ResearcherShanghai Medical College of Fudan UniversityActive in the last 2 years
Zheng Zhang
Matched on ORCID iD 0000-0002-1396-2634; OpenAlex author ID A5100459096. Sources: OpenAlex.
Their work
- Papers
- 18
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 35
2016: 1 paper · 2017: none · 2018: 2 papers · 2019: 5 papers · 2020: 3 papers · 2021: 4 papers · 2022: 1 paper · 2023: 1 paper · 2024: 1 paper
Papers 18
- Abrogation of KLF5 sensitizes BRCA1-proficient pancreatic cancer to PARP inhibition. Co-author DOI 10.3724/abbs.2023288↗
- Reshaping the Pancreatic Cancer Microenvironment at Different Stages with Chemotherapy. Co-author DOI 10.3390/cancers15092448↗
- The Crosstalk Between Immune Infiltration, Circulating Tumor Cells, and Metastasis in Pancreatic Cancer: Identification of HMGB3 From a Multiple Omics Analysis. Co-author DOI 10.3389/fgene.2022.892177↗
- Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic cancer. Co-author DOI 10.1002/ctm2.398↗
- SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2020.11.023↗
- FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metastasis of pancreatic cancer. Co-author DOI 10.1093/abbs/gmab074↗
- MTAP Deficiency-Induced Metabolic Reprogramming Creates a Vulnerability to Cotargeting De Novo Purine Synthesis and Glycolysis in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-20-0414↗
- Oncogenic function of TRIM2 in pancreatic cancer by activating ROS-related NRF2/ITGB7/FAK axis. Co-author DOI 10.1038/s41388-020-01452-3↗
- Pin1 promotes pancreatic cancer progression and metastasis by activation of NF-κB-IL-18 feedback loop. Co-author DOI 10.1111/cpr.12816↗
- Abrogation of ARF6 promotes RSL3-induced ferroptosis and mitigates gemcitabine resistance in pancreatic cancer cells. Co-author PMID 32368394↗
- UHRF1 promotes aerobic glycolysis and proliferation via suppression of SIRT4 in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2019.03.024↗
- Homeodomain-interacting protein kinase 2 suppresses proliferation and aerobic glycolysis via ERK/cMyc axis in pancreatic cancer. Co-author DOI 10.1111/cpr.12603↗
- Role of hepatocyte nuclear factor 4 alpha in cell proliferation and gemcitabine resistance in pancreatic adenocarcinoma. Co-author DOI 10.1186/s12935-019-0767-4↗
- PRMT5 enhances tumorigenicity and glycolysis in pancreatic cancer via the FBW7/cMyc axis. Co-author DOI 10.1186/s12964-019-0344-4↗
- FGFBP1, a downstream target of the FBW7/c-Myc axis, promotes cell proliferation and migration in pancreatic cancer. Co-author PMID 31911852↗
- Role of angiogenesis in pancreatic cancer biology and therapy. Co-author DOI 10.1016/j.biopha.2018.09.136↗
- dCK negatively regulates the NRF2/ARE axis and ROS production in pancreatic cancer. Co-author DOI 10.1111/cpr.12456↗
- Inhibiting tumor necrosis factor-alpha diminishes desmoplasia and inflammation to overcome chemoresistance in pancreatic ductal adenocarcinoma. Co-author DOI 10.18632/oncotarget.13212↗
Closest collaborators
The 25 strongest of 35 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Shunrong Ji · 14 shared papers, 2018–2021 · strength 1.36
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic ca… DOI 10.1002/ctm2.398↗
- Yi Qin · 14 shared papers, 2018–2021 · strength 1.36
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic ca… DOI 10.1002/ctm2.398↗
- Qiangsheng Hu · 13 shared papers, 2018–2021 · strength 1.27
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic ca… DOI 10.1002/ctm2.398↗
- Wensheng Liu · 13 shared papers, 2018–2021 · strength 1.27
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic ca… DOI 10.1002/ctm2.398↗
- Xianjun Yu · 13 shared papers, 2018–2021 · strength 1.26
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic ca… DOI 10.1002/ctm2.398↗
- Xiaowu Xu · 12 shared papers, 2019–2021 · strength 1.19
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic ca… DOI 10.1002/ctm2.398↗
