ResearcherUniversity of PisaActive in the last 2 years
Paola Cappello
Matched on ORCID iD 0000-0002-5321-7794; OpenAlex author ID A5050414562. Sources: OpenAlex.
Their work
- Papers
- 39 (first author 5, last author 4)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 116
2011: 1 paper · 2012: 1 paper · 2013: 4 papers · 2014: 1 paper · 2015: 1 paper · 2016: 4 papers · 2017: 2 papers · 2018: 4 papers · 2019: 1 paper · 2020: 4 papers · 2021: 5 papers · 2022: 1 paper · 2023: 2 papers · 2024: 4 papers · 2025: 1 paper · 2026: 3 papers
Papers 39
- AISP position statement: Standardising biological sample collection and handling for advanced diagnostics and multi-omic analyses in pancreatic cancer. Co-author DOI 10.1016/j.dld.2026.08.021↗
- The H63D Polymorphism in the HFE Gene Promotes Aggressive Disease Phenotypes in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1078-0432.ccr-25-4154↗
- Multi-omics and functional analysis of a bioengineered vascularized pancreatic cancer model reveal an immunosuppressive and therapy-resistant niche Co-author DOI 10.64898/2026.03.05.709702↗
- The absence of IL17A favours cytotoxic cell function and improves antigen-specific immunotherapies in pancreatic adenocarcinoma. Last author DOI 10.1002/ctm2.70442↗
- Exploiting pancreatic cancer metabolism: challenges and opportunities. Co-author DOI 10.1016/j.molmed.2024.03.008↗
- PI3Kγ inhibition combined with DNA vaccination unleashes a B-cell-dependent antitumor immunity that hampers pancreatic cancer. Co-author DOI 10.1186/s13046-024-03080-1↗
- In pancreatic cancer patients, chemotherapy reshapes the gene expression profile and antigen receptor repertoire of T lymphocytes and enhances their effector r… Co-author DOI 10.3389/fimmu.2024.1427424↗
- Multiplexed Imaging Mass Cytometry Analysis in Preclinical Models of Pancreatic Cancer. Co-author DOI 10.3390/ijms25031389↗
- IL-1β+ macrophages fuel pathogenic inflammation in pancreatic cancer. Co-author DOI 10.1038/s41586-023-06685-2↗
- Lysosomal lipid switch sensitises to nutrient deprivation and mTOR targeting in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2021-325117↗
- Docking Protein p130Cas Regulates Acinar to Ductal Metaplasia During Pancreatic Adenocarcinoma Development and Pancreatitis. Co-author DOI 10.1053/j.gastro.2021.12.242↗
- Impact of COVID-19 on Pancreatic Cancer Research and the Path Forward. Co-author DOI 10.1053/j.gastro.2021.06.080↗
- IL17A critically shapes the transcriptional program of fibroblasts in pancreatic cancer and switches on their protumorigenic functions. Last author DOI 10.1073/pnas.2020395118↗
- Intratumoral injection of TLR9 agonist promotes an immunopermissive microenvironment transition and causes cooperative antitumor activity in combination with a… Co-author DOI 10.1136/jitc-2021-002876↗
- Phosphoproteomics Identifies PI3K Inhibitor-selective Adaptive Responses in Pancreatic Cancer Cell Therapy and Resistance. Co-author DOI 10.1158/1535-7163.mct-20-0981↗
- The Glycolytic Pathway as a Target for Novel Onco-Immunology Therapies in Pancreatic Cancer. Co-author DOI 10.3390/molecules26061642↗
- In pancreatic cancer, chemotherapy increases antitumor responses to tumor-associated antigens and potentiates DNA vaccination. Co-author DOI 10.1136/jitc-2020-001071↗
- Metabolome of Pancreatic Juice Delineates Distinct Clinical Profiles of Pancreatic Cancer and Reveals a Link between Glucose Metabolism and PD-1+ Cells. Co-author DOI 10.1158/2326-6066.cir-19-0403↗
