ResearcherTohoku UniversityNo recent work listed
Naoki Yoshida
Matched on ORCID iD 0000-0002-7954-1445; OpenAlex author ID A5091337537. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 13
2013: 1 paper · 2014: none · 2015: none · 2016: none · 2017: 2 papers · 2018: 1 paper · 2019: none · 2020: 2 papers · 2021: 1 paper · 2022: 1 paper
Papers 8
- Clinical features and prognostic impact of asymptomatic pancreatic cancer. Co-author DOI 10.1038/s41598-022-08083-6↗
- Focal Parenchymal Atrophy of the Pancreas Is Frequently Observed on Pre-Diagnostic Computed Tomography in Patients with Pancreatic Cancer: A Case-Control Study. Co-author DOI 10.3390/diagnostics11091693↗
- New-Onset or Exacerbation of Diabetes Mellitus Is a Clue to the Early Diagnosis of Pancreatic Cancer. Co-author DOI 10.1620/tjem.252.353↗
- Focal Parenchymal Atrophy and Fat Replacement Are Clues for Early Diagnosis of Pancreatic Cancer with Abnormalities of the Main Pancreatic Duct. Co-author DOI 10.1620/tjem.252.63↗
- Exosomes derived from pancreatic cancer cells induce activation and profibrogenic activities in pancreatic stellate cells. First author DOI 10.1016/j.bbrc.2017.10.141↗
- Kindlin-2 in pancreatic stellate cells promotes the progression of pancreatic cancer. Co-author DOI 10.1016/j.canlet.2017.01.008↗
- Exosomes Derived From Pancreatic Stellate Cells: MicroRNA Signature and Effects on Pancreatic Cancer Cells. Co-author DOI 10.1097/mpa.0000000000000722↗
- miR-210 regulates the interaction between pancreatic cancer cells and stellate cells. Co-author DOI 10.1016/j.bbrc.2013.06.097↗
Closest collaborators
The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Shin Hamada · 8 shared papers, 2013–2022 · strength 0.73
- Paper Clinical features and prognostic impact of asymptomatic pancreatic cancer. DOI 10.1038/s41598-022-08083-6↗
- Paper Focal Parenchymal Atrophy of the Pancreas Is Frequently Observed on Pre-Diagnostic Computed Tomography in Patients with… DOI 10.3390/diagnostics11091693↗
- Paper Exosomes derived from pancreatic cancer cells induce activation and profibrogenic activities in pancreatic stellate cel… DOI 10.1016/j.bbrc.2017.10.141↗
- Tetsuya Takikawa · 8 shared papers, 2013–2022 · strength 0.73
- Paper Clinical features and prognostic impact of asymptomatic pancreatic cancer. DOI 10.1038/s41598-022-08083-6↗
- Paper Focal Parenchymal Atrophy of the Pancreas Is Frequently Observed on Pre-Diagnostic Computed Tomography in Patients with… DOI 10.3390/diagnostics11091693↗
- Paper Exosomes derived from pancreatic cancer cells induce activation and profibrogenic activities in pancreatic stellate cel… DOI 10.1016/j.bbrc.2017.10.141↗
- Atsushi Masamune · 8 shared papers, 2013–2022 · strength 0.73
- Paper Clinical features and prognostic impact of asymptomatic pancreatic cancer. DOI 10.1038/s41598-022-08083-6↗
- Paper Focal Parenchymal Atrophy of the Pancreas Is Frequently Observed on Pre-Diagnostic Computed Tomography in Patients with… DOI 10.3390/diagnostics11091693↗
- Paper Exosomes derived from pancreatic cancer cells induce activation and profibrogenic activities in pancreatic stellate cel… DOI 10.1016/j.bbrc.2017.10.141↗
- Kazuhiro Kikuta · 5 shared papers, 2017–2022 · strength 0.51
- Paper Clinical features and prognostic impact of asymptomatic pancreatic cancer. DOI 10.1038/s41598-022-08083-6↗
- Paper Focal Parenchymal Atrophy of the Pancreas Is Frequently Observed on Pre-Diagnostic Computed Tomography in Patients with… DOI 10.3390/diagnostics11091693↗
- Paper New-Onset or Exacerbation of Diabetes Mellitus Is a Clue to the Early Diagnosis of Pancreatic Cancer. DOI 10.1620/tjem.252.353↗
- Shin Miura · 4 shared papers, 2020–2022 · strength 0.44
- Paper Clinical features and prognostic impact of asymptomatic pancreatic cancer. DOI 10.1038/s41598-022-08083-6↗
- Paper Focal Parenchymal Atrophy of the Pancreas Is Frequently Observed on Pre-Diagnostic Computed Tomography in Patients with… DOI 10.3390/diagnostics11091693↗
- Paper New-Onset or Exacerbation of Diabetes Mellitus Is a Clue to the Early Diagnosis of Pancreatic Cancer. DOI 10.1620/tjem.252.353↗
- Ryotaro Matsumoto · 4 shared papers, 2020–2022 · strength 0.44
- Paper Clinical features and prognostic impact of asymptomatic pancreatic cancer. DOI 10.1038/s41598-022-08083-6↗
- Paper Focal Parenchymal Atrophy of the Pancreas Is Frequently Observed on Pre-Diagnostic Computed Tomography in Patients with… DOI 10.3390/diagnostics11091693↗
- Paper New-Onset or Exacerbation of Diabetes Mellitus Is a Clue to the Early Diagnosis of Pancreatic Cancer. DOI 10.1620/tjem.252.353↗
- Michiaki Unno · 4 shared papers, 2017–2022 · strength 0.40
- Paper Clinical features and prognostic impact of asymptomatic pancreatic cancer. DOI 10.1038/s41598-022-08083-6↗
- Paper New-Onset or Exacerbation of Diabetes Mellitus Is a Clue to the Early Diagnosis of Pancreatic Cancer. DOI 10.1620/tjem.252.353↗
- Paper Focal Parenchymal Atrophy and Fat Replacement Are Clues for Early Diagnosis of Pancreatic Cancer with Abnormalities of … DOI 10.1620/tjem.252.63↗
- Tooru Shimosegawa · 4 shared papers, 2013–2018 · strength 0.30
- Paper Exosomes derived from pancreatic cancer cells induce activation and profibrogenic activities in pancreatic stellate cel… DOI 10.1016/j.bbrc.2017.10.141↗
- Paper Exosomes Derived From Pancreatic Stellate Cells: MicroRNA Signature and Effects on Pancreatic Cancer Cells. DOI 10.1097/mpa.0000000000000722↗
- Paper Kindlin-2 in pancreatic stellate cells promotes the progression of pancreatic cancer. DOI 10.1016/j.canlet.2017.01.008↗
- Fumiya Kataoka · 2 shared papers, 2021–2022 · strength 0.25
- Masahiro Iseki · 2 shared papers, 2020 · strength 0.19
- Kei Nakagawa · 1 shared paper, 2022 · strength 0.13
- Toru Furukawa · 1 shared paper, 2020 · strength 0.09
- Fuyuhiko Motoi · 1 shared paper, 2017 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.