ResearcherShanghai Medical College of Fudan UniversityActive in the last 2 years
Weiding Wu
Matched on ORCID iD 0000-0002-7188-1066; OpenAlex author ID A5058383798. Sources: OpenAlex.
Their work
- Papers
- 17 (first author 0, last author 10)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 41
2019: 1 paper · 2020: none · 2021: 1 paper · 2022: 4 papers · 2023: 5 papers · 2024: 3 papers · 2025: 2 papers · 2026: 1 paper
Papers 17
- Mucosal-associated invariant T cells promote PDAC progression via TL1A-CSF-1 axis. Last author DOI 10.1007/s00535-026-02468-4↗
- Genomic mutation and Clinicopathological features of KRAS wild-type tumors in Chinese cohort with pancreatic adenocarcinoma. Last author DOI 10.1016/j.ygeno.2025.111048↗
- Effect of Laparoscopic Versus Open Distal Pancreatectomy on Recurrence-free Survival in Patients With Left-sided Pancreatic Cancer: A Randomized Controlled Tri… Co-author DOI 10.1097/sla.0000000000006681↗
- The complement C3a/C3aR pathway is associated with treatment resistance to gemcitabine-based neoadjuvant therapy in pancreatic cancer. Last author DOI 10.1016/j.csbj.2024.09.032↗
- The GFPT2-O-GlcNAcylation-YBX1 axis promotes IL-18 secretion to regulate the tumor immune microenvironment in pancreatic cancer. Co-author DOI 10.1038/s41419-024-06589-7↗
- Suppression of pancreatic cancer proliferation through TXNIP-mediated inhibition of the MAPK signaling pathway. Co-author DOI 10.3724/abbs.2023286↗
- Tissue-resident CXCR4+ macrophage as a poor prognosis signature promotes pancreatic ductal adenocarcinoma progression. Last author DOI 10.1002/ijc.34468↗
- Deubiquitinating PABPC1 by USP10 upregulates CLK2 translation to promote tumor progression in pancreatic ductal adenocarcinoma. Last author DOI 10.1016/j.canlet.2023.216411↗
- Knockdown of TACC3 inhibits tumor cell proliferation and increases chemosensitivity in pancreatic cancer. Last author DOI 10.1038/s41419-023-06313-x↗
- Inhibition of epithelial-to-mesenchymal transition augments antitumor efficacy of nanotherapeutics in pancreatic ductal adenocarcinoma. Last author DOI 10.1111/febs.16879↗
- Novel strategy for oncogenic alteration-induced lipid metabolism reprogramming in pancreatic cancer. Co-author DOI 10.3724/abbs.2023045↗
- SIGLEC15 amplifies immunosuppressive properties of tumor-associated macrophages in pancreatic cancer. Last author DOI 10.1016/j.canlet.2022.01.026↗
- Hypoxia-reprogrammed regulatory group 2 innate lymphoid cells promote immunosuppression in pancreatic cancer. Co-author DOI 10.1016/j.ebiom.2022.104016↗
- Neoadjuvant therapy alters the immune microenvironment in pancreatic cancer. Last author DOI 10.3389/fimmu.2022.956984↗
- TGF-β1-induced RAP2 regulates invasion in pancreatic cancer. Last author DOI 10.3724/abbs.2022015↗
- High GFPT1 expression predicts unfavorable outcomes in patients with resectable pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s12957-021-02147-z↗
- Cross-talk among AFAP1-AS1, ACVR1 and microRNA-384 regulates the stemness of pancreatic cancer cells and tumorigenicity in nude mice. Co-author DOI 10.1186/s13046-019-1051-0↗
Closest collaborators
The 25 strongest of 41 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Kaizhou Jin · 16 shared papers, 2021–2026 · strength 3.09
- Paper Mucosal-associated invariant T cells promote PDAC progression via TL1A-CSF-1 axis. DOI 10.1007/s00535-026-02468-4↗