- Diabetes promotes invasive pancreatic cancer by increasing systemic and tumour carbonyl stress in Kras G12D/+ mice. Co-author DOI 10.1186/s13046-020-01665-0↗
- The dark side of immunotherapy: pancreatic cancer. Last author DOI 10.20517/cdr.2020.13↗
- Stromal protein βig-h3 reprogrammes tumour microenvironment in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2018-317570↗
- The advanced glycation end-product Nϵ -carboxymethyllysine promotes progression of pancreatic cancer: implications for diabetes-associated risk and its prevent… Co-author DOI 10.1002/path.5072↗
- Depletion of tumor-associated macrophages switches the epigenetic profile of pancreatic cancer infiltrating T cells and restores their anti-tumor phenotype. Last author DOI 10.1080/2162402x.2017.1393596↗
- Soluble stroma-related biomarkers of pancreatic cancer. Co-author DOI 10.15252/emmm.201708741↗
- Next Generation Immunotherapy for Pancreatic Cancer: DNA Vaccination is Seeking New Combo Partners. Co-author DOI 10.3390/cancers10020051↗
- Alpha-enolase (ENO1) controls alpha v/beta 3 integrin expression and regulates pancreatic cancer adhesion, invasion, and metastasis. Co-author DOI 10.1186/s13045-016-0385-8↗
- Endogenous glutamine decrease is associated with pancreatic cancer progression. Co-author DOI 10.18632/oncotarget.20545↗
- A bioluminescent mouse model of proliferation to highlight early stages of pancreatic cancer: A suitable tool for preclinical studies. Co-author DOI 10.1016/j.aanat.2015.11.010↗
- Spatial distribution of B cells predicts prognosis in human pancreatic adenocarcinoma. Co-author DOI 10.1080/2162402x.2015.1085147↗
- Macrophage PI3Kγ Drives Pancreatic Ductal Adenocarcinoma Progression. First author DOI 10.1158/2159-8290.cd-15-1346↗
- Peripheral ENO1-specific T cells mirror the intratumoral immune response and their presence is a potential prognostic factor for pancreatic adenocarcinoma. First author DOI 10.3892/ijo.2016.3524↗
- Targeting of surface alpha-enolase inhibits the invasiveness of pancreatic cancer cells. Co-author DOI 10.18632/oncotarget.3572↗
- Class II transactivator-induced MHC class II expression in pancreatic cancer cells leads to tumor rejection and a specific antitumor memory response. First author DOI 10.1097/mpa.0000000000000160↗
- Ex vivo analysis of pancreatic cancer-infiltrating T lymphocytes reveals that ENO-specific Tregs accumulate in tumor tissue and inhibit Th1/Th17 effector cell … Co-author DOI 10.1007/s00262-013-1429-3↗
- Vaccination with ENO1 DNA prolongs survival of genetically engineered mice with pancreatic cancer. Co-author DOI 10.1053/j.gastro.2013.01.020↗
- Autoantibodies to Ezrin are an early sign of pancreatic cancer in humans and in genetically engineered mouse models. First author DOI 10.1186/1756-8722-6-67↗
- A self antigen reopens the games in pancreatic cancer. Co-author DOI 10.4161/onci.24384↗
- Molecular and genetic bases of pancreatic cancer. Co-author DOI 10.2174/138945012800564077↗
- Circulating autoantibodies to phosphorylated α-enolase are a hallmark of pancreatic cancer. First author DOI 10.1021/pr100213b↗
Closest collaborators
The 25 strongest of 116 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Francesco Novelli · 32 shared papers, 2011–2026 · strength 2.89
- Paper The H63D Polymorphism in the HFE Gene Promotes Aggressive Disease Phenotypes in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-25-4154↗
- Paper The absence of IL17A favours cytotoxic cell function and improves antigen-specific immunotherapies in pancreatic adenoc… DOI 10.1002/ctm2.70442↗
