ResearcherUniversity of California, IrvineActive in the last 2 years
Rima Singh
Matched on ORCID iD 0000-0002-6822-7987; OpenAlex author ID A5047870893. Sources: OpenAlex.
Their work
- Papers
- 5
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 48
2022: 2 papers · 2023: none · 2024: none · 2025: none · 2026: 3 papers
Papers 5
- Comparative Analysis of Primary and Liver Fibroblasts Reveals MET as a Potent Target in Pancreatic Cancer Metastasis. Co-author DOI 10.1016/j.jcmgh.2026.101872↗
- Mevalonate Biosynthesis is a Metabolic Vulnerability of Gemcitabine-resistant Pancreatic Cancer. Co-author DOI 10.1053/j.gastro.2026.09.027↗
- ATM Deficiency Induces TGFβ-Mediated Stromal Programming in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-26-0138↗
- Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic adenocarcinoma cells. Co-author DOI 10.1038/s43018-022-00463-1↗
- Multiomic characterization of pancreatic cancer-associated macrophage polarization reveals deregulated metabolic programs driven by the GM-CSF-PI3K pathway. Co-author DOI 10.7554/elife.73796↗
Closest collaborators
The 25 strongest of 48 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Christopher J. Halbrook · 5 shared papers, 2022–2026 · strength 0.84
- Paper Comparative Analysis of Primary and Liver Fibroblasts Reveals MET as a Potent Target in Pancreatic Cancer Metastasis. DOI 10.1016/j.jcmgh.2026.101872↗
- Paper Mevalonate Biosynthesis is a Metabolic Vulnerability of Gemcitabine-resistant Pancreatic Cancer. DOI 10.1053/j.gastro.2026.09.027↗
- Paper ATM Deficiency Induces TGFβ-Mediated Stromal Programming in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-0138↗
- Austin D. Silva · 3 shared papers, 2026 · strength 0.61
- Paper Comparative Analysis of Primary and Liver Fibroblasts Reveals MET as a Potent Target in Pancreatic Cancer Metastasis. DOI 10.1016/j.jcmgh.2026.101872↗
- Paper Mevalonate Biosynthesis is a Metabolic Vulnerability of Gemcitabine-resistant Pancreatic Cancer. DOI 10.1053/j.gastro.2026.09.027↗
- Paper ATM Deficiency Induces TGFβ-Mediated Stromal Programming in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-0138↗
- Alica K. Michels · 3 shared papers, 2026 · strength 0.61
- Paper Comparative Analysis of Primary and Liver Fibroblasts Reveals MET as a Potent Target in Pancreatic Cancer Metastasis. DOI 10.1016/j.jcmgh.2026.101872↗
- Paper Mevalonate Biosynthesis is a Metabolic Vulnerability of Gemcitabine-resistant Pancreatic Cancer. DOI 10.1053/j.gastro.2026.09.027↗
- Paper ATM Deficiency Induces TGFβ-Mediated Stromal Programming in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-0138↗
- Nina G. Steele · 3 shared papers, 2022–2026 · strength 0.46
- Paper Comparative Analysis of Primary and Liver Fibroblasts Reveals MET as a Potent Target in Pancreatic Cancer Metastasis. DOI 10.1016/j.jcmgh.2026.101872↗
- Paper Multiomic characterization of pancreatic cancer-associated macrophage polarization reveals deregulated metabolic progra… DOI 10.7554/elife.73796↗
- Paper Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic … DOI 10.1038/s43018-022-00463-1↗
- Alexander Kleger · 2 shared papers, 2026 · strength 0.38
- Thomas Theodor Werner Seufferlein · 2 shared papers, 2026 · strength 0.38
- Jennifer P. Morton · 2 shared papers, 2022–2026 · strength 0.33
- Paper Comparative Analysis of Primary and Liver Fibroblasts Reveals MET as a Potent Target in Pancreatic Cancer Metastasis. DOI 10.1016/j.jcmgh.2026.101872↗
- Paper Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic … DOI 10.1038/s43018-022-00463-1↗
- Seth Boyer · 2 shared papers, 2022 · strength 0.24
- Paper Multiomic characterization of pancreatic cancer-associated macrophage polarization reveals deregulated metabolic progra… DOI 10.7554/elife.73796↗
- Paper Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic … DOI 10.1038/s43018-022-00463-1↗
- Marina Pasca di Magliano · 2 shared papers, 2022 · strength 0.24
- Paper Multiomic characterization of pancreatic cancer-associated macrophage polarization reveals deregulated metabolic progra… DOI 10.7554/elife.73796↗
- Paper Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic … DOI 10.1038/s43018-022-00463-1↗
- Li Zhang · 2 shared papers, 2022 · strength 0.24
- Paper Multiomic characterization of pancreatic cancer-associated macrophage polarization reveals deregulated metabolic progra… DOI 10.7554/elife.73796↗
- Paper Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic … DOI 10.1038/s43018-022-00463-1↗
- Costas Andreas Lyssiotis · 2 shared papers, 2022 · strength 0.24
- Paper Multiomic characterization of pancreatic cancer-associated macrophage polarization reveals deregulated metabolic progra… DOI 10.7554/elife.73796↗
- Paper Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic … DOI 10.1038/s43018-022-00463-1↗
- Oliver Gene McDonald · 1 shared paper, 2026 · strength 0.23
- Nikki K. Lytle · 1 shared paper, 2026 · strength 0.23
- David K. Imagawa · 1 shared paper, 2026 · strength 0.23
- Ian M. Loveless · 1 shared paper, 2026 · strength 0.23
- Zeljka Jutric · 1 shared paper, 2026 · strength 0.23
- Mohammed Aldakkak · 1 shared paper, 2026 · strength 0.23
- Alexander Muir · 1 shared paper, 2026 · strength 0.20
- Jennifer Brooke Valerin · 1 shared paper, 2026 · strength 0.20
- Chantal Allgöwer · 1 shared paper, 2026 · strength 0.20
- Tim Eiseler · 1 shared paper, 2026 · strength 0.20
- Kevin Christian Montecillo Gulay · 1 shared paper, 2026 · strength 0.20
- Aimee L Edinger · 1 shared paper, 2026 · strength 0.20
- David Kuang-Fu Chang · 1 shared paper, 2026 · strength 0.20
- Hervé Tiriac · 1 shared paper, 2026 · strength 0.20
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.