ResearcherShanghai Jiao Tong UniversityNo recent work listed
Kexian Li
Matched on ORCID iD 0000-0002-1293-5538; OpenAlex author ID A5103019656. Sources: OpenAlex.
Their work
- Papers
- 11 (first author 7, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 21
2020: 1 paper · 2021: 5 papers · 2022: 3 papers · 2023: 2 papers
Papers 11
- Retraction Note: The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modification of mRNA. First author DOI 10.1186/s13045-023-01506-5↗
- LINC01137 facilitates pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis First author DOI 10.21203/rs.3.rs-2411868/v1↗
- The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modification of mRNA. First author DOI 10.1186/s13045-022-01338-9↗
- LINC01137 involvement in pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis First author DOI 10.21203/rs.3.rs-1516835/v1↗
- LINC01137 facilitate pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis Co-author DOI 10.21203/rs.3.rs-2065916/v1↗
- A Novel DNA Replication-Related Signature Predicting Recurrence After R0 Resection of Pancreatic Ductal Adenocarcinoma: Prognostic Value and Clinical Implicati… First author DOI 10.3389/fcell.2021.619549↗
- An EMT-Related Gene Signature for Predicting Response to Adjuvant Chemotherapy in Pancreatic Ductal Adenocarcinoma. First author DOI 10.3389/fcell.2021.665161↗
- A Novel Ferroptosis-Related Gene Signature Predicts Recurrence in Patients With Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3389/fmolb.2021.650264↗
- Transcriptomic Profiling Identifies DCBLD2 as a Diagnostic and Prognostic Biomarker in Pancreatic Ductal Adenocarcinoma. First author DOI 10.3389/fmolb.2021.659168↗
- Development and Validation of a 7-Gene Prognostic Signature to Improve Survival Prediction in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3389/fmolb.2021.676291↗
- Development and validation of a cancer stem cell-related signature for prognostic prediction in pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s12967-020-02527-1↗
Closest collaborators
The 21 strongest of 21 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Zengyu Feng · 11 shared papers, 2020–2023 · strength 1.62
- Paper An EMT-Related Gene Signature for Predicting Response to Adjuvant Chemotherapy in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fcell.2021.665161↗
- Paper Retraction Note: The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modificat… DOI 10.1186/s13045-023-01506-5↗
- Paper A Novel DNA Replication-Related Signature Predicting Recurrence After R0 Resection of Pancreatic Ductal Adenocarcinoma:… DOI 10.3389/fcell.2021.619549↗
- Chenghong Peng · 10 shared papers, 2021–2023 · strength 1.50
- Paper An EMT-Related Gene Signature for Predicting Response to Adjuvant Chemotherapy in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fcell.2021.665161↗
- Paper Retraction Note: The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modificat… DOI 10.1186/s13045-023-01506-5↗
- Paper A Novel DNA Replication-Related Signature Predicting Recurrence After R0 Resection of Pancreatic Ductal Adenocarcinoma:… DOI 10.3389/fcell.2021.619549↗
- Yulian Wu · 5 shared papers, 2021 · strength 0.79
- Paper An EMT-Related Gene Signature for Predicting Response to Adjuvant Chemotherapy in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fcell.2021.665161↗
- Paper A Novel DNA Replication-Related Signature Predicting Recurrence After R0 Resection of Pancreatic Ductal Adenocarcinoma:… DOI 10.3389/fcell.2021.619549↗
- Paper Transcriptomic Profiling Identifies DCBLD2 as a Diagnostic and Prognostic Biomarker in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fmolb.2021.659168↗
- Kai Qin · 5 shared papers, 2022–2023 · strength 0.70
- Paper Retraction Note: The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modificat… DOI 10.1186/s13045-023-01506-5↗
- Paper LINC01137 facilitates pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2411868/v1↗
- Paper The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modification of mRNA. DOI 10.1186/s13045-022-01338-9↗
- Lingxi Jiang · 5 shared papers, 2020–2023 · strength 0.68
- Paper Retraction Note: The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modificat… DOI 10.1186/s13045-023-01506-5↗
- Paper The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modification of mRNA. DOI 10.1186/s13045-022-01338-9↗
- Paper LINC01137 involvement in pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-1516835/v1↗
- Hao Chen · 3 shared papers, 2022–2023 · strength 0.45
- Paper Retraction Note: The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modificat… DOI 10.1186/s13045-023-01506-5↗
- Paper LINC01137 facilitates pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2411868/v1↗
- Paper The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modification of mRNA. DOI 10.1186/s13045-022-01338-9↗
- Jiewei Lin · 3 shared papers, 2022–2023 · strength 0.41
- Paper LINC01137 facilitates pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2411868/v1↗
- Paper LINC01137 involvement in pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-1516835/v1↗
- Paper LINC01137 facilitate pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2065916/v1↗
- Lijie Han · 3 shared papers, 2022–2023 · strength 0.41
- Paper LINC01137 facilitates pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2411868/v1↗
- Paper LINC01137 involvement in pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-1516835/v1↗
- Paper LINC01137 facilitate pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2065916/v1↗
- Yizhi Cao · 3 shared papers, 2022–2023 · strength 0.41
- Paper LINC01137 facilitates pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2411868/v1↗
- Paper LINC01137 involvement in pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-1516835/v1↗
- Paper LINC01137 facilitate pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2065916/v1↗
- Yongsheng Jiang · 3 shared papers, 2022–2023 · strength 0.41
- Paper LINC01137 facilitates pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2411868/v1↗
- Paper LINC01137 involvement in pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-1516835/v1↗
- Paper LINC01137 facilitate pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2065916/v1↗
- Hao Chen · 3 shared papers, 2020–2022 · strength 0.38
- Paper LINC01137 involvement in pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-1516835/v1↗
- Paper LINC01137 facilitate pancreatic cancer stemness via the miR-7155-5p/KLF12/AKT axis DOI 10.21203/rs.3.rs-2065916/v1↗
- Paper Development and validation of a cancer stem cell-related signature for prognostic prediction in pancreatic ductal adeno… DOI 10.1186/s12967-020-02527-1↗
- Jiancheng Wang · 2 shared papers, 2022–2023 · strength 0.28
- Jiaxin Luo · 2 shared papers, 2022–2023 · strength 0.28
- Baiyong Shen · 1 shared paper, 2023 · strength 0.16
- Hao Qian · 1 shared paper, 2021 · strength 0.16
- L. Jiang · 1 shared paper, 2023 · strength 0.15
- Tao Li · 1 shared paper, 2021 · strength 0.15
- Baiyong Shen · 1 shared paper, 2022 · strength 0.14
- Peng Chen · 1 shared paper, 2022 · strength 0.13
- Jiancheng Wang · 1 shared paper, 2022 · strength 0.13
- Chenghong Peng · 1 shared paper, 2020 · strength 0.13
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.