cancer Cells With Activated KRAS in Response to Hypoxia. Co-author DOI 10.1053/j.gastro.2019.05.004↗
- Base excision repair deficiency signatures implicate germline and somatic MUTYH aberrations in pancreatic ductal adenocarcinoma and breast cancer oncogenesis. Co-author DOI 10.1101/mcs.a003681↗
- NRG1 Gene Fusions Are Recurrent, Clinically Actionable Gene Rearrangements in KRAS Wild-Type Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1078-0432.ccr-19-0191↗
- Temporal Dynamics of Genomic Alterations in a BRCA1 Germline-Mutated Pancreatic Cancer With Low Genomic Instability Burden but Exceptional Response to Fluorour… Co-author DOI 10.1200/po.18.00057↗
- A predictive analysis of the SP120 and 10D7G2 antibodies for human equilibrative nucleoside transporter 1 (hENT1) in pancreatic ductal adenocarcinoma treated w… Co-author DOI 10.1002/cjp2.75↗
Closest collaborators
The 25 strongest of 73 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- David F. Schaeffer · 26 shared papers, 2017–2026 · strength 3.38
- Paper New-onset diabetes as an emerging risk group for early detection of pancreatic cancer: current evidence, clinical chall… DOI 10.3389/fgstr.2025.1645459↗
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper KRAS-induced STN1 (OBFC1) promotes proper CTC1-STN1-TEN1 complex-independent DNA double-strand break repair and cell cy… DOI 10.1093/nar/gkaf983↗
- Daniel John Renouf · 26 shared papers, 2017–2026 · strength 3.38
- Paper New-onset diabetes as an emerging risk group for early detection of pancreatic cancer: current evidence, clinical chall… DOI 10.3389/fgstr.2025.1645459↗
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper KRAS-induced STN1 (OBFC1) promotes proper CTC1-STN1-TEN1 complex-independent DNA double-strand break repair and cell cy… DOI 10.1093/nar/gkaf983↗
- James Thomas Topham · 19 shared papers, 2019–2026 · strength 2.49
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper KRAS-induced STN1 (OBFC1) promotes proper CTC1-STN1-TEN1 complex-independent DNA double-strand break repair and cell cy… DOI 10.1093/nar/gkaf983↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Steve E. Kalloger · 14 shared papers, 2017–2025 · strength 1.47
- Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
- Paper Pancreatic cancer tumor organoids exhibit subtype-specific differences in metabolic profiles. DOI 10.1186/s40170-024-00357-z↗
- Paper Advancing the Care of Pancreatic Cancer Patients: Moving Beyond Just Tumour Tissue. DOI 10.1177/11772719211049852↗
- Jennifer Jane Knox · 12 shared papers, 2019–2026 · strength 1.44
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
- Steve Gallinger · 12 shared papers, 2019–2026 · strength 1.39
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Faiyaz Notta · 10 shared papers, 2020–2026 · strength 1.16
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Grainne Mary O'Kane · 10 shared papers, 2020–2025 · strength 1.12
- Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Clinically impactful metabolic subtypes of pancreatic ductal adenocarcinoma (PDAC). DOI 10.3389/fgene.2023.1282824↗
- Oliver F. Bathe · 6 shared papers, 2021–2025 · strength 0.80
- Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Clinically impactful metabolic subtypes of pancreatic ductal adenocarcinoma (PDAC). DOI 10.3389/fgene.2023.1282824↗
- George Zogopoulos · 6 shared papers, 2020–2026 · strength 0.77
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Clinically impactful metabolic subtypes of pancreatic ductal adenocarcinoma (PDAC). DOI 10.3389/fgene.2023.1282824↗
- Hui‐Li Wong · 9 shared papers, 2017–2022 · strength 0.76
- Paper Stroma vs epithelium-enhanced prognostics through histologic stratification in pancreatic ductal adenocarcinoma. DOI 10.1002/ijc.33304↗
- Paper Tumor infiltrating neutrophils and gland formation predict overall survival and molecular subgroups in pancreatic ducta… DOI 10.1002/cam4.3695↗
- Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
- Rachel Anne Goodwin · 5 shared papers, 2021–2025 · strength 0.64
- Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
- Michael K.C. Lee · 6 shared papers, 2019–2021 · strength 0.53
- Paper Tumor infiltrating neutrophils and gland formation predict overall survival and molecular subgroups in pancreatic ducta… DOI 10.1002/cam4.3695↗
- Paper Delving into Early-onset Pancreatic Ductal Adenocarcinoma: How Does Age Fit In? DOI 10.1158/1078-0432.ccr-20-1042↗
- Paper Subtype-Discordant Pancreatic Ductal Adenocarcinoma Tumors Show Intermediate Clinical and Molecular Characteristics. DOI 10.1158/1078-0432.ccr-20-2831↗
- Sharlene Gill · 3 shared papers, 2019–2026 · strength 0.48
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Paper NRG1 Gene Fusions Are Recurrent, Clinically Actionable Gene Rearrangements in KRAS Wild-Type Pancreatic Ductal Adenocar… DOI 10.1158/1078-0432.ccr-19-0191↗
- Erica S. Tsang · 4 shared papers, 2021–2025 · strength 0.46
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
- Paper Delving into Early-onset Pancreatic Ductal Adenocarcinoma: How Does Age Fit In? DOI 10.1158/1078-0432.ccr-20-1042↗
- Lan Valerie Tao · 2 shared papers, 2024–2025 · strength 0.46
- Hassan A. Ali · 3 shared papers, 2022–2025 · strength 0.46
- Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
- Paper Pancreatic cancer tumor organoids exhibit subtype-specific differences in metabolic profiles. DOI 10.1186/s40170-024-00357-z↗
- Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
- Ravi Ramjeesingh · 2 shared papers, 2026 · strength 0.42
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Mustapha Ali Tehfe · 2 shared papers, 2026 · strength 0.42
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Petr Kavan · 2 shared papers, 2026 · strength 0.42
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Christina Kim · 2 shared papers, 2026 · strength 0.42
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Derek J. Jonker · 2 shared papers, 2026 · strength 0.42
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Chris J. O’Callaghan · 2 shared papers, 2026 · strength 0.42
- Paper DNA Repair gene alterations and efficacy from gemcitabine and nab-paclitaxel with/without durvalumab and tremelimumab i… DOI 10.1038/s41467-026-70120-z↗
- Paper Clinical and Molecular Correlates of Circulating Tumor Fraction in Patients with Metastatic Pancreatic Ductal Adenocarc… DOI 10.1158/1078-0432.ccr-26-0793↗
- Sandra Elisabeth Fischer · 4 shared papers, 2020–2025 · strength 0.41
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Clinical and genomic characterisation of mismatch repair deficient pancreatic adenocarcinoma. DOI 10.1136/gutjnl-2020-320730↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Robert C. Grant · 4 shared papers, 2020–2025 · strength 0.41
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Clinical and genomic characterisation of mismatch repair deficient pancreatic adenocarcinoma. DOI 10.1136/gutjnl-2020-320730↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.