ResearcherSheng Jing HospitalNo recent work listed
Haoyi Jin
Matched on ORCID iD 0000-0002-6685-1102; OpenAlex author ID A5102733693. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 1, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 14
2016: 1 paper · 2017: 1 paper · 2018: 2 papers · 2019: 3 papers · 2020: 2 papers
Papers 9
- Role of exosomal microRNA-125b-5p in conferring the metastatic phenotype among pancreatic cancer cells with different potential of metastasis. Last author DOI 10.1016/j.lfs.2020.117857↗
- Identification of dissociation factors in pancreatic Cancer using a mass spectrometry-based proteomic approach. Co-author DOI 10.1186/s12885-020-6522-3↗
- ITGA6 and RPSA synergistically promote pancreatic cancer invasion and metastasis via PI3K and MAPK signaling pathways. Last author DOI 10.1016/j.yexcr.2019.03.022↗
- Differential secretome of pancreatic cancer cells in serum-containing conditioned medium reveals CCT8 as a new biomarker of pancreatic cancer invasion and meta… Co-author DOI 10.1186/s12935-019-0980-1↗
- Identification of RE1-Silencing Transcription Factor as a Promoter of Metastasis in Pancreatic Cancer. Co-author DOI 10.3389/fonc.2019.00291↗
- Exosomal zinc transporter ZIP4 promotes cancer growth and is a novel diagnostic biomarker for pancreatic cancer. Co-author DOI 10.1111/cas.13737↗
- Integrated whole genome microarray analysis and immunohistochemical assay identifies COL11A1, GJB2 and CTRL as predictive biomarkers for pancreatic cancer. First author DOI 10.1186/s12935-018-0669-x↗
- The role of pancreatic cancer-derived exosomes in cancer progress and their potential application as biomarkers. Co-author DOI 10.1007/s12094-017-1625-2↗
- Quantitative secretomic analysis of pancreatic cancer cells in serum-containing conditioned medium. Co-author DOI 10.1038/srep37606↗
Closest collaborators
The 14 strongest of 14 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Xiaodong Tan · 8 shared papers, 2016–2020 · strength 0.81
- Paper Identification of dissociation factors in pancreatic Cancer using a mass spectrometry-based proteomic approach. DOI 10.1186/s12885-020-6522-3↗
- Paper Integrated whole genome microarray analysis and immunohistochemical assay identifies COL11A1, GJB2 and CTRL as predicti… DOI 10.1186/s12935-018-0669-x↗
- Paper Differential secretome of pancreatic cancer cells in serum-containing conditioned medium reveals CCT8 as a new biomarke… DOI 10.1186/s12935-019-0980-1↗
- Peng Liu · 7 shared papers, 2016–2020 · strength 0.69
- Paper Identification of dissociation factors in pancreatic Cancer using a mass spectrometry-based proteomic approach. DOI 10.1186/s12885-020-6522-3↗
- Paper Differential secretome of pancreatic cancer cells in serum-containing conditioned medium reveals CCT8 as a new biomarke… DOI 10.1186/s12935-019-0980-1↗
- Paper Role of exosomal microRNA-125b-5p in conferring the metastatic phenotype among pancreatic cancer cells with different p… DOI 10.1016/j.lfs.2020.117857↗
- Yunhao Wu · 6 shared papers, 2016–2020 · strength 0.59
- Paper Identification of dissociation factors in pancreatic Cancer using a mass spectrometry-based proteomic approach. DOI 10.1186/s12885-020-6522-3↗
- Paper Differential secretome of pancreatic cancer cells in serum-containing conditioned medium reveals CCT8 as a new biomarke… DOI 10.1186/s12935-019-0980-1↗
- Paper Role of exosomal microRNA-125b-5p in conferring the metastatic phenotype among pancreatic cancer cells with different p… DOI 10.1016/j.lfs.2020.117857↗
- Mengwei Wu · 4 shared papers, 2016–2020 · strength 0.35
- Paper Role of exosomal microRNA-125b-5p in conferring the metastatic phenotype among pancreatic cancer cells with different p… DOI 10.1016/j.lfs.2020.117857↗
- Paper ITGA6 and RPSA synergistically promote pancreatic cancer invasion and metastasis via PI3K and MAPK signaling pathways. DOI 10.1016/j.yexcr.2019.03.022↗
- Paper Exosomal zinc transporter ZIP4 promotes cancer growth and is a novel diagnostic biomarker for pancreatic cancer. DOI 10.1111/cas.13737↗
- Lingming Kong · 3 shared papers, 2019–2020 · strength 0.34
- Paper Identification of dissociation factors in pancreatic Cancer using a mass spectrometry-based proteomic approach. DOI 10.1186/s12885-020-6522-3↗
- Paper Differential secretome of pancreatic cancer cells in serum-containing conditioned medium reveals CCT8 as a new biomarke… DOI 10.1186/s12935-019-0980-1↗
- Paper Identification of RE1-Silencing Transcription Factor as a Promoter of Metastasis in Pancreatic Cancer. DOI 10.3389/fonc.2019.00291↗
- Bing Song · 2 shared papers, 2019–2020 · strength 0.24
- Paper Identification of dissociation factors in pancreatic Cancer using a mass spectrometry-based proteomic approach. DOI 10.1186/s12885-020-6522-3↗
- Paper Differential secretome of pancreatic cancer cells in serum-containing conditioned medium reveals CCT8 as a new biomarke… DOI 10.1186/s12935-019-0980-1↗
- Defeng Sun · 2 shared papers, 2018–2019 · strength 0.22
- Yifan Yang · 2 shared papers, 2019–2020 · strength 0.20
- Paper Role of exosomal microRNA-125b-5p in conferring the metastatic phenotype among pancreatic cancer cells with different p… DOI 10.1016/j.lfs.2020.117857↗
- Paper ITGA6 and RPSA synergistically promote pancreatic cancer invasion and metastasis via PI3K and MAPK signaling pathways. DOI 10.1016/j.yexcr.2019.03.022↗
- Xinlu Liu · 2 shared papers, 2019–2020 · strength 0.20
- Paper Role of exosomal microRNA-125b-5p in conferring the metastatic phenotype among pancreatic cancer cells with different p… DOI 10.1016/j.lfs.2020.117857↗
- Paper ITGA6 and RPSA synergistically promote pancreatic cancer invasion and metastasis via PI3K and MAPK signaling pathways. DOI 10.1016/j.yexcr.2019.03.022↗
- Keke Liang · 1 shared paper, 2020 · strength 0.13
- Jun Zhang · 1 shared paper, 2018 · strength 0.12
- Xiyang Tan · 1 shared paper, 2017 · strength 0.12
- Yang Gao · 1 shared paper, 2019 · strength 0.10
- Yukui Zhang · 1 shared paper, 2016 · strength 0.06
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.