ResearcherUniversité de SherbrookeNo recent work listed

Marie‐Josée Boucher

ORCID-verified (high confidence, 0.95), identity_conf@1As of 7 November 2026

Matched on ORCID iD 0000-0002-7615-3121; OpenAlex author ID A5051818638. Sources: OpenAlex.

Their work

Papers: 3 (first author 0, last author 3)
Trials: 0
Grants: 0
Labs led: 1
Collaborators: 2

Papers, trials and grants by year

2012: 1 paper · 2013: 1 paper · 2014: none · 2015: 1 paper

Papers 3

Glycogen synthase kinase-3 (GSK3) inhibition induces prosurvival autophagic signals in human pancreatic cancer cells. Last author DOI 10.1074/jbc.m114.616714 ↗
The MEK/ERK pathway promotes NOTCH signalling in pancreatic cancer cells. Last author DOI 10.1371/journal.pone.0085502 ↗
Inhibition of glycogen synthase kinase-3 activity triggers an apoptotic response in pancreatic cancer cells through JNK-dependent mechanisms. Last author DOI 10.1093/carcin/bgr309 ↗

Closest collaborators

The 2 strongest of 2 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Benoît Marchand · 2 shared papers, 2012–2015 · strength 0.11
- Paper Glycogen synthase kinase-3 (GSK3) inhibition induces prosurvival autophagic signals in human pancreatic cancer cells. DOI 10.1074/jbc.m114.616714 ↗
- Paper Inhibition of glycogen synthase kinase-3 activity triggers an apoptotic response in pancreatic cancer cells through JNK… DOI 10.1093/carcin/bgr309 ↗
Isabelle Tremblay · 2 shared papers, 2012–2013 · strength 0.09
- Paper The MEK/ERK pathway promotes NOTCH signalling in pancreatic cancer cells. DOI 10.1371/journal.pone.0085502 ↗
- Paper Inhibition of glycogen synthase kinase-3 activity triggers an apoptotic response in pancreatic cancer cells through JNK… DOI 10.1093/carcin/bgr309 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.