ResearcherWashington University in St. LouisActive in the last 2 years
Daoxiang Zhang
Matched on ORCID iD 0000-0002-4436-1188; OpenAlex author ID A5026024685. Sources: OpenAlex.
Their work
- Papers
- 10 (first author 1, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 23
2017: 1 paper · 2018: 2 papers · 2019: none · 2020: none · 2021: 1 paper · 2022: 1 paper · 2023: 1 paper · 2024: none · 2025: 1 paper · 2026: 3 papers
Papers 10
- Tetrastigma hemsleyanum ELN-derived miR-396b suppresses pancreatic cancer cell proliferation by targeting IL33. Last author DOI 10.1016/j.bbrc.2026.154163↗
- Targeting HAS2 to enhance anti-tumor immunity in pancreatic cancer via PD-L1 regulation. Last author DOI 10.1038/s42003-026-10009-1↗
- VDR Activation Suppresses Pancreatic Cancer Metastasis Through Inhibition of the ERK Signaling Pathway. Co-author DOI 10.3390/cancers18142296↗
- Cancer-Associated Fibroblast-Centric Risk Model Predicts Immunotherapy Resistance in Pancreatic Cancer and Reveals PLOD2 as a Key Stromal Therapeutic Target. Co-author DOI 10.31083/fbl46316↗
- Induction of cancer neoantigens facilitates development of clinically relevant models for the study of pancreatic cancer immunobiology. Co-author DOI 10.1007/s00262-023-03463-x↗
- IRAK4 Signaling Drives Resistance to Checkpoint Immunotherapy in Pancreatic Ductal Adenocarcinoma. First author DOI 10.1053/j.gastro.2022.02.035↗
- The MK2/Hsp27 axis is a major survival mechanism for pancreatic ductal adenocarcinoma under genotoxic stress. Co-author DOI 10.1126/scitranslmed.abb5445↗
- Tumor-Stroma IL1β-IRAK4 Feedforward Circuitry Drives Tumor Fibrosis, Chemoresistance, and Poor Prognosis in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-17-1366↗
- Concurrent HER or PI3K Inhibition Potentiates the Antitumor Effect of the ERK Inhibitor Ulixertinib in Preclinical Pancreatic Cancer Models. Co-author DOI 10.1158/1535-7163.mct-17-1142↗
- Constitutive IRAK4 Activation Underlies Poor Prognosis and Chemoresistance in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1078-0432.ccr-16-1121↗
Closest collaborators
The 23 strongest of 23 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Mengjie Lv · 3 shared papers, 2025–2026 · strength 0.85
- Paper Tetrastigma hemsleyanum ELN-derived miR-396b suppresses pancreatic cancer cell proliferation by targeting IL33. DOI 10.1016/j.bbrc.2026.154163↗
- Paper Cancer-Associated Fibroblast-Centric Risk Model Predicts Immunotherapy Resistance in Pancreatic Cancer and Reveals PLOD… DOI 10.31083/fbl46316↗
- Paper Targeting HAS2 to enhance anti-tumor immunity in pancreatic cancer via PD-L1 regulation. DOI 10.1038/s42003-026-10009-1↗
- Kian‐Huat Lim · 6 shared papers, 2017–2023 · strength 0.63
- Paper Induction of cancer neoantigens facilitates development of clinically relevant models for the study of pancreatic cance… DOI 10.1007/s00262-023-03463-x↗
- Paper IRAK4 Signaling Drives Resistance to Checkpoint Immunotherapy in Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2022.02.035↗
- Paper The MK2/Hsp27 axis is a major survival mechanism for pancreatic ductal adenocarcinoma under genotoxic stress. DOI 10.1126/scitranslmed.abb5445↗
- Pengwei Cao · 2 shared papers, 2025–2026 · strength 0.46
- David G DeNardo · 3 shared papers, 2017–2023 · strength 0.35
