ResearcherWuhan UniversityActive in the last 2 years
Hanjun Li
Matched on ORCID iD 0000-0002-7017-8782; OpenAlex author ID A5071554167. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 1, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 10
2021: 2 papers · 2022: 1 paper · 2023: 1 paper · 2024: none · 2025: 1 paper
Papers 5
- UPP1 and AHSA1 as emerging biomarkers and targets in pancreatic cancer: A proteomic approach. Last author DOI 10.17305/bb.2025.11958↗
- CASC9 potentiates gemcitabine resistance in pancreatic cancer by reciprocally activating NRF2 and the NF-κB signaling pathway. Co-author DOI 10.1007/s10565-022-09746-w↗
- microRNA-21-5p from M2 macrophage-derived extracellular vesicles promotes the differentiation and activity of pancreatic cancer stem cells by mediating KLF3. First author DOI 10.1007/s10565-021-09597-x↗
- Circular RNA CDR1as promotes tumor progression by regulating miR-432-5p/E2F3 axis in pancreatic cancer. Co-author DOI 10.1186/s12935-021-01812-3↗
- Hypoxia-induced lncRNA CASC9 enhances glycolysis and the epithelial-mesenchymal transition of pancreatic cancer by a positive feedback loop with AKT/HIF-1α sig… Co-author PMID 33520364↗
Closest collaborators
The 10 strongest of 10 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jing Tao · 4 shared papers, 2021–2023 · strength 0.65
- Paper CASC9 potentiates gemcitabine resistance in pancreatic cancer by reciprocally activating NRF2 and the NF-κB signaling p… DOI 10.1007/s10565-022-09746-w↗
- Paper microRNA-21-5p from M2 macrophage-derived extracellular vesicles promotes the differentiation and activity of pancreati… DOI 10.1007/s10565-021-09597-x↗
- Paper Circular RNA CDR1as promotes tumor progression by regulating miR-432-5p/E2F3 axis in pancreatic cancer. DOI 10.1186/s12935-021-01812-3↗
- Kongfan Zhu · 1 shared paper, 2025 · strength 0.33
- Zhi‐Gang Tang · 2 shared papers, 2021–2023 · strength 0.32
- Paper CASC9 potentiates gemcitabine resistance in pancreatic cancer by reciprocally activating NRF2 and the NF-κB signaling p… DOI 10.1007/s10565-022-09746-w↗
- Paper Hypoxia-induced lncRNA CASC9 enhances glycolysis and the epithelial-mesenchymal transition of pancreatic cancer by a po… PMID 33520364↗
- Zhengle Zhang · 2 shared papers, 2021–2023 · strength 0.32
- Paper CASC9 potentiates gemcitabine resistance in pancreatic cancer by reciprocally activating NRF2 and the NF-κB signaling p… DOI 10.1007/s10565-022-09746-w↗
- Paper Hypoxia-induced lncRNA CASC9 enhances glycolysis and the epithelial-mesenchymal transition of pancreatic cancer by a po… PMID 33520364↗
- Jian Hua Chang · 2 shared papers, 2021–2022 · strength 0.32
- Paper microRNA-21-5p from M2 macrophage-derived extracellular vesicles promotes the differentiation and activity of pancreati… DOI 10.1007/s10565-021-09597-x↗
- Paper Circular RNA CDR1as promotes tumor progression by regulating miR-432-5p/E2F3 axis in pancreatic cancer. DOI 10.1186/s12935-021-01812-3↗
- Xingcheng Xiong · 2 shared papers, 2021–2022 · strength 0.32
- Paper microRNA-21-5p from M2 macrophage-derived extracellular vesicles promotes the differentiation and activity of pancreati… DOI 10.1007/s10565-021-09597-x↗
- Paper Circular RNA CDR1as promotes tumor progression by regulating miR-432-5p/E2F3 axis in pancreatic cancer. DOI 10.1186/s12935-021-01812-3↗
- Pei Mei · 2 shared papers, 2021–2022 · strength 0.29
- Paper microRNA-21-5p from M2 macrophage-derived extracellular vesicles promotes the differentiation and activity of pancreati… DOI 10.1007/s10565-021-09597-x↗
- Paper Hypoxia-induced lncRNA CASC9 enhances glycolysis and the epithelial-mesenchymal transition of pancreatic cancer by a po… PMID 33520364↗
- Longjiang Chen · 1 shared paper, 2023 · strength 0.20
- Weimin Hu · 1 shared paper, 2022 · strength 0.17
- Hehe Li · 1 shared paper, 2021 · strength 0.12
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.