Jarosław Daniluk

Closest collaborators

The 21 strongest of 21 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

1. Agnieszka Katarzyna Swidnicka-Siergiejko · 3 shared papers, 2017–2025 · strength 0.50
   • Paper The development of pancreatic cancer is accompanied by significant changes in the immune response in genetically predis… DOI 10.3389/fonc.2025.1603293↗
   • Paper Microbiome-Friend or Foe of Pancreatic Cancer? DOI 10.3390/jcm10235624↗
   • Paper Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53. DOI 10.1038/onc.2016.461↗
2. Urszula Daniluk · 2 shared papers, 2021–2025 · strength 0.44
   • Paper The development of pancreatic cancer is accompanied by significant changes in the immune response in genetically predis… DOI 10.3389/fonc.2025.1603293↗
   • Paper Microbiome-Friend or Foe of Pancreatic Cancer? DOI 10.3390/jcm10235624↗
3. A Dâbrowski · 2 shared papers, 2021–2025 · strength 0.44
   • Paper The development of pancreatic cancer is accompanied by significant changes in the immune response in genetically predis… DOI 10.3389/fonc.2025.1603293↗
   • Paper Microbiome-Friend or Foe of Pancreatic Cancer? DOI 10.3390/jcm10235624↗
4. Wiktoria Maria Izdebska · 1 shared paper, 2023 · strength 0.31
   • Paper Microbiome and MicroRNA or Long Non-Coding RNA-Two Modern Approaches to Understanding Pancreatic Ductal Adenocarcinoma. DOI 10.3390/jcm12175643↗
5. Jacek Nikliński · 1 shared paper, 2023 · strength 0.31
   • Paper Microbiome and MicroRNA or Long Non-Coding RNA-Two Modern Approaches to Understanding Pancreatic Ductal Adenocarcinoma. DOI 10.3390/jcm12175643↗
6. Katarzyna Guzińska-Ustymowicz · 1 shared paper, 2025 · strength 0.27
   • Paper The development of pancreatic cancer is accompanied by significant changes in the immune response in genetically predis… DOI 10.3389/fonc.2025.1603293↗
7. Anna Pryczynicz · 1 shared paper, 2025 · strength 0.27
   • Paper The development of pancreatic cancer is accompanied by significant changes in the immune response in genetically predis… DOI 10.3389/fonc.2025.1603293↗
8. Zobeida Cruz‐Monserrate · 2 shared papers, 2013–2017 · strength 0.09
   • Paper Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53. DOI 10.1038/onc.2016.461↗
   • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
9. Baoan Ji · 2 shared papers, 2013–2017 · strength 0.09
   • Paper Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53. DOI 10.1038/onc.2016.461↗
   • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
10. Craig D. Logsdon · 2 shared papers, 2013–2017 · strength 0.09
    • Paper Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53. DOI 10.1038/onc.2016.461↗
    • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
11. Sobeyda B. Gomez-Chou · 1 shared paper, 2017 · strength 0.06
    • Paper Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53. DOI 10.1038/onc.2016.461↗
12. Nancy G. Azizian · 1 shared paper, 2017 · strength 0.06
    • Paper Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53. DOI 10.1038/onc.2016.461↗
13. Haojie Huang · 1 shared paper, 2017 · strength 0.06
    • Paper Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53. DOI 10.1038/onc.2016.461↗
14. Anirban Maitra · 1 shared paper, 2017 · strength 0.06
    • Paper Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53. DOI 10.1038/onc.2016.461↗
15. Bincy Philip · 1 shared paper, 2013 · strength 0.03
    • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
16. Huamin Wang · 1 shared paper, 2013 · strength 0.03
    • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
17. Deyali Chatterjee · 1 shared paper, 2013 · strength 0.03
    • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
18. Yan Liu · 1 shared paper, 2013 · strength 0.03
    • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
19. Christina Lynn Roland · 1 shared paper, 2013 · strength 0.03
    • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
20. Jason B. Fleming · 1 shared paper, 2013 · strength 0.03
    • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗
21. Haojie Huang · 1 shared paper, 2013 · strength 0.03
    • Paper A high-fat diet activates oncogenic Kras and COX2 to induce development of pancreatic ductal adenocarcinoma in mice. DOI 10.1053/j.gastro.2013.08.018↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.