ResearcherShanghai Medical College of Fudan UniversityNo recent work listed
Yiyin Zhang
Matched on ORCID iD 0000-0002-4887-2401; OpenAlex author ID A5036813587. Sources: OpenAlex.
Their work
- Papers
- 20 (first author 3, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 39
2018: 4 papers · 2019: 4 papers · 2020: 6 papers · 2021: 1 paper · 2022: 1 paper · 2023: 4 papers
Papers 20
- Identification of exosomal miR-484 role in reprogramming mitochondrial metabolism in pancreatic cancer through Wnt/MAPK axis control. Co-author DOI 10.1016/j.phrs.2023.106980↗
- Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity and chemo-response. Co-author DOI 10.1016/j.xcrm.2023.101234↗
- CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in pancreatic ductal adenocarcinoma. Co-author DOI 10.1136/gutjnl-2022-329349↗
- Microbiota Regulates Pancreatic Cancer Carcinogenesis through Altered Immune Response. Co-author DOI 10.3390/microorganisms11051240↗
- Platelet-Vesicles-Encapsulated RSL-3 Enable Anti-Angiogenesis and Induce Ferroptosis to Inhibit Pancreatic Cancer Progress. Co-author DOI 10.3389/fendo.2022.865655↗
- Comment on "Postoperative Chemotherapy Benefits Patients Who Received Preoperative Therapy and Pancreatectomy for Pancreatic Adenocarcinoma". Co-author DOI 10.1097/sla.0000000000003989↗
- TGFB1-induced autophagy affects the pattern of pancreatic cancer progression in distinct ways depending on SMAD4 status. Co-author DOI 10.1080/15548627.2019.1628540↗
- PARP inhibitors in pancreatic cancer: molecular mechanisms and clinical applications. Co-author DOI 10.1186/s12943-020-01167-9↗
- Comments: The Prognostic Index Independently Predicts Survival in Patients With Pancreatic Ductal Adenocarcinoma Undergoing Resection. First author DOI 10.1245/s10434-020-08369-x↗
- The value of a metabolic reprogramming-related gene signature for pancreatic adenocarcinoma prognosis prediction. Co-author DOI 10.18632/aging.104134↗
- A miR-146a-5p/TRAF6/NF-kB p65 axis regulates pancreatic cancer chemoresistance: functional validation and clinical significance. Co-author DOI 10.7150/thno.40566↗
- The role of m6A-related genes in the prognosis and immune microenvironment of pancreatic adenocarcinoma. First author DOI 10.7717/peerj.9602↗
- Prognostic and diagnostic significance of galectins in pancreatic cancer: a systematic review and meta-analysis. First author DOI 10.1186/s12935-019-1025-5↗
- A PD-L2-based immune marker signature helps to predict survival in resected pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s40425-019-0703-0↗
- Role of Damage DNA-Binding Protein 1 in Pancreatic Cancer Progression and Chemoresistance. Co-author DOI 10.3390/cancers11121998↗
- Nab-paclitaxel plus gemcitabine as first-line treatment for advanced pancreatic cancer: a systematic review and meta-analysis. Co-author DOI 10.7150/jca.29898↗
- Anergic natural killer cells educated by tumor cells are associated with a poor prognosis in patients with advanced pancreatic ductal adenocarcinoma. Co-author DOI 10.1007/s00262-018-2235-8↗
- MUC16 C terminal-induced secretion of tumor-derived IL-6 contributes to tumor-associated Treg enrichment in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2018.01.017↗
- GPx1 is involved in the induction of protective autophagy in pancreatic cancer cells in response to glucose deprivation. Co-author DOI 10.1038/s41419-018-1244-z↗
- Novel agents for pancreatic ductal adenocarcinoma: emerging therapeutics and future directions. Co-author DOI 10.1186/s13045-017-0551-7↗
Closest collaborators
The 25 strongest of 39 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Xianjun Yu · 16 shared papers, 2018–2023 · strength 1.73
- Paper Comment on "Postoperative Chemotherapy Benefits Patients Who Received Preoperative Therapy and Pancreatectomy for Pancr… DOI 10.1097/sla.0000000000003989↗
- Paper CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in p… DOI 10.1136/gutjnl-2022-329349↗
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Si Shi · 12 shared papers, 2018–2023 · strength 1.40
- Paper Comment on "Postoperative Chemotherapy Benefits Patients Who Received Preoperative Therapy and Pancreatectomy for Pancr… DOI 10.1097/sla.0000000000003989↗
- Paper CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in p… DOI 10.1136/gutjnl-2022-329349↗
