Pancreatic Cancer Research

ResearcherSun Yat-sen UniversityNo recent work listed

Wenchao Gao

ORCID-verified (high confidence, 0.95), identity_conf@1As of 8 November 2026

Matched on ORCID iD 0000-0002-6106-6287; OpenAlex author ID A5023091772. Sources: OpenAlex.

Their work

Papers
10
Trials
0
Grants
0
Labs led
0
Collaborators
26
Papers, trials and grants by year

2014: 1 paper · 2015: 6 papers · 2016: 2 papers · 2017: none · 2018: none · 2019: none · 2020: none · 2021: none · 2022: 1 paper

Papers 10

  1. Corrigendum to "Long non-coding RNA LOC389641 promotes progression of pancreatic ductal adenocarcinoma and increases cell invasion by regulating E-cadherin in … Co-author DOI 10.1016/j.canlet.2022.215859↗
  2. The long non-coding RNA HOTAIR affects the radiosensitivity of pancreatic ductal adenocarcinoma by regulating the expression of Wnt inhibitory factor 1. Co-author DOI 10.1007/s13277-015-4234-0↗
  3. Long non-coding RNA LOC389641 promotes progression of pancreatic ductal adenocarcinoma and increases cell invasion by regulating E-cadherin in a TNFRSF10A-rela… Co-author DOI 10.1016/j.canlet.2015.12.010↗
  4. Analysis of global gene expression profiles suggests a role of acute inflammation in type 3C diabetes mellitus caused by pancreatic ductal adenocarcinoma. Co-author DOI 10.1007/s00125-014-3481-8↗
  5. The long non-coding RNA HOTTIP promotes progression and gemcitabine resistance by regulating HOXA13 in pancreatic cancer. Co-author DOI 10.1186/s12967-015-0442-z↗
  6. High expression of AFAP1-AS1 is associated with poor survival and short-term recurrence in pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s12967-015-0490-4↗
  7. Metabolic phenotypes in pancreatic cancer. Co-author DOI 10.1371/journal.pone.0115153↗
  8. Inhibition of glutamine metabolism counteracts pancreatic cancer stem cell features and sensitizes cells to radiotherapy. Co-author DOI 10.18632/oncotarget.5150↗
  9. Expression profile of long non-coding RNAs in pancreatic cancer and their clinical significance as biomarkers. Co-author DOI 10.18632/oncotarget.5533↗
  10. Macrophage migration inhibitory factor is overexpressed in pancreatic cancer tissues and impairs insulin secretion function of β-cell. Co-author DOI 10.1186/1479-5876-12-92↗

Closest collaborators

The 25 strongest of 26 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Quanbo Zhou · 9 shared papers, 2014–2022 · strength 0.46
  2. Yimin Liu · 8 shared papers, 2015–2022 · strength 0.43
  3. Shangyou Zheng · 7 shared papers, 2015–2022 · strength 0.39
  4. Xiaohui Zhao · 7 shared papers, 2015–2022 · strength 0.39
  5. Huilin Ye · 7 shared papers, 2014–2022 · strength 0.37
  6. Yingxue Wang · 5 shared papers, 2015–2022 · strength 0.31
  7. Zhihua Li · 5 shared papers, 2015–2022 · strength 0.31
  8. Zhiqiang Fu · 7 shared papers, 2014–2016 · strength 0.30
  9. Langping Tan · 5 shared papers, 2014–2022 · strength 0.29
  10. Rufu Chen · 6 shared papers, 2015–2016 · strength 0.27
  11. Wenzhu Li · 4 shared papers, 2015–2022 · strength 0.26
  12. Huimou Chen · 4 shared papers, 2015–2022 · strength 0.26
  13. Rufu Chen · 3 shared papers, 2014–2022 · strength 0.20
  14. Yu Zhou · 4 shared papers, 2015 · strength 0.17
  15. Yuming Luo · 2 shared papers, 2016–2022 · strength 0.17
  16. Min Yu · 3 shared papers, 2014–2015 · strength 0.12
  17. Yanhui Jiang · 2 shared papers, 2016 · strength 0.10
  18. Zhi-Hua Li · 2 shared papers, 2014–2015 · strength 0.07
  19. Yibiao Ye · 1 shared paper, 2015 · strength 0.05
  20. Guolin Li · 1 shared paper, 2016 · strength 0.05
  21. Lei Zhao · 1 shared paper, 2015 · strength 0.05
  22. Zhihua Li · 1 shared paper, 2015 · strength 0.04
  23. Doudou Li · 1 shared paper, 2015 · strength 0.04
  24. Jin-Long Zeng · 1 shared paper, 2015 · strength 0.04
  25. Yu Zhou · 1 shared paper, 2015 · strength 0.04

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.