ResearcherKyushu UniversityNo recent work listed
Motofumi Kumazoe
Matched on ORCID iD 0000-0002-3002-2994; OpenAlex author ID A5009883829. Sources: OpenAlex.
Their work
- Papers
- 4 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 6
2017: 3 papers · 2018: none · 2019: none · 2020: none · 2021: none · 2022: none · 2023: 1 paper
Papers 4
- PPAR/PDK4 pathway is involved in the anticancer effects of cGMP in pancreatic cancer. First author DOI 10.1016/j.bbrc.2023.06.043↗
- FOXO3 is essential for CD44 expression in pancreatic cancer cells. Co-author DOI 10.1038/onc.2016.426↗
- PDE3 inhibitor and EGCG combination treatment suppress cancer stem cell properties in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41598-017-02162-9↗
- The FOXO3/PGC-1β signaling axis is essential for cancer stem cell properties of pancreatic ductal adenocarcinoma. Co-author DOI 10.1074/jbc.m116.772111↗
Closest collaborators
The 6 strongest of 6 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hirofumi Tachibana · 4 shared papers, 2017–2023 · strength 0.38
- Paper PPAR/PDK4 pathway is involved in the anticancer effects of cGMP in pancreatic cancer. DOI 10.1016/j.bbrc.2023.06.043↗
- Paper The FOXO3/PGC-1β signaling axis is essential for cancer stem cell properties of pancreatic ductal adenocarcinoma. DOI 10.1074/jbc.m116.772111↗
- Paper FOXO3 is essential for CD44 expression in pancreatic cancer cells. DOI 10.1038/onc.2016.426↗
- Mai Yamashita · 2 shared papers, 2017–2023 · strength 0.26
- Yoshinori Fujimura · 1 shared paper, 2023 · strength 0.20
- Masaharu Murata · 1 shared paper, 2017 · strength 0.06
- Shuntaro Tsukamoto · 1 shared paper, 2017 · strength 0.06
- Takashi Takahashi · 1 shared paper, 2017 · strength 0.06
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.