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Qingcai Meng · 11 shared papers, 2018–2023 · strength 1.21
- Paper CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in p… DOI 10.1136/gutjnl-2022-329349↗
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Paper TGFB1-induced autophagy affects the pattern of pancreatic cancer progression in distinct ways depending on SMAD4 status. DOI 10.1080/15548627.2019.1628540↗
- Jie Hua · 11 shared papers, 2018–2023 · strength 1.21
- Paper CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in p… DOI 10.1136/gutjnl-2022-329349↗
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Paper TGFB1-induced autophagy affects the pattern of pancreatic cancer progression in distinct ways depending on SMAD4 status. DOI 10.1080/15548627.2019.1628540↗
- Chen Liang · 10 shared papers, 2018–2023 · strength 1.10
- Paper CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in p… DOI 10.1136/gutjnl-2022-329349↗
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Paper TGFB1-induced autophagy affects the pattern of pancreatic cancer progression in distinct ways depending on SMAD4 status. DOI 10.1080/15548627.2019.1628540↗
- Jin Xu · 10 shared papers, 2018–2023 · strength 1.05
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Paper TGFB1-induced autophagy affects the pattern of pancreatic cancer progression in distinct ways depending on SMAD4 status. DOI 10.1080/15548627.2019.1628540↗
- Paper A miR-146a-5p/TRAF6/NF-kB p65 axis regulates pancreatic cancer chemoresistance: functional validation and clinical sign… DOI 10.7150/thno.40566↗
- Yuelong Liang · 3 shared papers, 2022–2023 · strength 0.52
- Paper Microbiota Regulates Pancreatic Cancer Carcinogenesis through Altered Immune Response. DOI 10.3390/microorganisms11051240↗
- Paper Identification of exosomal miR-484 role in reprogramming mitochondrial metabolism in pancreatic cancer through Wnt/MAPK… DOI 10.1016/j.phrs.2023.106980↗
- Paper Platelet-Vesicles-Encapsulated RSL-3 Enable Anti-Angiogenesis and Induce Ferroptosis to Inhibit Pancreatic Cancer Progr… DOI 10.3389/fendo.2022.865655↗
- Rong Tang · 3 shared papers, 2020–2023 · strength 0.41
- Paper CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in p… DOI 10.1136/gutjnl-2022-329349↗
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Paper The role of m6A-related genes in the prognosis and immune microenvironment of pancreatic adenocarcinoma. DOI 10.7717/peerj.9602↗
- Miaoyan Wei · 4 shared papers, 2019–2020 · strength 0.41
- Paper A miR-146a-5p/TRAF6/NF-kB p65 axis regulates pancreatic cancer chemoresistance: functional validation and clinical sign… DOI 10.7150/thno.40566↗
- Paper PARP inhibitors in pancreatic cancer: molecular mechanisms and clinical applications. DOI 10.1186/s12943-020-01167-9↗
- Paper The value of a metabolic reprogramming-related gene signature for pancreatic adenocarcinoma prognosis prediction. DOI 10.18632/aging.104134↗
- Jiang Liu · 4 shared papers, 2019–2020 · strength 0.40
- Paper PARP inhibitors in pancreatic cancer: molecular mechanisms and clinical applications. DOI 10.1186/s12943-020-01167-9↗
- Paper The value of a metabolic reprogramming-related gene signature for pancreatic adenocarcinoma prognosis prediction. DOI 10.18632/aging.104134↗
- Paper Nab-paclitaxel plus gemcitabine as first-line treatment for advanced pancreatic cancer: a systematic review and meta-an… DOI 10.7150/jca.29898↗
- Bo Zhang · 3 shared papers, 2020–2023 · strength 0.36
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Paper The role of m6A-related genes in the prognosis and immune microenvironment of pancreatic adenocarcinoma. DOI 10.7717/peerj.9602↗
- Paper The value of a metabolic reprogramming-related gene signature for pancreatic adenocarcinoma prognosis prediction. DOI 10.18632/aging.104134↗
- Tianyu Lin · 2 shared papers, 2022–2023 · strength 0.33
- Paper Identification of exosomal miR-484 role in reprogramming mitochondrial metabolism in pancreatic cancer through Wnt/MAPK… DOI 10.1016/j.phrs.2023.106980↗
- Paper Platelet-Vesicles-Encapsulated RSL-3 Enable Anti-Angiogenesis and Induce Ferroptosis to Inhibit Pancreatic Cancer Progr… DOI 10.3389/fendo.2022.865655↗
- Wei Wang · 2 shared papers, 2023 · strength 0.30
