ResearcherSecond Affiliated Hospital of Xi'an Jiaotong UniversityNo recent work listed
Gang Cao
Matched on ORCID iD 0000-0002-6801-0294; OpenAlex author ID A5101880977. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 1, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 22
2012: 1 paper · 2013: 1 paper · 2014: none · 2015: none · 2016: none · 2017: none · 2018: 2 papers · 2019: 1 paper · 2020: 3 papers · 2021: none · 2022: 1 paper
Papers 9
- Conversion of epithelial-to-mesenchymal transition to mesenchymal-to-epithelial transition is mediated by oxygen concentration in pancreatic cancer cells. Co-author DOI 10.3892/ol.2022.13227↗
- Magnolol Suppresses Pancreatic Cancer Development In Vivo and In Vitro via Negatively Regulating TGF-β/Smad Signaling. Last author DOI 10.3389/fonc.2020.597672↗
- MicroRNA-4516 suppresses pancreatic cancer development via negatively regulating orthodenticle homeobox 1. Co-author DOI 10.7150/ijbs.45933↗
- The JAM-B/c-src/MMP9 pathway is associated with progression and regulates the invasion of pancreatic cancer. Co-author DOI 10.7150/jca.40953↗
- Upregulation of LASP2 inhibits pancreatic cancer cell migration and invasion through suppressing TGF-β-induced EMT. Co-author DOI 10.1002/jcb.28638↗
- High expression of microRNA-4295 contributes to cell proliferation and invasion of pancreatic ductal adenocarcinoma by the down-regulation of Glypican-5. Co-author DOI 10.1016/j.bbrc.2018.02.023↗
- Conversion of epithelial-to-mesenchymal transition to mesenchymal-to-epithelial transition is mediated by oxygen concentration in pancreatic cancer cells. Co-author DOI 10.3892/ol.2018.8219↗
- Early manifestations of pancreatic cancer: the effect of cancer-nerve interaction. Co-author DOI 10.1016/j.mehy.2013.05.010↗
- The bidirectional interation between pancreatic cancer and diabetes. First author DOI 10.1186/1477-7819-10-171↗
Closest collaborators
The 22 strongest of 22 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Wenbin Yang · 4 shared papers, 2018–2022 · strength 0.45
- Paper Conversion of epithelial-to-mesenchymal transition to mesenchymal-to-epithelial transition is mediated by oxygen concen… DOI 10.3892/ol.2022.13227↗
- Paper The JAM-B/c-src/MMP9 pathway is associated with progression and regulates the invasion of pancreatic cancer. DOI 10.7150/jca.40953↗
- Paper High expression of microRNA-4295 contributes to cell proliferation and invasion of pancreatic ductal adenocarcinoma by … DOI 10.1016/j.bbrc.2018.02.023↗
- Tao Shan · 4 shared papers, 2018–2022 · strength 0.43
- Paper Conversion of epithelial-to-mesenchymal transition to mesenchymal-to-epithelial transition is mediated by oxygen concen… DOI 10.3892/ol.2022.13227↗
- Paper Magnolol Suppresses Pancreatic Cancer Development In Vivo and In Vitro via Negatively Regulating TGF-β/Smad Signaling. DOI 10.3389/fonc.2020.597672↗
- Paper MicroRNA-4516 suppresses pancreatic cancer development via negatively regulating orthodenticle homeobox 1. DOI 10.7150/ijbs.45933↗
- Junhui Li · 3 shared papers, 2018–2020 · strength 0.34
- Paper The JAM-B/c-src/MMP9 pathway is associated with progression and regulates the invasion of pancreatic cancer. DOI 10.7150/jca.40953↗
- Paper Magnolol Suppresses Pancreatic Cancer Development In Vivo and In Vitro via Negatively Regulating TGF-β/Smad Signaling. DOI 10.3389/fonc.2020.597672↗
- Paper High expression of microRNA-4295 contributes to cell proliferation and invasion of pancreatic ductal adenocarcinoma by … DOI 10.1016/j.bbrc.2018.02.023↗
- Shuo Chen · 3 shared papers, 2020 · strength 0.34
- Paper The JAM-B/c-src/MMP9 pathway is associated with progression and regulates the invasion of pancreatic cancer. DOI 10.7150/jca.40953↗
- Paper Magnolol Suppresses Pancreatic Cancer Development In Vivo and In Vitro via Negatively Regulating TGF-β/Smad Signaling. DOI 10.3389/fonc.2020.597672↗
- Paper MicroRNA-4516 suppresses pancreatic cancer development via negatively regulating orthodenticle homeobox 1. DOI 10.7150/ijbs.45933↗
- Qinggong Yuan · 2 shared papers, 2018–2019 · strength 0.23
- Paper Upregulation of LASP2 inhibits pancreatic cancer cell migration and invasion through suppressing TGF-β-induced EMT. DOI 10.1002/jcb.28638↗
- Paper High expression of microRNA-4295 contributes to cell proliferation and invasion of pancreatic ductal adenocarcinoma by … DOI 10.1016/j.bbrc.2018.02.023↗
- Shuo Chen · 2 shared papers, 2018–2022 · strength 0.21
- Paper Conversion of epithelial-to-mesenchymal transition to mesenchymal-to-epithelial transition is mediated by oxygen concen… DOI 10.3892/ol.2022.13227↗
- Paper Conversion of epithelial-to-mesenchymal transition to mesenchymal-to-epithelial transition is mediated by oxygen concen… DOI 10.3892/ol.2018.8219↗
- Ya’an Kang · 2 shared papers, 2018–2022 · strength 0.21
- Paper Conversion of epithelial-to-mesenchymal transition to mesenchymal-to-epithelial transition is mediated by oxygen concen… DOI 10.3892/ol.2022.13227↗
- Paper Conversion of epithelial-to-mesenchymal transition to mesenchymal-to-epithelial transition is mediated by oxygen concen… DOI 10.3892/ol.2018.8219↗
- Jiaqi Shen · 2 shared papers, 2020 · strength 0.21
- Li Zhang · 2 shared papers, 2013–2020 · strength 0.17
- Wunai Zhang · 1 shared paper, 2020 · strength 0.13
- Wen‐Bin Yang · 1 shared paper, 2019 · strength 0.12
- Yan Zhang · 1 shared paper, 2019 · strength 0.12
- Yang Liu · 1 shared paper, 2020 · strength 0.11
- Qingyong Ma · 2 shared papers, 2012–2013 · strength 0.08
- Wei Li · 1 shared paper, 2018 · strength 0.07
- Xi Chen · 1 shared paper, 2018 · strength 0.07
- Li Wang · 1 shared paper, 2018 · strength 0.07
- Junhui Li · 1 shared paper, 2013 · strength 0.04
- Junhui Li · 1 shared paper, 2012 · strength 0.04
- Wei Li · 1 shared paper, 2012 · strength 0.04
- Han Liu · 1 shared paper, 2012 · strength 0.04
- Liang Han · 1 shared paper, 2012 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.