- Paper PI3Kγ inhibition combined with DNA vaccination unleashes a B-cell-dependent antitumor immunity that hampers pancreatic … DOI 10.1186/s13046-024-03080-1↗
- Claudia Curcio · 8 shared papers, 2017–2025 · strength 1.08
- Paper The absence of IL17A favours cytotoxic cell function and improves antigen-specific immunotherapies in pancreatic adenoc… DOI 10.1002/ctm2.70442↗
- Paper PI3Kγ inhibition combined with DNA vaccination unleashes a B-cell-dependent antitumor immunity that hampers pancreatic … DOI 10.1186/s13046-024-03080-1↗
- Paper In pancreatic cancer patients, chemotherapy reshapes the gene expression profile and antigen receptor repertoire of T l… DOI 10.3389/fimmu.2024.1427424↗
- Silvia Brugiapaglia · 6 shared papers, 2020–2025 · strength 0.98
- Paper The absence of IL17A favours cytotoxic cell function and improves antigen-specific immunotherapies in pancreatic adenoc… DOI 10.1002/ctm2.70442↗
- Paper PI3Kγ inhibition combined with DNA vaccination unleashes a B-cell-dependent antitumor immunity that hampers pancreatic … DOI 10.1186/s13046-024-03080-1↗
- Paper In pancreatic cancer patients, chemotherapy reshapes the gene expression profile and antigen receptor repertoire of T l… DOI 10.3389/fimmu.2024.1427424↗
- Cecilia Roux · 6 shared papers, 2017–2025 · strength 0.78
- Paper The absence of IL17A favours cytotoxic cell function and improves antigen-specific immunotherapies in pancreatic adenoc… DOI 10.1002/ctm2.70442↗
- Paper PI3Kγ inhibition combined with DNA vaccination unleashes a B-cell-dependent antitumor immunity that hampers pancreatic … DOI 10.1186/s13046-024-03080-1↗
- Paper The dark side of immunotherapy: pancreatic cancer. DOI 10.20517/cdr.2020.13↗
- Elisa Giovannetti · 4 shared papers, 2021–2026 · strength 0.67
- Paper AISP position statement: Standardising biological sample collection and handling for advanced diagnostics and multi-omi… DOI 10.1016/j.dld.2026.08.021↗
- Paper Docking Protein p130Cas Regulates Acinar to Ductal Metaplasia During Pancreatic Adenocarcinoma Development and Pancreat… DOI 10.1053/j.gastro.2021.12.242↗
- Paper Lysosomal lipid switch sensitises to nutrient deprivation and mTOR targeting in pancreatic cancer. DOI 10.1136/gutjnl-2021-325117↗
- Miriam Martini · 4 shared papers, 2021–2024 · strength 0.65
- Paper Exploiting pancreatic cancer metabolism: challenges and opportunities. DOI 10.1016/j.molmed.2024.03.008↗
- Paper Docking Protein p130Cas Regulates Acinar to Ductal Metaplasia During Pancreatic Adenocarcinoma Development and Pancreat… DOI 10.1053/j.gastro.2021.12.242↗
- Paper Lysosomal lipid switch sensitises to nutrient deprivation and mTOR targeting in pancreatic cancer. DOI 10.1136/gutjnl-2021-325117↗
- Federica Marchesi · 4 shared papers, 2016–2026 · strength 0.63
- Paper AISP position statement: Standardising biological sample collection and handling for advanced diagnostics and multi-omi… DOI 10.1016/j.dld.2026.08.021↗
- Paper Multiplexed Imaging Mass Cytometry Analysis in Preclinical Models of Pancreatic Cancer. DOI 10.3390/ijms25031389↗
- Paper Metabolome of Pancreatic Juice Delineates Distinct Clinical Profiles of Pancreatic Cancer and Reveals a Link between Gl… DOI 10.1158/2326-6066.cir-19-0403↗
- Gianluca Mucciolo · 4 shared papers, 2020–2025 · strength 0.62
- Paper The absence of IL17A favours cytotoxic cell function and improves antigen-specific immunotherapies in pancreatic adenoc… DOI 10.1002/ctm2.70442↗
- Paper PI3Kγ inhibition combined with DNA vaccination unleashes a B-cell-dependent antitumor immunity that hampers pancreatic … DOI 10.1186/s13046-024-03080-1↗