- Paper The complement C3a/C3aR pathway is associated with treatment resistance to gemcitabine-based neoadjuvant therapy in pan… DOI 10.1016/j.csbj.2024.09.032↗
- Paper The GFPT2-O-GlcNAcylation-YBX1 axis promotes IL-18 secretion to regulate the tumor immune microenvironment in pancreati… DOI 10.1038/s41419-024-06589-7↗
- Xianjun Yu · 15 shared papers, 2021–2026 · strength 2.85
- Paper Mucosal-associated invariant T cells promote PDAC progression via TL1A-CSF-1 axis. DOI 10.1007/s00535-026-02468-4↗
- Paper The complement C3a/C3aR pathway is associated with treatment resistance to gemcitabine-based neoadjuvant therapy in pan… DOI 10.1016/j.csbj.2024.09.032↗
- Paper The GFPT2-O-GlcNAcylation-YBX1 axis promotes IL-18 secretion to regulate the tumor immune microenvironment in pancreati… DOI 10.1038/s41419-024-06589-7↗
- Long‐Yun Ye · 13 shared papers, 2022–2026 · strength 2.62
- Paper Mucosal-associated invariant T cells promote PDAC progression via TL1A-CSF-1 axis. DOI 10.1007/s00535-026-02468-4↗
- Paper The complement C3a/C3aR pathway is associated with treatment resistance to gemcitabine-based neoadjuvant therapy in pan… DOI 10.1016/j.csbj.2024.09.032↗
- Paper The GFPT2-O-GlcNAcylation-YBX1 axis promotes IL-18 secretion to regulate the tumor immune microenvironment in pancreati… DOI 10.1038/s41419-024-06589-7↗
- Qinglin Fei · 9 shared papers, 2022–2026 · strength 1.72
- Paper Mucosal-associated invariant T cells promote PDAC progression via TL1A-CSF-1 axis. DOI 10.1007/s00535-026-02468-4↗
- Paper Genomic mutation and Clinicopathological features of KRAS wild-type tumors in Chinese cohort with pancreatic adenocarci… DOI 10.1016/j.ygeno.2025.111048↗
- Paper Suppression of pancreatic cancer proliferation through TXNIP-mediated inhibition of the MAPK signaling pathway. DOI 10.3724/abbs.2023286↗
- Saimeng Shi · 7 shared papers, 2022–2024 · strength 1.28
- Paper The complement C3a/C3aR pathway is associated with treatment resistance to gemcitabine-based neoadjuvant therapy in pan… DOI 10.1016/j.csbj.2024.09.032↗
- Paper Suppression of pancreatic cancer proliferation through TXNIP-mediated inhibition of the MAPK signaling pathway. DOI 10.3724/abbs.2023286↗
- Paper Knockdown of TACC3 inhibits tumor cell proliferation and increases chemosensitivity in pancreatic cancer. DOI 10.1038/s41419-023-06313-x↗
- Tianjiao Li · 6 shared papers, 2022–2024 · strength 1.00
- Paper The GFPT2-O-GlcNAcylation-YBX1 axis promotes IL-18 secretion to regulate the tumor immune microenvironment in pancreati… DOI 10.1038/s41419-024-06589-7↗
- Paper Tissue-resident CXCR4+ macrophage as a poor prognosis signature promotes pancreatic ductal adenocarcinoma progression. DOI 10.1002/ijc.34468↗
- Paper Deubiquitinating PABPC1 by USP10 upregulates CLK2 translation to promote tumor progression in pancreatic ductal adenoca… DOI 10.1016/j.canlet.2023.216411↗
- Huiru Zhang · 5 shared papers, 2022–2024 · strength 0.91
- Paper The GFPT2-O-GlcNAcylation-YBX1 axis promotes IL-18 secretion to regulate the tumor immune microenvironment in pancreati… DOI 10.1038/s41419-024-06589-7↗
- Paper Neoadjuvant therapy alters the immune microenvironment in pancreatic cancer. DOI 10.3389/fimmu.2022.956984↗
- Paper Tissue-resident CXCR4+ macrophage as a poor prognosis signature promotes pancreatic ductal adenocarcinoma progression. DOI 10.1002/ijc.34468↗
- Duancheng Guo · 4 shared papers, 2023–2024 · strength 0.90
