ResearcherTianjin Medical University Cancer Institute and HospitalActive in the last 2 years
Hui Li
Matched on ORCID iD 0000-0002-4391-9397; OpenAlex author ID A5102899009. Sources: OpenAlex.
Their work
- Papers
- 3 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 14
2024: 1 paper · 2025: none · 2026: 2 papers
Papers 3
- Tumor-Derived Interleukin 35 Promotes Fibrosis in the Tumor Microenvironment of Pancreatic Cancer by Activating Pancreatic Stellate Cells. Co-author DOI 10.1002/advs.202509074↗
- Retraction Note: BICC1 drives pancreatic cancer progression by inducing VEGF-independent angiogenesis. First author DOI 10.1038/s41392-025-02561-9↗
- BICC1 drives pancreatic cancer stemness and chemoresistance by facilitating tryptophan metabolism. First author DOI 10.1126/sciadv.adj8650↗
Closest collaborators
The 14 strongest of 14 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Chongbiao Huang · 3 shared papers, 2024–2026 · strength 0.66
- Paper Tumor-Derived Interleukin 35 Promotes Fibrosis in the Tumor Microenvironment of Pancreatic Cancer by Activating Pancrea… DOI 10.1002/advs.202509074↗
- Paper Retraction Note: BICC1 drives pancreatic cancer progression by inducing VEGF-independent angiogenesis. DOI 10.1038/s41392-025-02561-9↗
- Paper BICC1 drives pancreatic cancer stemness and chemoresistance by facilitating tryptophan metabolism. DOI 10.1126/sciadv.adj8650↗
- Chao Xu · 3 shared papers, 2024–2026 · strength 0.66
- Paper Tumor-Derived Interleukin 35 Promotes Fibrosis in the Tumor Microenvironment of Pancreatic Cancer by Activating Pancrea… DOI 10.1002/advs.202509074↗
- Paper Retraction Note: BICC1 drives pancreatic cancer progression by inducing VEGF-independent angiogenesis. DOI 10.1038/s41392-025-02561-9↗
- Paper BICC1 drives pancreatic cancer stemness and chemoresistance by facilitating tryptophan metabolism. DOI 10.1126/sciadv.adj8650↗
- Huizhi Sun · 3 shared papers, 2024–2026 · strength 0.66
- Paper Tumor-Derived Interleukin 35 Promotes Fibrosis in the Tumor Microenvironment of Pancreatic Cancer by Activating Pancrea… DOI 10.1002/advs.202509074↗
- Paper Retraction Note: BICC1 drives pancreatic cancer progression by inducing VEGF-independent angiogenesis. DOI 10.1038/s41392-025-02561-9↗
- Paper BICC1 drives pancreatic cancer stemness and chemoresistance by facilitating tryptophan metabolism. DOI 10.1126/sciadv.adj8650↗
- Jihui Hao · 3 shared papers, 2024–2026 · strength 0.66
- Paper Tumor-Derived Interleukin 35 Promotes Fibrosis in the Tumor Microenvironment of Pancreatic Cancer by Activating Pancrea… DOI 10.1002/advs.202509074↗
- Paper Retraction Note: BICC1 drives pancreatic cancer progression by inducing VEGF-independent angiogenesis. DOI 10.1038/s41392-025-02561-9↗
- Paper BICC1 drives pancreatic cancer stemness and chemoresistance by facilitating tryptophan metabolism. DOI 10.1126/sciadv.adj8650↗
- Peng Xie · 2 shared papers, 2024–2026 · strength 0.42
- Yukuan Feng · 2 shared papers, 2024–2026 · strength 0.42
- Xiuchao Wang · 2 shared papers, 2024–2026 · strength 0.41
- Tiansuo Zhao · 2 shared papers, 2024–2026 · strength 0.41
- Wei Lin · 1 shared paper, 2026 · strength 0.25
- Zengxun Li · 1 shared paper, 2026 · strength 0.24
- Shengyu Yang · 1 shared paper, 2024 · strength 0.17
- Danqi Fu · 1 shared paper, 2024 · strength 0.17
- Song Gao · 1 shared paper, 2024 · strength 0.17
- Jing Liu · 1 shared paper, 2024 · strength 0.17
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.