ResearcherWenzhou Medical UniversityNo recent work listed
Bicheng Chen
Matched on ORCID iD 0000-0002-5663-7065; OpenAlex author ID A5111926646. Sources: OpenAlex.
Their work
- Papers
- 19 (first author 1, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 36
2014: 2 papers · 2015: none · 2016: 3 papers · 2017: 6 papers · 2018: 3 papers · 2019: 1 paper · 2020: 3 papers · 2021: 1 paper
Papers 19
- Periplocin inhibits the growth of pancreatic cancer by inducing apoptosis via AMPK-mTOR signaling. Co-author DOI 10.1002/cam4.3611↗
- KRT17 Functions as a Tumor Promoter and Regulates Proliferation, Migration and Invasion in Pancreatic Cancer via mTOR/S6k1 Pathway. Last author DOI 10.2147/cmar.s243129↗
- STRAP as a New Therapeutic Target for Poor Prognosis of Pancreatic Ductal Adenocarcinoma Patients Mainly Caused by TP53 Mutation. Co-author DOI 10.3389/fonc.2020.594224↗
- Upregulation of CENPM facilitates tumor metastasis via the mTOR/p70S6K signaling pathway in pancreatic cancer. Last author DOI 10.3892/or.2020.7673↗
- Baohuoside I Inhibits the Proliferation of Pancreatic Cancer Cells via mTOR/S6K1-Caspases/Bcl2/Bax Apoptotic Signaling. Co-author DOI 10.2147/cmar.s228926↗
- LncRNA-PVT1 promotes pancreatic cancer cells proliferation and migration through acting as a molecular sponge to regulate miR-448. Co-author DOI 10.1002/jcp.26072↗
- Brusatol Enhances the Chemotherapy Efficacy of Gemcitabine in Pancreatic Cancer via the Nrf2 Signalling Pathway. Co-author DOI 10.1155/2018/2360427↗
- Tyrphostin B42 attenuates trichostatin A-mediated resistance in pancreatic cancer cells by antagonizing IL-6/JAK2/STAT3 signaling. Co-author DOI 10.3892/or.2018.6241↗
- Brusatol inhibits growth and induces apoptosis in pancreatic cancer cells via JNK/p38 MAPK/NF-κb/Stat3/Bcl-2 signaling pathway. Co-author DOI 10.1016/j.bbrc.2017.04.133↗
- Dermokine contributes to epithelial-mesenchymal transition through increased activation of signal transducer and activator of transcription 3 in pancreatic can… Co-author DOI 10.1111/cas.13347↗
- The Lncrna-TUG1/EZH2 Axis Promotes Pancreatic Cancer Cell Proliferation, Migration and EMT Phenotype Formation Through Sponging Mir-382. Co-author DOI 10.1159/000479990↗
- HNRNPA2B1 regulates the epithelial-mesenchymal transition in pancreatic cancer cells through the ERK/snail signalling pathway. Co-author DOI 10.1186/s12935-016-0368-4↗
- Quercetin inhibits epithelial-mesenchymal transition, decreases invasiveness and metastasis, and reverses IL-6 induced epithelial-mesenchymal transition, expre… Co-author DOI 10.2147/ott.s136840↗
- Erratum: Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK signaling in pancreatic cancer cells [Corri… Co-author DOI 10.2147/ott.s140829↗
- Interleukin-32α inactivates JAK2/STAT3 signaling and reverses interleukin-6-induced epithelial-mesenchymal transition, invasion, and metastasis in pancreatic c… Co-author DOI 10.2147/ott.s103581↗
- Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK signaling in pancreatic cancer cells. Co-author DOI 10.2147/ott.s122653↗
- Sedum sarmentosum Bunge extract induces apoptosis and inhibits proliferation in pancreatic cancer cells via the hedgehog signaling pathway. First author DOI 10.3892/or.2016.4679↗
- Antitumor efficacy of α-solanine against pancreatic cancer in vitro and in vivo. Co-author DOI 10.1371/journal.pone.0087868↗
- Reversion of trichostatin A resistance via inhibition of the Wnt signaling pathway in human pancreatic cancer cells. Co-author DOI 10.3892/or.2014.3476↗
Closest collaborators
The 25 strongest of 36 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Mengtao Zhou · 10 shared papers, 2014–2018 · strength 0.65
- Paper Tyrphostin B42 attenuates trichostatin A-mediated resistance in pancreatic cancer cells by antagonizing IL-6/JAK2/STAT3… DOI 10.3892/or.2018.6241↗
