ResearcherUniversity of Nebraska Medical CenterNo recent work listed
Sakthivel Muniyan
Matched on ORCID iD 0000-0001-9405-7857; OpenAlex author ID A5005791120. Sources: OpenAlex.
Their work
- Papers
- 7
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 39
2016: 2 papers · 2017: none · 2018: none · 2019: none · 2020: 1 paper · 2021: 2 papers · 2022: 2 papers
Papers 7
- Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41388-022-02493-6↗
- MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. Co-author DOI 10.1158/1541-7786.mcr-21-0888↗
- Nuclear factor kappa-B contributes to cigarette smoke tolerance in pancreatic ductal adenocarcinoma through cysteine metabolism. Co-author DOI 10.1016/j.biopha.2021.112312↗
- SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1128/mcb.00135-21↗
- Acinar transformed ductal cells exhibit differential mucin expression in a tamoxifen-induced pancreatic ductal adenocarcinoma mouse model. Co-author DOI 10.1242/bio.052878↗
- MUC16 contributes to the metastasis of pancreatic ductal adenocarcinoma through focal adhesion mediated signaling mechanism. Co-author DOI 10.18632/genesandcancer.104↗
- Overexpression of PD2 leads to increased tumorigenicity and metastasis in pancreatic ductal adenocarcinoma. Co-author DOI 10.18632/oncotarget.6580↗
Closest collaborators
The 25 strongest of 39 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Surinder K. Batra · 7 shared papers, 2016–2022 · strength 0.70
- Paper Nuclear factor kappa-B contributes to cigarette smoke tolerance in pancreatic ductal adenocarcinoma through cysteine me… DOI 10.1016/j.biopha.2021.112312↗
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Satyanarayana Rachagani · 5 shared papers, 2016–2022 · strength 0.46
- Paper Nuclear factor kappa-B contributes to cigarette smoke tolerance in pancreatic ductal adenocarcinoma through cysteine me… DOI 10.1016/j.biopha.2021.112312↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Paper Acinar transformed ductal cells exhibit differential mucin expression in a tamoxifen-induced pancreatic ductal adenocar… DOI 10.1242/bio.052878↗
- Moorthy Palanimuthu Ponnusamy · 5 shared papers, 2016–2022 · strength 0.46
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Parthasarathy Seshacharyulu · 4 shared papers, 2016–2022 · strength 0.39
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Paper Acinar transformed ductal cells exhibit differential mucin expression in a tamoxifen-induced pancreatic ductal adenocar… DOI 10.1242/bio.052878↗
- Sanchita Rauth · 3 shared papers, 2021–2022 · strength 0.35
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Ashu Shah · 3 shared papers, 2021–2022 · strength 0.35
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Rama Krishna Nimmakayala · 3 shared papers, 2021–2022 · strength 0.35
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Kavita B. Mallya · 3 shared papers, 2020–2022 · strength 0.35
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Paper Acinar transformed ductal cells exhibit differential mucin expression in a tamoxifen-induced pancreatic ductal adenocar… DOI 10.1242/bio.052878↗
- Imayavaramban Lakshmanan · 4 shared papers, 2016–2022 · strength 0.35
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Paper MUC16 contributes to the metastasis of pancreatic ductal adenocarcinoma through focal adhesion mediated signaling mecha… DOI 10.18632/genesandcancer.104↗
- Dhanya Haridas · 3 shared papers, 2016–2022 · strength 0.28
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Paper Acinar transformed ductal cells exhibit differential mucin expression in a tamoxifen-induced pancreatic ductal adenocar… DOI 10.1242/bio.052878↗
- Paper MUC16 contributes to the metastasis of pancreatic ductal adenocarcinoma through focal adhesion mediated signaling mecha… DOI 10.18632/genesandcancer.104↗
- Pranita Atri · 2 shared papers, 2022 · strength 0.24
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Namita Bhyravbhatla · 2 shared papers, 2022 · strength 0.24
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Shailendra Kumar Gautam · 2 shared papers, 2022 · strength 0.24
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Saravanakumar Marimuthu · 2 shared papers, 2022 · strength 0.24
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Maneesh Jain · 2 shared papers, 2022 · strength 0.24
- Paper MUC16 Promotes Liver Metastasis of Pancreatic Ductal Adenocarcinoma by Upregulating NRP2-Associated Cell Adhesion. DOI 10.1158/1541-7786.mcr-21-0888↗
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Lynette M. Smith · 3 shared papers, 2016–2022 · strength 0.22
- Paper Muc16 depletion diminishes KRAS-induced tumorigenesis and metastasis by altering tumor microenvironment factors in panc… DOI 10.1038/s41388-022-02493-6↗
- Paper MUC16 contributes to the metastasis of pancreatic ductal adenocarcinoma through focal adhesion mediated signaling mecha… DOI 10.18632/genesandcancer.104↗
- Paper Overexpression of PD2 leads to increased tumorigenicity and metastasis in pancreatic ductal adenocarcinoma. DOI 10.18632/oncotarget.6580↗
- Saswati Karmakar · 2 shared papers, 2016–2021 · strength 0.17
- Daryl J. Murry · 1 shared paper, 2021 · strength 0.13
- Venugopal Gunda · 1 shared paper, 2021 · strength 0.13
- Nagabhishek Sirpu Natesh · 1 shared paper, 2021 · strength 0.13
- Michael Anthony Hollingsworth · 1 shared paper, 2022 · strength 0.13
- Paul M. Grandgenett · 1 shared paper, 2022 · strength 0.13
- Koelina Ganguly · 1 shared paper, 2021 · strength 0.11
- Sushil Kumar · 1 shared paper, 2021 · strength 0.11
- Chi H. Lin · 1 shared paper, 2021 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.