ResearcherSheng Jing HospitalNo recent work listed
Juan Zeng
Matched on ORCID iD 0000-0001-8227-0415; OpenAlex author ID A5083210325. Sources: OpenAlex.
Their work
- Papers
- 6 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 7
2015: 1 paper · 2016: none · 2017: 1 paper · 2018: none · 2019: none · 2020: 1 paper · 2021: 2 papers · 2022: 1 paper
Papers 6
- MicroRNA‑190b expression predicts a good prognosis and attenuates the malignant progression of pancreatic cancer by targeting MEF2C and TCF4. Co-author DOI 10.3892/or.2021.8223↗
- m6A demethylase FTO suppresses pancreatic cancer tumorigenesis by demethylating PJA2 and inhibiting Wnt signaling. Co-author DOI 10.1016/j.omtn.2021.06.005↗
- Genetic alterations and functional networks of m6A RNA methylation regulators in pancreatic cancer based on data mining. Co-author DOI 10.1186/s12967-021-03001-2↗
- Regulatory Network Analysis Reveals Prognosis-Related CircRNAs in Pancreatic Cancer Cells First author DOI 10.21203/rs.3.rs-122183/v1↗
- HPCDb: an integrated database of pancreatic cancer Co-author DOI 10.1101/169771↗
- Sp1-driven up-regulation of miR-19a decreases RHOB and promotes pancreatic cancer. Co-author DOI 10.18632/oncotarget.3975↗
Closest collaborators
The 7 strongest of 7 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Xianghong Yang · 5 shared papers, 2015–2022 · strength 0.63
- Paper MicroRNA‑190b expression predicts a good prognosis and attenuates the malignant progression of pancreatic cancer by tar… DOI 10.3892/or.2021.8223↗
- Paper m6A demethylase FTO suppresses pancreatic cancer tumorigenesis by demethylating PJA2 and inhibiting Wnt signaling. DOI 10.1016/j.omtn.2021.06.005↗
- Paper Genetic alterations and functional networks of m6A RNA methylation regulators in pancreatic cancer based on data mining. DOI 10.1186/s12967-021-03001-2↗
- Heying Zhang · 5 shared papers, 2015–2021 · strength 0.55
- Paper m6A demethylase FTO suppresses pancreatic cancer tumorigenesis by demethylating PJA2 and inhibiting Wnt signaling. DOI 10.1016/j.omtn.2021.06.005↗
- Paper Genetic alterations and functional networks of m6A RNA methylation regulators in pancreatic cancer based on data mining. DOI 10.1186/s12967-021-03001-2↗
- Paper Regulatory Network Analysis Reveals Prognosis-Related CircRNAs in Pancreatic Cancer Cells DOI 10.21203/rs.3.rs-122183/v1↗
- Yonggang Tan · 5 shared papers, 2015–2021 · strength 0.55
- Paper m6A demethylase FTO suppresses pancreatic cancer tumorigenesis by demethylating PJA2 and inhibiting Wnt signaling. DOI 10.1016/j.omtn.2021.06.005↗
- Paper Genetic alterations and functional networks of m6A RNA methylation regulators in pancreatic cancer based on data mining. DOI 10.1186/s12967-021-03001-2↗
- Paper Regulatory Network Analysis Reveals Prognosis-Related CircRNAs in Pancreatic Cancer Cells DOI 10.21203/rs.3.rs-122183/v1↗
- Yunwei Li · 3 shared papers, 2021–2022 · strength 0.52
- Paper MicroRNA‑190b expression predicts a good prognosis and attenuates the malignant progression of pancreatic cancer by tar… DOI 10.3892/or.2021.8223↗
- Paper m6A demethylase FTO suppresses pancreatic cancer tumorigenesis by demethylating PJA2 and inhibiting Wnt signaling. DOI 10.1016/j.omtn.2021.06.005↗
- Paper Genetic alterations and functional networks of m6A RNA methylation regulators in pancreatic cancer based on data mining. DOI 10.1186/s12967-021-03001-2↗
- Huawei Zou · 3 shared papers, 2015–2020 · strength 0.24
- Paper Regulatory Network Analysis Reveals Prognosis-Related CircRNAs in Pancreatic Cancer Cells DOI 10.21203/rs.3.rs-122183/v1↗
- Paper HPCDb: an integrated database of pancreatic cancer DOI 10.1101/169771↗
- Paper Sp1-driven up-regulation of miR-19a decreases RHOB and promotes pancreatic cancer. DOI 10.18632/oncotarget.3975↗
- Dan Li · 1 shared paper, 2017 · strength 0.07
- Wei Cao · 1 shared paper, 2015 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.