ResearcherThe University of MelbourneActive in the last 2 years
Ching‐Seng Ang
Matched on ORCID iD 0000-0001-7861-6719; OpenAlex author ID A5058055203. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 8
2022: 1 paper · 2023: 1 paper · 2024: 1 paper · 2025: 5 papers
Papers 8
- Knockout of p21-Activated Kinase 4 Stimulates MHC I Expression of Pancreatic Cancer Cells via an Autophagy-Independent Pathway. Co-author DOI 10.3390/cancers17030511↗
- The Effects of PAK-Regulated Tumour Vasculature on Gemcitabine Response of Pancreatic Cancer. Co-author DOI 10.3390/cancers17213434↗
- Evaluation of Nanoparticle-Based Plasma Enrichment on Individuals with Primary and Metastatic Pancreatic Cancer. Co-author DOI 10.3390/cancers17233765↗
- Targeting PAK1 or PAK4 Uncovers Different Mechanisms of Vascular Reprogramming in Pancreatic Cancer. Co-author DOI 10.3390/cells14221806↗
- P-21 Kinase 1 or 4 Knockout Stimulated Anti-Tumour Immunity Against Pancreatic Cancer by Enhancing Vascular Normalisation. Co-author DOI 10.3390/ijms26178357↗
- Inhibition of P21-activated kinases 1 and 4 synergistically suppresses the growth of pancreatic cancer by stimulating anti-tumour immunity. Co-author DOI 10.1186/s12964-024-01670-2↗
- CXCL5 knockdown attenuated gemcitabine resistance of pancreatic cancer through regulation of cancer cells and tumour stroma. Co-author PMID 37193135↗
- A novel PAK4 inhibitor suppresses pancreatic cancer growth and enhances the inhibitory effect of gemcitabine. Co-author DOI 10.1016/j.tranon.2021.101329↗
Closest collaborators
The 8 strongest of 8 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Mehrdad Nikfarjam · 8 shared papers, 2022–2025 · strength 2.00
- Paper Knockout of p21-Activated Kinase 4 Stimulates MHC I Expression of Pancreatic Cancer Cells via an Autophagy-Independent … DOI 10.3390/cancers17030511↗
- Paper Targeting PAK1 or PAK4 Uncovers Different Mechanisms of Vascular Reprogramming in Pancreatic Cancer. DOI 10.3390/cells14221806↗
- Paper The Effects of PAK-Regulated Tumour Vasculature on Gemcitabine Response of Pancreatic Cancer. DOI 10.3390/cancers17213434↗
- Hong Zhen He · 7 shared papers, 2022–2025 · strength 1.77
- Paper Knockout of p21-Activated Kinase 4 Stimulates MHC I Expression of Pancreatic Cancer Cells via an Autophagy-Independent … DOI 10.3390/cancers17030511↗
- Paper Targeting PAK1 or PAK4 Uncovers Different Mechanisms of Vascular Reprogramming in Pancreatic Cancer. DOI 10.3390/cells14221806↗
- Paper The Effects of PAK-Regulated Tumour Vasculature on Gemcitabine Response of Pancreatic Cancer. DOI 10.3390/cancers17213434↗
- Yi Ma · 5 shared papers, 2023–2025 · strength 1.25
- Paper Knockout of p21-Activated Kinase 4 Stimulates MHC I Expression of Pancreatic Cancer Cells via an Autophagy-Independent … DOI 10.3390/cancers17030511↗
- Paper The Effects of PAK-Regulated Tumour Vasculature on Gemcitabine Response of Pancreatic Cancer. DOI 10.3390/cancers17213434↗
- Paper P-21 Kinase 1 or 4 Knockout Stimulated Anti-Tumour Immunity Against Pancreatic Cancer by Enhancing Vascular Normalisati… DOI 10.3390/ijms26178357↗
- Arian Ansardamavandi · 3 shared papers, 2025 · strength 0.84
- Paper Targeting PAK1 or PAK4 Uncovers Different Mechanisms of Vascular Reprogramming in Pancreatic Cancer. DOI 10.3390/cells14221806↗
- Paper The Effects of PAK-Regulated Tumour Vasculature on Gemcitabine Response of Pancreatic Cancer. DOI 10.3390/cancers17213434↗
- Paper P-21 Kinase 1 or 4 Knockout Stimulated Anti-Tumour Immunity Against Pancreatic Cancer by Enhancing Vascular Normalisati… DOI 10.3390/ijms26178357↗
- Hong He · 1 shared paper, 2024 · strength 0.23
- Kai Wang · 1 shared paper, 2023 · strength 0.18
- Nien‐Hung Lee · 1 shared paper, 2023 · strength 0.18
- Dong Li · 1 shared paper, 2022 · strength 0.17
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.