ResearcherTianjin Medical University Cancer Institute and HospitalActive in the last 2 years

Bin Wang

ORCID-verified (high confidence, 0.95), identity_conf@1As of 7 November 2026

Matched on ORCID iD 0000-0001-7665-6290; OpenAlex author ID A5115602018. Sources: OpenAlex.

Their work

Papers: 2
Trials: 0
Grants: 0
Labs led: 0
Collaborators: 12

Papers, trials and grants by year

2024: 1 paper · 2025: 1 paper

Papers 2

The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.canlet.2024.217408 ↗
Metabolic and Immunological Implications of MME+CAF-Mediated Hypoxia Signaling in Pancreatic Cancer Progression: Therapeutic Insights and Translational Opportu… Co-author DOI 10.1186/s12575-024-00254-1 ↗

Closest collaborators

The 12 strongest of 12 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Yongjie Xie · 2 shared papers, 2024–2025 · strength 0.39
- Paper Metabolic and Immunological Implications of MME+CAF-Mediated Hypoxia Signaling in Pancreatic Cancer Progression: Therap… DOI 10.1186/s12575-024-00254-1 ↗
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Zhanyu Pan · 1 shared paper, 2024 · strength 0.20
- Paper Metabolic and Immunological Implications of MME+CAF-Mediated Hypoxia Signaling in Pancreatic Cancer Progression: Therap… DOI 10.1186/s12575-024-00254-1 ↗
Weishuai Liu · 1 shared paper, 2024 · strength 0.20
- Paper Metabolic and Immunological Implications of MME+CAF-Mediated Hypoxia Signaling in Pancreatic Cancer Progression: Therap… DOI 10.1186/s12575-024-00254-1 ↗
Chongbiao Huang · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Xiuchao Wang · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Tiansuo Zhao · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Yiping Zou · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Jun Yu · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Qingxiao Fang · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Yukuan Feng · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Tianxing Zhou · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗
Jihui Hao · 1 shared paper, 2025 · strength 0.19
- Paper The nuclear condensates of ESE3/EHF induce cellular senescence without the associated inflammatory secretory phenotype … DOI 10.1016/j.canlet.2024.217408 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.