ResearcherBirmingham VA Medical CenterNo recent work listed
Yabing Chen
Matched on ORCID iD 0000-0001-9676-6657; OpenAlex author ID A5038178209. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 0, last author 6)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 8
2011: 1 paper · 2012: none · 2013: 1 paper · 2014: none · 2015: 1 paper · 2016: 1 paper · 2017: 1 paper · 2018: none · 2019: 1 paper · 2020: 1 paper
Papers 7
- Regulation of pancreatic cancer TRAIL resistance by protein O-GlcNAcylation. Last author DOI 10.1038/s41374-019-0365-z↗
- Cytoplasmic PARP-1 promotes pancreatic cancer tumorigenesis and resistance. Last author DOI 10.1002/ijc.32108↗
- The long non-coding RNA HOTAIR enhances pancreatic cancer resistance to TNF-related apoptosis-inducing ligand. Last author DOI 10.1074/jbc.m117.786830↗
- Identification of initial leads directed at the calmodulin-binding region on the Src-SH2 domain that exhibit anti-proliferation activity against pancreatic can… Co-author DOI 10.1016/j.bmcl.2016.01.027↗
- Calmodulin antagonists promote TRA-8 therapy of resistant pancreatic cancer. Last author DOI 10.18632/oncotarget.4490↗
- PARP-1 regulates resistance of pancreatic cancer to TRAIL therapy. Last author DOI 10.1158/1078-0432.ccr-13-0516↗
- Calmodulin mediates Fas-induced FADD-independent survival signaling in pancreatic cancer cells via activation of Src-extracellular signal-regulated kinase (ERK… Last author DOI 10.1074/jbc.m110.202804↗
Closest collaborators
The 8 strongest of 8 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Tong Zhou · 5 shared papers, 2013–2020 · strength 0.41
- Paper Regulation of pancreatic cancer TRAIL resistance by protein O-GlcNAcylation. DOI 10.1038/s41374-019-0365-z↗
- Paper Cytoplasmic PARP-1 promotes pancreatic cancer tumorigenesis and resistance. DOI 10.1002/ijc.32108↗
- Paper The long non-coding RNA HOTAIR enhances pancreatic cancer resistance to TNF-related apoptosis-inducing ligand. DOI 10.1074/jbc.m117.786830↗
- Fei Xu · 4 shared papers, 2015–2020 · strength 0.37
- Paper Regulation of pancreatic cancer TRAIL resistance by protein O-GlcNAcylation. DOI 10.1038/s41374-019-0365-z↗
- Paper Cytoplasmic PARP-1 promotes pancreatic cancer tumorigenesis and resistance. DOI 10.1002/ijc.32108↗
- Paper The long non-coding RNA HOTAIR enhances pancreatic cancer resistance to TNF-related apoptosis-inducing ligand. DOI 10.1074/jbc.m117.786830↗
- Yong Sun · 4 shared papers, 2013–2020 · strength 0.33
- Paper Regulation of pancreatic cancer TRAIL resistance by protein O-GlcNAcylation. DOI 10.1038/s41374-019-0365-z↗
- Paper Cytoplasmic PARP-1 promotes pancreatic cancer tumorigenesis and resistance. DOI 10.1002/ijc.32108↗
- Paper Calmodulin antagonists promote TRA-8 therapy of resistant pancreatic cancer. DOI 10.18632/oncotarget.4490↗
- Kaiyu Yuan · 5 shared papers, 2011–2020 · strength 0.31
- Paper Regulation of pancreatic cancer TRAIL resistance by protein O-GlcNAcylation. DOI 10.1038/s41374-019-0365-z↗
- Paper Calmodulin antagonists promote TRA-8 therapy of resistant pancreatic cancer. DOI 10.18632/oncotarget.4490↗
- Paper Identification of initial leads directed at the calmodulin-binding region on the Src-SH2 domain that exhibit anti-proli… DOI 10.1016/j.bmcl.2016.01.027↗
- Shanzhong Yang · 3 shared papers, 2017–2020 · strength 0.31
- Paper Regulation of pancreatic cancer TRAIL resistance by protein O-GlcNAcylation. DOI 10.1038/s41374-019-0365-z↗
- Paper Cytoplasmic PARP-1 promotes pancreatic cancer tumorigenesis and resistance. DOI 10.1002/ijc.32108↗
- Paper The long non-coding RNA HOTAIR enhances pancreatic cancer resistance to TNF-related apoptosis-inducing ligand. DOI 10.1074/jbc.m117.786830↗
- Nirag Chandrakant Jhala · 1 shared paper, 2019 · strength 0.11
- Ywh-Min Tzou · 1 shared paper, 2016 · strength 0.06
- Nepalli Rama Krishna · 1 shared paper, 2016 · strength 0.06
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.