ResearcherKobe University HospitalNo recent work listed
Takashi Aoi
Matched on ORCID iD 0000-0002-3963-5922; OpenAlex author ID A5063237654. Sources: OpenAlex.
Their work
- Papers
- 2 (first author 0, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 7
2022: 1 paper · 2023: 1 paper
Papers 2
- Blockage of retinoic acid signaling via RARγ suppressed the proliferation of pancreatic cancer cells by arresting the cell cycle progression of the G1-S phase. Last author DOI 10.1186/s12935-023-02928-4↗
- Increased expression of SPRR1A is associated with a poor prognosis in pancreatic ductal adenocarcinoma. Last author DOI 10.1371/journal.pone.0266620↗
Closest collaborators
The 7 strongest of 7 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Kohei Yamakawa · 2 shared papers, 2022–2023 · strength 0.36
- Paper Blockage of retinoic acid signaling via RARγ suppressed the proliferation of pancreatic cancer cells by arresting the c… DOI 10.1186/s12935-023-02928-4↗
- Paper Increased expression of SPRR1A is associated with a poor prognosis in pancreatic ductal adenocarcinoma. DOI 10.1371/journal.pone.0266620↗
- Yuzo Kodama · 2 shared papers, 2022–2023 · strength 0.36
- Paper Blockage of retinoic acid signaling via RARγ suppressed the proliferation of pancreatic cancer cells by arresting the c… DOI 10.1186/s12935-023-02928-4↗
- Paper Increased expression of SPRR1A is associated with a poor prognosis in pancreatic ductal adenocarcinoma. DOI 10.1371/journal.pone.0266620↗
- Akihito Machinaga · 1 shared paper, 2023 · strength 0.20
- Hiroaki Yanagimoto · 1 shared paper, 2022 · strength 0.16
- Atsuhiro Masuda · 1 shared paper, 2022 · strength 0.16
- Hirochika Toyama · 1 shared paper, 2022 · strength 0.16
- Takumi Fukumoto · 1 shared paper, 2022 · strength 0.16
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.