- Paper Induction of cancer neoantigens facilitates development of clinically relevant models for the study of pancreatic cance… DOI 10.1007/s00262-023-03463-x↗
- Paper IRAK4 Signaling Drives Resistance to Checkpoint Immunotherapy in Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2022.02.035↗
- Paper Constitutive IRAK4 Activation Underlies Poor Prognosis and Chemoresistance in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-16-1121↗
- Patrick M. Grierson · 3 shared papers, 2018–2022 · strength 0.34
- Paper IRAK4 Signaling Drives Resistance to Checkpoint Immunotherapy in Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2022.02.035↗
- Paper The MK2/Hsp27 axis is a major survival mechanism for pancreatic ductal adenocarcinoma under genotoxic stress. DOI 10.1126/scitranslmed.abb5445↗
- Paper Concurrent HER or PI3K Inhibition Potentiates the Antitumor Effect of the ERK Inhibitor Ulixertinib in Preclinical Panc… DOI 10.1158/1535-7163.mct-17-1142↗
- Paarth B. Dodhiawala · 3 shared papers, 2018–2022 · strength 0.34
- Paper IRAK4 Signaling Drives Resistance to Checkpoint Immunotherapy in Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2022.02.035↗
- Paper The MK2/Hsp27 axis is a major survival mechanism for pancreatic ductal adenocarcinoma under genotoxic stress. DOI 10.1126/scitranslmed.abb5445↗
- Paper Concurrent HER or PI3K Inhibition Potentiates the Antitumor Effect of the ERK Inhibitor Ulixertinib in Preclinical Panc… DOI 10.1158/1535-7163.mct-17-1142↗
- Yanping Wang · 1 shared paper, 2026 · strength 0.33
- Lin Li · 2 shared papers, 2021–2022 · strength 0.26
- Fubao Liu · 1 shared paper, 2025 · strength 0.24
- Chuifang Kong · 1 shared paper, 2026 · strength 0.23
- Hongmei Jiang · 3 shared papers, 2017–2018 · strength 0.23
- Paper Tumor-Stroma IL1β-IRAK4 Feedforward Circuitry Drives Tumor Fibrosis, Chemoresistance, and Poor Prognosis in Pancreatic … DOI 10.1158/0008-5472.can-17-1366↗
- Paper Concurrent HER or PI3K Inhibition Potentiates the Antitumor Effect of the ERK Inhibitor Ulixertinib in Preclinical Panc… DOI 10.1158/1535-7163.mct-17-1142↗
- Paper Constitutive IRAK4 Activation Underlies Poor Prognosis and Chemoresistance in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-16-1121↗
- Andrea Wang‐Gillam · 3 shared papers, 2017–2018 · strength 0.23
- Paper Tumor-Stroma IL1β-IRAK4 Feedforward Circuitry Drives Tumor Fibrosis, Chemoresistance, and Poor Prognosis in Pancreatic … DOI 10.1158/0008-5472.can-17-1366↗
- Paper Concurrent HER or PI3K Inhibition Potentiates the Antitumor Effect of the ERK Inhibitor Ulixertinib in Preclinical Panc… DOI 10.1158/1535-7163.mct-17-1142↗
- Paper Constitutive IRAK4 Activation Underlies Poor Prognosis and Chemoresistance in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-16-1121↗
- John M. Baer · 1 shared paper, 2023 · strength 0.14
- William G. Hawkins · 1 shared paper, 2023 · strength 0.14
- Feng Gao · 1 shared paper, 2023 · strength 0.14
- Usman Younis Panni · 1 shared paper, 2023 · strength 0.14
- C. Alston James · 1 shared paper, 2023 · strength 0.14
- William E. Gillanders · 1 shared paper, 2023 · strength 0.14
- Varintra E. Lander · 1 shared paper, 2022 · strength 0.14
- Liang‐I Kang · 1 shared paper, 2022 · strength 0.14
- Vikas Somani · 1 shared paper, 2022 · strength 0.14
- Qiong Li · 1 shared paper, 2018 · strength 0.08
- Albert Craig Lockhart · 1 shared paper, 2017 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.