ResearcherCharles UniversityActive in the last 2 years

Saba Selvi

ORCID-verified (high confidence, 0.95), identity_conf@1As of 7 November 2026

Matched on ORCID iD 0000-0002-6965-1384; OpenAlex author ID A5033717753. Sources: OpenAlex.

Their work

Papers: 1
Trials: 0
Grants: 0
Labs led: 0
Collaborators: 8

Papers, trials and grants by year

2025: 1 paper

Papers 1

Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. Co-author DOI 10.1016/j.semcancer.2025.05.005 ↗

Closest collaborators

The 8 strongest of 8 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Kari Hemminki · 1 shared paper, 2025 · strength 0.22
- Paper Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. DOI 10.1016/j.semcancer.2025.05.005 ↗
Božena Smolková · 1 shared paper, 2025 · strength 0.22
- Paper Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. DOI 10.1016/j.semcancer.2025.05.005 ↗
Ľudmila Vodičková · 1 shared paper, 2025 · strength 0.22
- Paper Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. DOI 10.1016/j.semcancer.2025.05.005 ↗
Pavel Vodička · 1 shared paper, 2025 · strength 0.22
- Paper Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. DOI 10.1016/j.semcancer.2025.05.005 ↗
Erika Halášová · 1 shared paper, 2025 · strength 0.22
- Paper Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. DOI 10.1016/j.semcancer.2025.05.005 ↗
Daniele Campa · 1 shared paper, 2025 · strength 0.22
- Paper Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. DOI 10.1016/j.semcancer.2025.05.005 ↗
Manuel Gentiluomo · 1 shared paper, 2025 · strength 0.22
- Paper Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. DOI 10.1016/j.semcancer.2025.05.005 ↗
Cosmeri Rizzato · 1 shared paper, 2025 · strength 0.22
- Paper Genomic instability, DNA damage response and telomere homeostasis in pancreatic cancer. DOI 10.1016/j.semcancer.2025.05.005 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.