ResearcherSun Yat-sen UniversityActive in the last 2 years
Bo Li
Matched on ORCID iD 0000-0002-4456-7303; OpenAlex author ID A5100374429. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 3, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 33
2011: 1 paper · 2012: none · 2013: none · 2014: none · 2015: none · 2016: none · 2017: 1 paper · 2018: none · 2019: 1 paper · 2020: none · 2021: none · 2022: 1 paper · 2023: 1 paper · 2024: 1 paper · 2025: 2 papers
Papers 8
- Identification and validation of a T cell receptor targeting KRAS G12V in HLA-A*11:01 pancreatic cancer patients. Co-author DOI 10.1172/jci.insight.181873↗
- Comprehensive Bioinformatics Analyses and Experimental Validation of the Cell Cycle Related Protein SAPCD2 as a New Biomarker and Potential Therapeutic Target … First author DOI 10.2147/jir.s501850↗
- The molecular mechanism underlying KRAS regulation on STK31 expression in pancreatic ductal adenocarcinoma. Co-author DOI 10.1111/cas.16286↗
- A novel aging-associated lncRNA prognostic signature for predicting immunotherapy and chemotherapy response in pancreatic cancer Co-author DOI 10.21203/rs.3.rs-3431528/v1↗
- Systemic therapy and perioperative management improve the prognosis of pancreatic ductal adenocarcinoma: A retrospective cohort study of 2000 consecutive cases. First author DOI 10.1016/j.ijsu.2022.106786↗
- NMIIA promotes tumor growth and metastasis by activating the Wnt/β-catenin signaling pathway and EMT in pancreatic cancer. Co-author DOI 10.1038/s41388-019-0806-6↗
- The epithelial to mesenchymal transition (EMT) and cancer stem cells: implication for treatment resistance in pancreatic cancer. First author DOI 10.1186/s12943-017-0624-9↗
- Tumour cryoablation combined with palliative bypass surgery in the treatment of unresectable pancreatic cancer: a retrospective study of 142 patients. Last author DOI 10.1136/pgmj.2010.098350↗
Closest collaborators
The 25 strongest of 33 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Suizhi Gao · 2 shared papers, 2022–2025 · strength 0.32
- Paper Identification and validation of a T cell receptor targeting KRAS G12V in HLA-A*11:01 pancreatic cancer patients. DOI 10.1172/jci.insight.181873↗
- Paper Systemic therapy and perioperative management improve the prognosis of pancreatic ductal adenocarcinoma: A retrospectiv… DOI 10.1016/j.ijsu.2022.106786↗
- Kailian Zheng · 2 shared papers, 2022–2025 · strength 0.32
- Paper Identification and validation of a T cell receptor targeting KRAS G12V in HLA-A*11:01 pancreatic cancer patients. DOI 10.1172/jci.insight.181873↗
- Paper Systemic therapy and perioperative management improve the prognosis of pancreatic ductal adenocarcinoma: A retrospectiv… DOI 10.1016/j.ijsu.2022.106786↗
- Shiwei Guo · 2 shared papers, 2022–2025 · strength 0.32
- Paper Identification and validation of a T cell receptor targeting KRAS G12V in HLA-A*11:01 pancreatic cancer patients. DOI 10.1172/jci.insight.181873↗
- Paper Systemic therapy and perioperative management improve the prognosis of pancreatic ductal adenocarcinoma: A retrospectiv… DOI 10.1016/j.ijsu.2022.106786↗
- Xiaoyi Yin · 2 shared papers, 2022–2025 · strength 0.32
- Paper Identification and validation of a T cell receptor targeting KRAS G12V in HLA-A*11:01 pancreatic cancer patients. DOI 10.1172/jci.insight.181873↗
- Paper Systemic therapy and perioperative management improve the prognosis of pancreatic ductal adenocarcinoma: A retrospectiv… DOI 10.1016/j.ijsu.2022.106786↗
- Xiaohan Shi · 2 shared papers, 2022–2025 · strength 0.32
- Paper Identification and validation of a T cell receptor targeting KRAS G12V in HLA-A*11:01 pancreatic cancer patients. DOI 10.1172/jci.insight.181873↗
- Paper Systemic therapy and perioperative management improve the prognosis of pancreatic ductal adenocarcinoma: A retrospectiv… DOI 10.1016/j.ijsu.2022.106786↗
- Yuting Liu · 1 shared paper, 2025 · strength 0.24
- Jian Qi · 1 shared paper, 2025 · strength 0.24
- Chi Han Li · 1 shared paper, 2024 · strength 0.23
- Chi Hin Wong · 1 shared paper, 2024 · strength 0.23
- Yuting Liu · 1 shared paper, 2024 · strength 0.23
- Stephen Lam Chan · 1 shared paper, 2024 · strength 0.23
- Yangchao Chen · 1 shared paper, 2024 · strength 0.23
- Li Yan · 1 shared paper, 2023 · strength 0.19
- Yuling Chen · 1 shared paper, 2023 · strength 0.19
- Yuanchun Ye · 1 shared paper, 2023 · strength 0.19
- Lingyu Zhu · 1 shared paper, 2025 · strength 0.19
- Xiongfei Xu · 1 shared paper, 2025 · strength 0.19
- Yunguang Li · 1 shared paper, 2025 · strength 0.19
- Dong Gao · 1 shared paper, 2025 · strength 0.19
- Gang Jin · 1 shared paper, 2025 · strength 0.19
- Eric Tran · 1 shared paper, 2025 · strength 0.19
- Yao Zong YUAN · 2 shared papers, 2017–2019 · strength 0.18
- Paper NMIIA promotes tumor growth and metastasis by activating the Wnt/β-catenin signaling pathway and EMT in pancreatic canc… DOI 10.1038/s41388-019-0806-6↗
- Paper The epithelial to mesenchymal transition (EMT) and cancer stem cells: implication for treatment resistance in pancreati… DOI 10.1186/s12943-017-0624-9↗
- Dong Li · 2 shared papers, 2017–2019 · strength 0.18
- Paper NMIIA promotes tumor growth and metastasis by activating the Wnt/β-catenin signaling pathway and EMT in pancreatic canc… DOI 10.1038/s41388-019-0806-6↗
- Paper The epithelial to mesenchymal transition (EMT) and cancer stem cells: implication for treatment resistance in pancreati… DOI 10.1186/s12943-017-0624-9↗
- Pingting Zhou · 2 shared papers, 2017–2019 · strength 0.18
- Paper NMIIA promotes tumor growth and metastasis by activating the Wnt/β-catenin signaling pathway and EMT in pancreatic canc… DOI 10.1038/s41388-019-0806-6↗
- Paper The epithelial to mesenchymal transition (EMT) and cancer stem cells: implication for treatment resistance in pancreati… DOI 10.1186/s12943-017-0624-9↗
- Xiangui Hu · 1 shared paper, 2022 · strength 0.13
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.