- Paper CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in p… DOI 10.1136/gutjnl-2022-329349↗
- Paper Targeting neoadjuvant chemotherapy-induced metabolic reprogramming in pancreatic cancer promotes anti-tumor immunity an… DOI 10.1016/j.xcrm.2023.101234↗
- Qiqing Sun · 2 shared papers, 2019–2020 · strength 0.30
- Paper Comments: The Prognostic Index Independently Predicts Survival in Patients With Pancreatic Ductal Adenocarcinoma Underg… DOI 10.1245/s10434-020-08369-x↗
- Paper Prognostic and diagnostic significance of galectins in pancreatic cancer: a systematic review and meta-analysis. DOI 10.1186/s12935-019-1025-5↗
- Zhen Tan · 2 shared papers, 2020–2023 · strength 0.26
- Paper CRIP1 fosters MDSC trafficking and resets tumour microenvironment via facilitating NF-κB/p65 nuclear translocation in p… DOI 10.1136/gutjnl-2022-329349↗
- Paper The value of a metabolic reprogramming-related gene signature for pancreatic adenocarcinoma prognosis prediction. DOI 10.18632/aging.104134↗
- Chao Yang · 3 shared papers, 2018 · strength 0.22
- Paper Anergic natural killer cells educated by tumor cells are associated with a poor prognosis in patients with advanced pan… DOI 10.1007/s00262-018-2235-8↗
- Paper MUC16 C terminal-induced secretion of tumor-derived IL-6 contributes to tumor-associated Treg enrichment in pancreatic … DOI 10.1016/j.canlet.2018.01.017↗
- Paper Novel agents for pancreatic ductal adenocarcinoma: emerging therapeutics and future directions. DOI 10.1186/s13045-017-0551-7↗
- Kun Fan · 3 shared papers, 2018 · strength 0.22
- Paper Anergic natural killer cells educated by tumor cells are associated with a poor prognosis in patients with advanced pan… DOI 10.1007/s00262-018-2235-8↗
- Paper MUC16 C terminal-induced secretion of tumor-derived IL-6 contributes to tumor-associated Treg enrichment in pancreatic … DOI 10.1016/j.canlet.2018.01.017↗
- Paper Novel agents for pancreatic ductal adenocarcinoma: emerging therapeutics and future directions. DOI 10.1186/s13045-017-0551-7↗
- Zhiyao Fan · 3 shared papers, 2018 · strength 0.22
- Paper Anergic natural killer cells educated by tumor cells are associated with a poor prognosis in patients with advanced pan… DOI 10.1007/s00262-018-2235-8↗
- Paper MUC16 C terminal-induced secretion of tumor-derived IL-6 contributes to tumor-associated Treg enrichment in pancreatic … DOI 10.1016/j.canlet.2018.01.017↗
- Paper Novel agents for pancreatic ductal adenocarcinoma: emerging therapeutics and future directions. DOI 10.1186/s13045-017-0551-7↗
- Chen Liu · 3 shared papers, 2018 · strength 0.22
- Paper Anergic natural killer cells educated by tumor cells are associated with a poor prognosis in patients with advanced pan… DOI 10.1007/s00262-018-2235-8↗
- Paper MUC16 C terminal-induced secretion of tumor-derived IL-6 contributes to tumor-associated Treg enrichment in pancreatic … DOI 10.1016/j.canlet.2018.01.017↗
- Paper Novel agents for pancreatic ductal adenocarcinoma: emerging therapeutics and future directions. DOI 10.1186/s13045-017-0551-7↗
- Guopei Luo · 3 shared papers, 2018 · strength 0.22
- Paper Anergic natural killer cells educated by tumor cells are associated with a poor prognosis in patients with advanced pan… DOI 10.1007/s00262-018-2235-8↗
- Paper MUC16 C terminal-induced secretion of tumor-derived IL-6 contributes to tumor-associated Treg enrichment in pancreatic … DOI 10.1016/j.canlet.2018.01.017↗
- Paper Novel agents for pancreatic ductal adenocarcinoma: emerging therapeutics and future directions. DOI 10.1186/s13045-017-0551-7↗
- Kaizhou Jin · 3 shared papers, 2018 · strength 0.22
- Paper Anergic natural killer cells educated by tumor cells are associated with a poor prognosis in patients with advanced pan… DOI 10.1007/s00262-018-2235-8↗
- Paper MUC16 C terminal-induced secretion of tumor-derived IL-6 contributes to tumor-associated Treg enrichment in pancreatic … DOI 10.1016/j.canlet.2018.01.017↗
- Paper Novel agents for pancreatic ductal adenocarcinoma: emerging therapeutics and future directions. DOI 10.1186/s13045-017-0551-7↗
- Yi-Han Chai · 1 shared paper, 2023 · strength 0.19
- Xiaowu Xu · 1 shared paper, 2020 · strength 0.19
- Guoping Ding · 1 shared paper, 2023 · strength 0.18
- Xiaofan Pu · 1 shared paper, 2023 · strength 0.18
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.