- Paper The complement C3a/C3aR pathway is associated with treatment resistance to gemcitabine-based neoadjuvant therapy in pan… DOI 10.1016/j.csbj.2024.09.032↗
- Paper Novel strategy for oncogenic alteration-induced lipid metabolism reprogramming in pancreatic cancer. DOI 10.3724/abbs.2023045↗
- Paper Suppression of pancreatic cancer proliferation through TXNIP-mediated inhibition of the MAPK signaling pathway. DOI 10.3724/abbs.2023286↗
- Zhenyu Liao · 5 shared papers, 2022–2023 · strength 0.75
- Paper Tissue-resident CXCR4+ macrophage as a poor prognosis signature promotes pancreatic ductal adenocarcinoma progression. DOI 10.1002/ijc.34468↗
- Paper Deubiquitinating PABPC1 by USP10 upregulates CLK2 translation to promote tumor progression in pancreatic ductal adenoca… DOI 10.1016/j.canlet.2023.216411↗
- Paper Inhibition of epithelial-to-mesenchymal transition augments antitumor efficacy of nanotherapeutics in pancreatic ductal… DOI 10.1111/febs.16879↗
- Chen Liu · 4 shared papers, 2021–2025 · strength 0.71
- Paper The GFPT2-O-GlcNAcylation-YBX1 axis promotes IL-18 secretion to regulate the tumor immune microenvironment in pancreati… DOI 10.1038/s41419-024-06589-7↗
- Paper Effect of Laparoscopic Versus Open Distal Pancreatectomy on Recurrence-free Survival in Patients With Left-sided Pancre… DOI 10.1097/sla.0000000000006681↗
- Paper TGF-β1-induced RAP2 regulates invasion in pancreatic cancer. DOI 10.3724/abbs.2022015↗
- Xuan Lin · 3 shared papers, 2022–2023 · strength 0.43
- Paper Tissue-resident CXCR4+ macrophage as a poor prognosis signature promotes pancreatic ductal adenocarcinoma progression. DOI 10.1002/ijc.34468↗
- Paper Hypoxia-reprogrammed regulatory group 2 innate lymphoid cells promote immunosuppression in pancreatic cancer. DOI 10.1016/j.ebiom.2022.104016↗
- Paper SIGLEC15 amplifies immunosuppressive properties of tumor-associated macrophages in pancreatic cancer. DOI 10.1016/j.canlet.2022.01.026↗
- Zhiwen Xiao · 3 shared papers, 2022 · strength 0.42
- Paper TGF-β1-induced RAP2 regulates invasion in pancreatic cancer. DOI 10.3724/abbs.2022015↗
- Paper Hypoxia-reprogrammed regulatory group 2 innate lymphoid cells promote immunosuppression in pancreatic cancer. DOI 10.1016/j.ebiom.2022.104016↗
- Paper SIGLEC15 amplifies immunosuppressive properties of tumor-associated macrophages in pancreatic cancer. DOI 10.1016/j.canlet.2022.01.026↗
- Guopei Luo · 2 shared papers, 2021–2025 · strength 0.31
- Paper Effect of Laparoscopic Versus Open Distal Pancreatectomy on Recurrence-free Survival in Patients With Left-sided Pancre… DOI 10.1097/sla.0000000000006681↗
- Paper High GFPT1 expression predicts unfavorable outcomes in patients with resectable pancreatic ductal adenocarcinoma. DOI 10.1186/s12957-021-02147-z↗
- He Cheng · 2 shared papers, 2021–2022 · strength 0.28
- Huaxiang Xu · 2 shared papers, 2022 · strength 0.26
- Liang Liu · 2 shared papers, 2022 · strength 0.26
- Xianjun Yu · 1 shared paper, 2025 · strength 0.24
- Bing Peng · 1 shared paper, 2025 · strength 0.19
- Xu Wang · 1 shared paper, 2025 · strength 0.19
- Jin Xu · 1 shared paper, 2025 · strength 0.19
- He Cheng · 1 shared paper, 2025 · strength 0.19
- Wenyan Xu · 1 shared paper, 2025 · strength 0.19
- Wei Wang · 1 shared paper, 2025 · strength 0.19
- Chenghao Shao · 1 shared paper, 2025 · strength 0.19
- Rong Liu · 1 shared paper, 2025 · strength 0.19
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.