ResearcherUniversity of Electronic Science and Technology of ChinaActive in the last 2 years
Jiao Wu
Matched on ORCID iD 0000-0002-0053-7479; OpenAlex author ID A5103265351. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 4
2021: 2 papers · 2022: 1 paper · 2023: 1 paper · 2024: none · 2025: none · 2026: 1 paper
Papers 5
- Intratumoral microbiome: a key regulator and novel therapeutic target for chemoresistance in pancreatic cancer. First author DOI 10.3389/fcimb.2026.1860523↗
- Effect of STK3 on proliferation and apoptosis of pancreatic cancer cells via PI3K/AKT/mTOR pathway. Co-author DOI 10.1016/j.cellsig.2023.110642↗
- TNFSF9 promotes metastasis of pancreatic cancer by regulating M2 polarization of macrophages through Src/FAK/p-Akt/IL-1β signaling. Co-author DOI 10.1016/j.intimp.2021.108429↗
- TNFSF9 Is a Prognostic Biomarker and Correlated with Immune Infiltrates in Pancreatic Cancer. Co-author DOI 10.1007/s12029-020-00371-6↗
- TNFSF9 promotes metastasis of pancreatic cancer through Wnt/Snail signaling and M2 polarization of macrophages. Co-author DOI 10.18632/aging.203497↗
Closest collaborators
The 4 strongest of 4 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Yunpeng Wang · 1 shared paper, 2026 · strength 0.63
- Zheng Jiang · 2 shared papers, 2021 · strength 0.37
- Zheng Jiang · 2 shared papers, 2022–2023 · strength 0.36
- Paper TNFSF9 promotes metastasis of pancreatic cancer by regulating M2 polarization of macrophages through Src/FAK/p-Akt/IL-1… DOI 10.1016/j.intimp.2021.108429↗
- Paper Effect of STK3 on proliferation and apoptosis of pancreatic cancer cells via PI3K/AKT/mTOR pathway. DOI 10.1016/j.cellsig.2023.110642↗
- Jun Chen · 2 shared papers, 2021–2023 · strength 0.33
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.