ResearcherCold Spring Harbor LaboratoryActive in the last 2 years
Claudia Tonelli
Matched on ORCID iD 0000-0002-5626-3403; OpenAlex author ID A5056456330. Sources: OpenAlex.
Their work
- Papers
- 7
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 29
2019: 1 paper · 2020: 1 paper · 2021: none · 2022: 2 papers · 2023: none · 2024: 2 papers · 2025: 1 paper
Papers 7
- FGFR2 Abrogation Intercepts Pancreatic Ductal Adenocarcinoma Development. Co-author DOI 10.1158/0008-5472.can-24-4576↗
- Engrailed-1 Promotes Pancreatic Cancer Metastasis. Co-author DOI 10.1002/advs.202308537↗
- A mucus production programme promotes classical pancreatic ductal adenocarcinoma. Co-author DOI 10.1136/gutjnl-2023-329839↗
- Loss of p53 tumor suppression function drives invasion and genomic instability in models of murine pancreatic cancer Co-author DOI 10.1101/2021.12.31.472823↗
- SPDEF promotes the classical subtype of pancreatic ductal adenocarcinoma Co-author DOI 10.1101/2022.03.18.484951↗
- SOAT1 promotes mevalonate pathway dependency in pancreatic cancer. Co-author DOI 10.1084/jem.20192389↗
- Glutamine Anabolism Plays a Critical Role in Pancreatic Cancer by Coupling Carbon and Nitrogen Metabolism. Co-author DOI 10.1016/j.celrep.2019.09.056↗
Closest collaborators
The 25 strongest of 29 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- David Arthur Tuveson · 7 shared papers, 2019–2025 · strength 1.09
- Paper FGFR2 Abrogation Intercepts Pancreatic Ductal Adenocarcinoma Development. DOI 10.1158/0008-5472.can-24-4576↗
- Paper Loss of p53 tumor suppression function drives invasion and genomic instability in models of murine pancreatic cancer DOI 10.1101/2021.12.31.472823↗
- Paper A mucus production programme promotes classical pancreatic ductal adenocarcinoma. DOI 10.1136/gutjnl-2023-329839↗
- Youngkyu Park · 6 shared papers, 2020–2025 · strength 1.02
- Paper FGFR2 Abrogation Intercepts Pancreatic Ductal Adenocarcinoma Development. DOI 10.1158/0008-5472.can-24-4576↗
- Paper Loss of p53 tumor suppression function drives invasion and genomic instability in models of murine pancreatic cancer DOI 10.1101/2021.12.31.472823↗
- Paper A mucus production programme promotes classical pancreatic ductal adenocarcinoma. DOI 10.1136/gutjnl-2023-329839↗
- Christopher R. Vakoc · 4 shared papers, 2020–2024 · strength 0.57
- Paper A mucus production programme promotes classical pancreatic ductal adenocarcinoma. DOI 10.1136/gutjnl-2023-329839↗
- Paper Engrailed-1 Promotes Pancreatic Cancer Metastasis. DOI 10.1002/advs.202308537↗
- Paper SPDEF promotes the classical subtype of pancreatic ductal adenocarcinoma DOI 10.1101/2022.03.18.484951↗
- Chang‐Il Hwang · 2 shared papers, 2020–2024 · strength 0.24
- Hao Yuan · 2 shared papers, 2020–2022 · strength 0.23
- Ralph H. Hruban MD · 1 shared paper, 2024 · strength 0.18
- Esak Lee · 1 shared paper, 2024 · strength 0.16
- Jean L. Grem · 1 shared paper, 2024 · strength 0.16
- Jae‐Seok Roe · 1 shared paper, 2024 · strength 0.16
- Jihao Xu · 1 shared paper, 2024 · strength 0.16
- Anna M. Kolarzyk · 1 shared paper, 2024 · strength 0.16
- Michael Anthony Hollingsworth · 1 shared paper, 2024 · strength 0.16
- Paul M. Grandgenett · 1 shared paper, 2024 · strength 0.16
- Hervé Tiriac · 1 shared paper, 2024 · strength 0.16
- Lindsey A. Baker · 1 shared paper, 2020 · strength 0.08
- Scott W. Lowe · 1 shared paper, 2020 · strength 0.08
- Tim D.D. Somerville · 1 shared paper, 2020 · strength 0.08
- Tobiloba E. Oni · 1 shared paper, 2020 · strength 0.08
- Giulia Biffi · 1 shared paper, 2020 · strength 0.08
- Nithya Sivaram · 1 shared paper, 2019 · strength 0.07
- Xufen Yu · 1 shared paper, 2019 · strength 0.07
- Jian Jun Jin · 1 shared paper, 2019 · strength 0.07
- Alex J. Bott · 1 shared paper, 2019 · strength 0.07
- Daniel J. Murphy · 1 shared paper, 2019 · strength 0.07
- Jennifer P. Morton · 1 shared paper, 2019 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.