ResearcherChangzhou No.2 People's HospitalActive in the last 2 years
Chunfu Zhu
Matched on ORCID iD 0000-0002-4363-5781; OpenAlex author ID A5101632309. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 1, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 14
2019: 1 paper · 2020: 3 papers · 2021: 2 papers · 2022: 1 paper · 2023: none · 2024: none · 2025: 1 paper
Papers 8
- tRF-Pro-CGG Suppresses Cell Proliferation and Promotes Apoptosis in Pancreatic Cancer. Co-author DOI 10.1007/s10620-025-08943-x↗
- The biological behavior of tRNA-derived fragment tRF-Leu-AAG in pancreatic cancer cells. Last author DOI 10.1080/21655979.2022.2064206↗
- Low expression of tRF-Pro-CGG predicts poor prognosis in pancreatic ductal adenocarcinoma. Last author DOI 10.1002/jcla.23742↗
- Investigation of Aberrantly Methylated Differentially Expressed Genes in Pancreatic Cancer by Integrated Bioinformatics Analysis Co-author DOI 10.21203/rs.3.rs-132250/v1↗
- TMEM158 promotes pancreatic cancer aggressiveness by activation of TGFβ1 and PI3K/AKT signaling pathway. Co-author DOI 10.1002/jcp.29181↗
- Expression and prognostic analyses of the insulin-like growth factor 2 mRNA binding protein family in human pancreatic cancer. Co-author DOI 10.1186/s12885-020-07590-x↗
- Insight into the effects of microRNA-23a-3p on pancreatic cancer and its underlying molecular mechanism. Co-author DOI 10.3892/ol.2019.11117↗
- Expression profile of tRNA-derived fragments in pancreatic cancer. First author DOI 10.3892/ol.2019.10601↗
Closest collaborators
The 14 strongest of 14 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Lei Jin · 5 shared papers, 2019–2025 · strength 0.83
- Paper tRF-Pro-CGG Suppresses Cell Proliferation and Promotes Apoptosis in Pancreatic Cancer. DOI 10.1007/s10620-025-08943-x↗
- Paper The biological behavior of tRNA-derived fragment tRF-Leu-AAG in pancreatic cancer cells. DOI 10.1080/21655979.2022.2064206↗
- Paper Expression profile of tRNA-derived fragments in pancreatic cancer. DOI 10.3892/ol.2019.10601↗
- Xiaohan Cui · 3 shared papers, 2020–2022 · strength 0.51
- Paper The biological behavior of tRNA-derived fragment tRF-Leu-AAG in pancreatic cancer cells. DOI 10.1080/21655979.2022.2064206↗
- Paper Expression and prognostic analyses of the insulin-like growth factor 2 mRNA binding protein family in human pancreatic … DOI 10.1186/s12885-020-07590-x↗
- Paper Investigation of Aberrantly Methylated Differentially Expressed Genes in Pancreatic Cancer by Integrated Bioinformatics… DOI 10.21203/rs.3.rs-132250/v1↗
- Xihu Qin · 3 shared papers, 2019–2025 · strength 0.51
- Paper tRF-Pro-CGG Suppresses Cell Proliferation and Promotes Apoptosis in Pancreatic Cancer. DOI 10.1007/s10620-025-08943-x↗
- Paper Expression profile of tRNA-derived fragments in pancreatic cancer. DOI 10.3892/ol.2019.10601↗
- Paper Insight into the effects of microRNA-23a-3p on pancreatic cancer and its underlying molecular mechanism. DOI 10.3892/ol.2019.11117↗
- Cailin Xue · 2 shared papers, 2021–2025 · strength 0.39
- Jiadeng Chao · 2 shared papers, 2020–2025 · strength 0.34
- Shizhen Sui · 2 shared papers, 2021–2022 · strength 0.33
- Xihu Qin · 2 shared papers, 2020–2021 · strength 0.31
- Paper Expression and prognostic analyses of the insulin-like growth factor 2 mRNA binding protein family in human pancreatic … DOI 10.1186/s12885-020-07590-x↗
- Paper Investigation of Aberrantly Methylated Differentially Expressed Genes in Pancreatic Cancer by Integrated Bioinformatics… DOI 10.21203/rs.3.rs-132250/v1↗
- Jikuan Zhu · 1 shared paper, 2025 · strength 0.25
- Jun Li · 1 shared paper, 2025 · strength 0.25
- Jun Li · 1 shared paper, 2021 · strength 0.12
- Liming Tang · 1 shared paper, 2020 · strength 0.11
- Yue Fu · 1 shared paper, 2020 · strength 0.11
- Na Yao · 1 shared paper, 2020 · strength 0.11
- Jun Li · 1 shared paper, 2020 · strength 0.10
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.