ResearcherSecond Military Medical UniversityNo recent work listed
Dejun Yang
Matched on ORCID iD 0000-0002-3489-4677; OpenAlex author ID A5101499028. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 0, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 14
2016: 2 papers · 2017: 1 paper · 2018: 2 papers · 2019: 1 paper · 2020: 1 paper
Papers 7
- FAM84B, amplified in pancreatic ductal adenocarcinoma, promotes tumorigenesis through the Wnt/β-catenin pathway. Last author DOI 10.18632/aging.103044↗
- MiR-760 enhances sensitivity of pancreatic cancer cells to gemcitabine through modulating Integrin β1. Co-author DOI 10.1042/bsr20192358↗
- Integrin β1 promotes gemcitabine resistance in pancreatic cancer through Cdc42 activation of PI3K p110β signaling. Co-author DOI 10.1016/j.bbrc.2018.09.061↗
- CDKN2B deletion is essential for pancreatic cancer development instead of unmeaningful co-deletion due to juxtaposition to CDKN2A. Co-author DOI 10.1038/onc.2017.316↗
- High Expression of Cell Division Cycle 42 Promotes Pancreatic Cancer Growth and Predicts Poor Outcome of Pancreatic Cancer Patients. Co-author DOI 10.1007/s10620-017-4451-z↗
- Low tristetraprolin expression promotes cell proliferation and predicts poor patients outcome in pancreatic cancer. Co-author DOI 10.18632/oncotarget.7397↗
- Deregulation of MicroRNA-375 inhibits cancer proliferation migration and chemosensitivity in pancreatic cancer through the association of HOXB3. Co-author PMID 27186281↗
Closest collaborators
The 14 strongest of 14 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Qingping Cai · 3 shared papers, 2016–2020 · strength 0.28
- Paper FAM84B, amplified in pancreatic ductal adenocarcinoma, promotes tumorigenesis through the Wnt/β-catenin pathway. DOI 10.18632/aging.103044↗
- Paper MiR-760 enhances sensitivity of pancreatic cancer cells to gemcitabine through modulating Integrin β1. DOI 10.1042/bsr20192358↗
- Paper Low tristetraprolin expression promotes cell proliferation and predicts poor patients outcome in pancreatic cancer. DOI 10.18632/oncotarget.7397↗
- Ronglin Yan · 3 shared papers, 2016–2020 · strength 0.25
- Paper FAM84B, amplified in pancreatic ductal adenocarcinoma, promotes tumorigenesis through the Wnt/β-catenin pathway. DOI 10.18632/aging.103044↗
- Paper High Expression of Cell Division Cycle 42 Promotes Pancreatic Cancer Growth and Predicts Poor Outcome of Pancreatic Can… DOI 10.1007/s10620-017-4451-z↗
- Paper Deregulation of MicroRNA-375 inhibits cancer proliferation migration and chemosensitivity in pancreatic cancer through … PMID 27186281↗
- Zhenxin Zhu · 2 shared papers, 2016–2019 · strength 0.17
- Qingping Cai · 2 shared papers, 2017–2018 · strength 0.16
- Paper Integrin β1 promotes gemcitabine resistance in pancreatic cancer through Cdc42 activation of PI3K p110β signaling. DOI 10.1016/j.bbrc.2018.09.061↗
- Paper High Expression of Cell Division Cycle 42 Promotes Pancreatic Cancer Growth and Predicts Poor Outcome of Pancreatic Can… DOI 10.1007/s10620-017-4451-z↗
- Ziran Wei · 2 shared papers, 2016–2017 · strength 0.14
- Paper High Expression of Cell Division Cycle 42 Promotes Pancreatic Cancer Growth and Predicts Poor Outcome of Pancreatic Can… DOI 10.1007/s10620-017-4451-z↗
- Paper Low tristetraprolin expression promotes cell proliferation and predicts poor patients outcome in pancreatic cancer. DOI 10.18632/oncotarget.7397↗
- Xin Zhang · 1 shared paper, 2020 · strength 0.11
- Yu Xin Zhang · 1 shared paper, 2017 · strength 0.07
- Xudong Zhao · 1 shared paper, 2018 · strength 0.07
- Qiu Tu · 1 shared paper, 2018 · strength 0.07
- Ren Lai · 1 shared paper, 2018 · strength 0.07
- Bin Sun · 1 shared paper, 2018 · strength 0.07
- Xin Zhang · 1 shared paper, 2016 · strength 0.07
- Xin Zhang · 1 shared paper, 2016 · strength 0.07
- Yuexiang Wang · 1 shared paper, 2016 · strength 0.06
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.