- Paper The dark side of immunotherapy: pancreatic cancer. DOI 10.20517/cdr.2020.13↗
- Aldo Scarpa · 7 shared papers, 2011–2023 · strength 0.57
- Paper Lysosomal lipid switch sensitises to nutrient deprivation and mTOR targeting in pancreatic cancer. DOI 10.1136/gutjnl-2021-325117↗
- Paper Intratumoral injection of TLR9 agonist promotes an immunopermissive microenvironment transition and causes cooperative … DOI 10.1136/jitc-2021-002876↗
- Paper IL17A critically shapes the transcriptional program of fibroblasts in pancreatic cancer and switches on their protumori… DOI 10.1073/pnas.2020395118↗
- Vincenzo Corbo · 4 shared papers, 2021–2023 · strength 0.46
- Paper Lysosomal lipid switch sensitises to nutrient deprivation and mTOR targeting in pancreatic cancer. DOI 10.1136/gutjnl-2021-325117↗
- Paper IL-1β+ macrophages fuel pathogenic inflammation in pancreatic cancer. DOI 10.1038/s41586-023-06685-2↗
- Paper Intratumoral injection of TLR9 agonist promotes an immunopermissive microenvironment transition and causes cooperative … DOI 10.1136/jitc-2021-002876↗
- Giulia Adriani · 2 shared papers, 2025–2026 · strength 0.45
- Paper Multi-omics and functional analysis of a bioengineered vascularized pancreatic cancer model reveal an immunosuppressive… DOI 10.64898/2026.03.05.709702↗
- Paper The absence of IL17A favours cytotoxic cell function and improves antigen-specific immunotherapies in pancreatic adenoc… DOI 10.1002/ctm2.70442↗
- Maria Chiara De Santis · 2 shared papers, 2023–2024 · strength 0.41
- Giorgia Tiberi · 2 shared papers, 2024–2025 · strength 0.40
- Alessandro Zerbi · 3 shared papers, 2016–2026 · strength 0.35
- Paper The H63D Polymorphism in the HFE Gene Promotes Aggressive Disease Phenotypes in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-25-4154↗
- Paper Metabolome of Pancreatic Juice Delineates Distinct Clinical Profiles of Pancreatic Cancer and Reveals a Link between Gl… DOI 10.1158/2326-6066.cir-19-0403↗
- Paper Spatial distribution of B cells predicts prognosis in human pancreatic adenocarcinoma. DOI 10.1080/2162402x.2015.1085147↗
- Claudio Doglioni · 2 shared papers, 2023–2026 · strength 0.34
- Massimo Falconi · 2 shared papers, 2023–2026 · strength 0.34
- Enza Lonardo · 1 shared paper, 2026 · strength 0.30
- Silvia Carrara · 1 shared paper, 2026 · strength 0.30
- Claudio Luchini · 1 shared paper, 2026 · strength 0.30
- Manuel Gentiluomo · 1 shared paper, 2026 · strength 0.30
- Salvatore Paiella · 1 shared paper, 2026 · strength 0.30
- Paola Allavena · 4 shared papers, 2016–2020 · strength 0.29
- Paper Depletion of tumor-associated macrophages switches the epigenetic profile of pancreatic cancer infiltrating T cells and… DOI 10.1080/2162402x.2017.1393596↗
- Paper Metabolome of Pancreatic Juice Delineates Distinct Clinical Profiles of Pancreatic Cancer and Reveals a Link between Gl… DOI 10.1158/2326-6066.cir-19-0403↗
- Paper Soluble stroma-related biomarkers of pancreatic cancer. DOI 10.15252/emmm.201708741↗
- Marco Erreni · 2 shared papers, 2020–2024 · strength 0.28
- Bruno Bockorny · 1 shared paper, 2024 · strength 0.28
- Francesca Cordero · 2 shared papers, 2020–2024 · strength 0.28
- Paper In pancreatic cancer patients, chemotherapy reshapes the gene expression profile and antigen receptor repertoire of T l… DOI 10.3389/fimmu.2024.1427424↗
- Paper In pancreatic cancer, chemotherapy increases antitumor responses to tumor-associated antigens and potentiates DNA vacci… DOI 10.1136/jitc-2020-001071↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.