ResearcherUniversity of UdineActive in the last 2 years
Valentina Rapozzi
Matched on ORCID iD 0000-0001-7619-5252; OpenAlex author ID A5058199765. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 4
2013: 1 paper · 2014: none · 2015: none · 2016: none · 2017: 1 paper · 2018: none · 2019: none · 2020: none · 2021: none · 2022: 1 paper · 2023: 1 paper · 2024: 1 paper · 2025: 2 papers
Papers 7
- KRAS and NRF2 drive metabolic reprogramming in pancreatic cancer cells: the influence of oxidative and nitrosative stress. Co-author DOI 10.3389/fcell.2025.1547582↗
- Correction: KRAS and NRF2 drive metabolic reprogramming in pancreatic cancer cells: the influence of oxidative and nitrosative stress. Co-author DOI 10.3389/fcell.2025.1661525↗
- NRF2 interacts with distal enhancer and inhibits nitric oxide synthase 2 expression in KRAS-driven pancreatic cancer cells. Co-author DOI 10.1016/j.bbamcr.2023.119606↗
- Suppression of the KRAS-NRF2 axis shifts arginine into the phosphocreatine energy system in pancreatic cancer cells. Co-author DOI 10.1016/j.isci.2023.108566↗
- Photosensitization of pancreatic cancer cells by cationic alkyl-porphyrins in free form or engrafted into POPC liposomes: The relationship between delivery mod… Co-author DOI 10.1016/j.jphotobiol.2022.112449↗
- Critical role of hnRNP A1 in activating KRAS transcription in pancreatic cancer cells: A molecular mechanism involving G4 DNA. First author DOI 10.1016/j.bbagen.2016.11.031↗
- MAZ-binding G4-decoy with locked nucleic acid and twisted intercalating nucleic acid modifications suppresses KRAS in pancreatic cancer cells and delays tumor … Co-author DOI 10.1093/nar/gkt127↗
Closest collaborators
The 4 strongest of 4 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Luigi Emilio Xodo · 7 shared papers, 2013–2025 · strength 1.47
- Paper KRAS and NRF2 drive metabolic reprogramming in pancreatic cancer cells: the influence of oxidative and nitrosative stre… DOI 10.3389/fcell.2025.1547582↗
- Paper Correction: KRAS and NRF2 drive metabolic reprogramming in pancreatic cancer cells: the influence of oxidative and nitr… DOI 10.3389/fcell.2025.1661525↗
- Paper NRF2 interacts with distal enhancer and inhibits nitric oxide synthase 2 expression in KRAS-driven pancreatic cancer ce… DOI 10.1016/j.bbamcr.2023.119606↗
- Eros Di Giorgio · 5 shared papers, 2022–2025 · strength 1.33
- Paper KRAS and NRF2 drive metabolic reprogramming in pancreatic cancer cells: the influence of oxidative and nitrosative stre… DOI 10.3389/fcell.2025.1547582↗
- Paper Correction: KRAS and NRF2 drive metabolic reprogramming in pancreatic cancer cells: the influence of oxidative and nitr… DOI 10.3389/fcell.2025.1661525↗
- Paper NRF2 interacts with distal enhancer and inhibits nitric oxide synthase 2 expression in KRAS-driven pancreatic cancer ce… DOI 10.1016/j.bbamcr.2023.119606↗
- Annalisa Ferino · 2 shared papers, 2022–2023 · strength 0.34
- Paper Suppression of the KRAS-NRF2 axis shifts arginine into the phosphocreatine energy system in pancreatic cancer cells. DOI 10.1016/j.isci.2023.108566↗
- Paper Photosensitization of pancreatic cancer cells by cationic alkyl-porphyrins in free form or engrafted into POPC liposome… DOI 10.1016/j.jphotobiol.2022.112449↗
- Susanna Cogoi · 2 shared papers, 2013–2017 · strength 0.14
- Paper Critical role of hnRNP A1 in activating KRAS transcription in pancreatic cancer cells: A molecular mechanism involving … DOI 10.1016/j.bbagen.2016.11.031↗
- Paper MAZ-binding G4-decoy with locked nucleic acid and twisted intercalating nucleic acid modifications suppresses KRAS in p… DOI 10.1093/nar/gkt127↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.