- Paper The Lncrna-TUG1/EZH2 Axis Promotes Pancreatic Cancer Cell Proliferation, Migration and EMT Phenotype Formation Through … DOI 10.1159/000479990↗
- Paper Brusatol Enhances the Chemotherapy Efficacy of Gemcitabine in Pancreatic Cancer via the Nrf2 Signalling Pathway. DOI 10.1155/2018/2360427↗
- Chenlei Zheng · 4 shared papers, 2019–2020 · strength 0.45
- Paper Upregulation of CENPM facilitates tumor metastasis via the mTOR/p70S6K signaling pathway in pancreatic cancer. DOI 10.3892/or.2020.7673↗
- Paper KRT17 Functions as a Tumor Promoter and Regulates Proliferation, Migration and Invasion in Pancreatic Cancer via mTOR/S… DOI 10.2147/cmar.s243129↗
- Paper STRAP as a New Therapeutic Target for Poor Prognosis of Pancreatic Ductal Adenocarcinoma Patients Mainly Caused by TP53… DOI 10.3389/fonc.2020.594224↗
- Dinglai Yu · 6 shared papers, 2016–2018 · strength 0.41
- Paper Brusatol Enhances the Chemotherapy Efficacy of Gemcitabine in Pancreatic Cancer via the Nrf2 Signalling Pathway. DOI 10.1155/2018/2360427↗
- Paper Brusatol inhibits growth and induces apoptosis in pancreatic cancer cells via JNK/p38 MAPK/NF-κb/Stat3/Bcl-2 signaling … DOI 10.1016/j.bbrc.2017.04.133↗
- Paper Erratum: Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK sig… DOI 10.2147/ott.s140829↗
- Linxiao Sun · 3 shared papers, 2019–2021 · strength 0.41
- Paper Periplocin inhibits the growth of pancreatic cancer by inducing apoptosis via AMPK-mTOR signaling. DOI 10.1002/cam4.3611↗
- Paper KRT17 Functions as a Tumor Promoter and Regulates Proliferation, Migration and Invasion in Pancreatic Cancer via mTOR/S… DOI 10.2147/cmar.s243129↗
- Paper Baohuoside I Inhibits the Proliferation of Pancreatic Cancer Cells via mTOR/S6K1-Caspases/Bcl2/Bax Apoptotic Signaling. DOI 10.2147/cmar.s228926↗
- Chaohao Huang · 5 shared papers, 2016–2018 · strength 0.34
- Paper Brusatol Enhances the Chemotherapy Efficacy of Gemcitabine in Pancreatic Cancer via the Nrf2 Signalling Pathway. DOI 10.1155/2018/2360427↗
- Paper Brusatol inhibits growth and induces apoptosis in pancreatic cancer cells via JNK/p38 MAPK/NF-κb/Stat3/Bcl-2 signaling … DOI 10.1016/j.bbrc.2017.04.133↗
- Paper Erratum: Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK sig… DOI 10.2147/ott.s140829↗
- Hongwei Sun · 4 shared papers, 2014–2018 · strength 0.28
- Paper LncRNA-PVT1 promotes pancreatic cancer cells proliferation and migration through acting as a molecular sponge to regula… DOI 10.1002/jcp.26072↗
- Paper The Lncrna-TUG1/EZH2 Axis Promotes Pancreatic Cancer Cell Proliferation, Migration and EMT Phenotype Formation Through … DOI 10.1159/000479990↗
- Paper Dermokine contributes to epithelial-mesenchymal transition through increased activation of signal transducer and activa… DOI 10.1111/cas.13347↗
- Yukai Xiang · 4 shared papers, 2017–2018 · strength 0.28
- Paper Brusatol Enhances the Chemotherapy Efficacy of Gemcitabine in Pancreatic Cancer via the Nrf2 Signalling Pathway. DOI 10.1155/2018/2360427↗
- Paper Brusatol inhibits growth and induces apoptosis in pancreatic cancer cells via JNK/p38 MAPK/NF-κb/Stat3/Bcl-2 signaling … DOI 10.1016/j.bbrc.2017.04.133↗
- Paper Quercetin inhibits epithelial-mesenchymal transition, decreases invasiveness and metastasis, and reverses IL-6 induced … DOI 10.2147/ott.s136840↗
- Xince Huang · 4 shared papers, 2016–2017 · strength 0.28
- Paper Brusatol inhibits growth and induces apoptosis in pancreatic cancer cells via JNK/p38 MAPK/NF-κb/Stat3/Bcl-2 signaling … DOI 10.1016/j.bbrc.2017.04.133↗
- Paper Erratum: Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK sig… DOI 10.2147/ott.s140829↗
- Paper HNRNPA2B1 regulates the epithelial-mesenchymal transition in pancreatic cancer cells through the ERK/snail signalling p… DOI 10.1186/s12935-016-0368-4↗
- Zongjing Chen · 3 shared papers, 2014–2018 · strength 0.22
- Paper LncRNA-PVT1 promotes pancreatic cancer cells proliferation and migration through acting as a molecular sponge to regula… DOI 10.1002/jcp.26072↗