- Wenyan Xu · 11 shared papers, 2018–2021 · strength 1.06
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper MTAP Deficiency-Induced Metabolic Reprogramming Creates a Vulnerability to Cotargeting De Novo Purine Synthesis and Gly… DOI 10.1158/0008-5472.can-20-0414↗
- Mengqi Liu · 8 shared papers, 2019–2021 · strength 0.81
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper MTAP Deficiency-Induced Metabolic Reprogramming Creates a Vulnerability to Cotargeting De Novo Purine Synthesis and Gly… DOI 10.1158/0008-5472.can-20-0414↗
- Qiqing Sun · 9 shared papers, 2018–2020 · strength 0.80
- Paper Oncogenic function of TRIM2 in pancreatic cancer by activating ROS-related NRF2/ITGB7/FAK axis. DOI 10.1038/s41388-020-01452-3↗
- Paper Pin1 promotes pancreatic cancer progression and metastasis by activation of NF-κB-IL-18 feedback loop. DOI 10.1111/cpr.12816↗
- Paper Abrogation of ARF6 promotes RSL3-induced ferroptosis and mitigates gemcitabine resistance in pancreatic cancer cells. PMID 32368394↗
- Qifeng Zhuo · 6 shared papers, 2020–2021 · strength 0.63
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic ca… DOI 10.1002/ctm2.398↗
- Paper MTAP Deficiency-Induced Metabolic Reprogramming Creates a Vulnerability to Cotargeting De Novo Purine Synthesis and Gly… DOI 10.1158/0008-5472.can-20-0414↗
- Ye Zeng · 5 shared papers, 2020–2021 · strength 0.53
- Paper FGFBP1-mediated crosstalk between fibroblasts and pancreatic cancer cells via FGF22/FGFR2 promotes invasion and metasta… DOI 10.1093/abbs/gmab074↗
- Paper Improved tumor control with antiangiogenic therapy after treatment with gemcitabine and nab-paclitaxel in pancreatic ca… DOI 10.1002/ctm2.398↗
- Paper Oncogenic function of TRIM2 in pancreatic cancer by activating ROS-related NRF2/ITGB7/FAK axis. DOI 10.1038/s41388-020-01452-3↗
- Wenquan Wang · 2 shared papers, 2023–2024 · strength 0.47
- Jin Xu · 4 shared papers, 2018–2019 · strength 0.33
- Paper Role of angiogenesis in pancreatic cancer biology and therapy. DOI 10.1016/j.biopha.2018.09.136↗
- Paper Homeodomain-interacting protein kinase 2 suppresses proliferation and aerobic glycolysis via ERK/cMyc axis in pancreati… DOI 10.1111/cpr.12603↗
- Paper PRMT5 enhances tumorigenicity and glycolysis in pancreatic cancer via the FBW7/cMyc axis. DOI 10.1186/s12964-019-0344-4↗
- Quanxing Ni · 4 shared papers, 2018–2019 · strength 0.33
- Paper UHRF1 promotes aerobic glycolysis and proliferation via suppression of SIRT4 in pancreatic cancer. DOI 10.1016/j.canlet.2019.03.024↗
- Paper Homeodomain-interacting protein kinase 2 suppresses proliferation and aerobic glycolysis via ERK/cMyc axis in pancreati… DOI 10.1111/cpr.12603↗
- Paper PRMT5 enhances tumorigenicity and glycolysis in pancreatic cancer via the FBW7/cMyc axis. DOI 10.1186/s12964-019-0344-4↗
- Ye Zeng · 3 shared papers, 2019–2021 · strength 0.31
- Paper SETD8 potentiates constitutive ERK1/2 activation via epigenetically silencing DUSP10 expression in pancreatic cancer. DOI 10.1016/j.canlet.2020.11.023↗
- Paper MTAP Deficiency-Induced Metabolic Reprogramming Creates a Vulnerability to Cotargeting De Novo Purine Synthesis and Gly… DOI 10.1158/0008-5472.can-20-0414↗
- Paper FGFBP1, a downstream target of the FBW7/c-Myc axis, promotes cell proliferation and migration in pancreatic cancer. PMID 31911852↗
- Maozhen Peng · 1 shared paper, 2023 · strength 0.24
- Liang Liu · 1 shared paper, 2023 · strength 0.24
- Jinbin Jia · 1 shared paper, 2024 · strength 0.23
- Liang Liu · 1 shared paper, 2024 · strength 0.23
- Zijin Xu · 1 shared paper, 2024 · strength 0.23
- Yun Jin · 1 shared paper, 2024 · strength 0.23
- Jiahua Zhou · 1 shared paper, 2022 · strength 0.16
- Haodong Tang · 1 shared paper, 2022 · strength 0.16
- Bo Zhang · 2 shared papers, 2018–2019 · strength 0.16
- Mengqi Liu · 1 shared paper, 2021 · strength 0.12
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.