- Paper The Lncrna-TUG1/EZH2 Axis Promotes Pancreatic Cancer Cell Proliferation, Migration and EMT Phenotype Formation Through … DOI 10.1159/000479990↗
- Paper Reversion of trichostatin A resistance via inhibition of the Wnt signaling pathway in human pancreatic cancer cells. DOI 10.3892/or.2014.3476↗
- Cheng Wang · 2 shared papers, 2019–2020 · strength 0.21
- Paper KRT17 Functions as a Tumor Promoter and Regulates Proliferation, Migration and Invasion in Pancreatic Cancer via mTOR/S… DOI 10.2147/cmar.s243129↗
- Paper Baohuoside I Inhibits the Proliferation of Pancreatic Cancer Cells via mTOR/S6K1-Caspases/Bcl2/Bax Apoptotic Signaling. DOI 10.2147/cmar.s228926↗
- Shengjie Dai · 3 shared papers, 2016–2017 · strength 0.20
- Paper Erratum: Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK sig… DOI 10.2147/ott.s140829↗
- Paper HNRNPA2B1 regulates the epithelial-mesenchymal transition in pancreatic cancer cells through the ERK/snail signalling p… DOI 10.1186/s12935-016-0368-4↗
- Paper Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK signaling in… DOI 10.2147/ott.s122653↗
- Yongheng Bai · 3 shared papers, 2014–2018 · strength 0.20
- Paper Tyrphostin B42 attenuates trichostatin A-mediated resistance in pancreatic cancer cells by antagonizing IL-6/JAK2/STAT3… DOI 10.3892/or.2018.6241↗
- Paper Sedum sarmentosum Bunge extract induces apoptosis and inhibits proliferation in pancreatic cancer cells via the hedgeho… DOI 10.3892/or.2016.4679↗
- Paper Reversion of trichostatin A resistance via inhibition of the Wnt signaling pathway in human pancreatic cancer cells. DOI 10.3892/or.2014.3476↗
- Gangyin Xie · 1 shared paper, 2021 · strength 0.19
- Liang Zhao · 2 shared papers, 2017–2018 · strength 0.18
- Keqing Shi · 2 shared papers, 2018–2020 · strength 0.18
- Paper STRAP as a New Therapeutic Target for Poor Prognosis of Pancreatic Ductal Adenocarcinoma Patients Mainly Caused by TP53… DOI 10.3389/fonc.2020.594224↗
- Paper Brusatol Enhances the Chemotherapy Efficacy of Gemcitabine in Pancreatic Cancer via the Nrf2 Signalling Pathway. DOI 10.1155/2018/2360427↗
- Mengtao Zhou · 2 shared papers, 2016–2017 · strength 0.14
- Paper Erratum: Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK sig… DOI 10.2147/ott.s140829↗
- Paper Sedum sarmentosum Bunge extract induces apoptosis and inhibits proliferation in pancreatic cancer cells via the hedgeho… DOI 10.3892/or.2016.4679↗
- Jie Zhang · 2 shared papers, 2017 · strength 0.14
- Paper Brusatol inhibits growth and induces apoptosis in pancreatic cancer cells via JNK/p38 MAPK/NF-κb/Stat3/Bcl-2 signaling … DOI 10.1016/j.bbrc.2017.04.133↗
- Paper HNRNPA2B1 regulates the epithelial-mesenchymal transition in pancreatic cancer cells through the ERK/snail signalling p… DOI 10.1186/s12935-016-0368-4↗
- Tingting Ye · 2 shared papers, 2016–2017 · strength 0.14
- Paper Erratum: Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK sig… DOI 10.2147/ott.s140829↗
- Paper Isoorientin induces apoptosis, decreases invasiveness, and downregulates VEGF secretion by activating AMPK signaling in… DOI 10.2147/ott.s122653↗
- Ding Li · 1 shared paper, 2020 · strength 0.11
- Wenqian Huang · 1 shared paper, 2020 · strength 0.11
- Hongbao Yang · 1 shared paper, 2020 · strength 0.11
- Percy David Papa Akuetteh · 1 shared paper, 2020 · strength 0.11
- Shanshan Hu · 1 shared paper, 2020 · strength 0.11
- Qiyu Zhang · 1 shared paper, 2020 · strength 0.11
- Jingfeng Chen · 2 shared papers, 2014–2016 · strength 0.10
- Paper Interleukin-32α inactivates JAK2/STAT3 signaling and reverses interleukin-6-induced epithelial-mesenchymal transition, … DOI 10.2147/ott.s103581↗
- Paper Reversion of trichostatin A resistance via inhibition of the Wnt signaling pathway in human pancreatic cancer cells. DOI 10.3892/or.2014.3476↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.